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Besides hemal sex migraine, cause the cause that have a headache a lot of, if abscess of all sorts of sexual disease, encephalitis, meningitis, head is affected inside skull; All sorts of cerebral blood-vessel pathological changes, if cerebral block, cerebral hemorrhage, arachnoid haemorrhage finishing speech, ventricle bleeds, Hypertensive Cerebral disease; A pathological change is taken inside skull, if colloid tumour mixes meningeal tumour, head,transfer tumor; All sorts of heads are traumatic if defeat of cerebral concussion, head cracks injury, putamen,issue haematoma; The waist is worn cause low skull pressure head painful; All sorts of neuralgic; The headache of disease of facial features division and infection, toxic, heatstroke and nerve function all can be caused mussily have a headache.

除了血管性偏头痛外，引起头痛的原因很多，如颅内感染性疾病、各种脑炎、脑膜炎、脑脓肿；各种脑血管病变，如脑梗塞、脑出血、蛛网膜下腔出血、脑室出血、高血压脑病等；颅内占位病变，如脑膜瘤、脑胶质瘤和转移肿瘤；各种脑外伤如脑震荡、脑挫裂伤、硬膜下血肿；腰穿引起低颅压头痛；各种神经痛；五官科疾病的头痛以及感染、中毒、中暑和神经功能紊乱均可引起头痛。

Results The clinical manifestation of brain stem hemorrhage secondary to cerebral infarction included sudden onset,evelution abruptly,coma,quadriplegia,higher fever and respiratory failure. CT usually do not identify the high dense location of brain stem. Large infarction of cerebral hemisphere may lead to shift the brain laterally and downward to produce herniation with subsequent brain stem compression,shift and distortion. Secondary brain stem hemorrhage occured usually in midderline area of midbrain or pontine.

结果 脑梗塞继发脑干出血的临床特点是发病急、进展快、昏迷、四肢瘫、过高热及呼吸衰竭，CT一般不能发现脑干高密度病灶，病理结果均为半球大病灶脑梗塞，严重海马回疝致脑干严重受压、变形和移位，继发脑干出血以中脑为主，可累及桥脑，主要位于中线部位。

Results According the MRI characteristric of hypoxic-ischemic encephalopath sequela:(1)The most common findings were white matter injury in 47 cases:①Periventricular leukomalacia in 23 cases;②Selective cerebral neuronal necrosis of basilar node lentiform nucleus and optic colliculus in 9 cases;③Corpus callosum damaged in 15 cases;(2)The most damaged of grey matter in 18 cases;(3)Focal or multifocal ischemic crerbral intenerate in 9 cases;(4)Diffuse crerbral injury in 5 cases.

结果 根据儿童缺血缺氧性脑病后遗症的MRI影像特点，将其分为：(1)脑白质损伤为主：47例；①脑室周围脑白质脱髓鞘、软化：23例；②基底节豆状核、视丘神经元选择性损害：9例；③胼胝体受损变薄：15例；(2)脑灰质损害为主：18例；(3)局部或多处脑软化：9例；(4)广泛脑损害：5例。

Results According the MRI characteristric of hypoxic\|ischemic encephalopath sequela:(1)The most common findings were white matter injury in 47 cases:①Periventricular leukomalacia in 23 cases;②Selective cerebral neuronal necrosis of basilar node lentiform nucleus and optic colliculus in 9 case...

结果 根据儿童缺血缺氧性脑病后遗症的MRI影像特点，将其分为：（1）脑白质损伤为主：47 例；①脑室周围脑白质脱髓鞘、软化：23 例；②基底节豆状核、视丘神经元选择性损害：9 例；③胼胝体受损变薄：15 例；（2）脑灰质损害为主：18 例；（3）局部或多处脑软化：9 例；（4）广泛脑损害：5 例。

Of the 26 lesions with pontine and bulbar involvement, three had no association with other lesions (Fig 2). Twelve were associated with tegmental and superior cerebellar peduncular extension, and six showed corticospinal tract involvement in continuity with an MDJ lesion. In one patient with additional telencephalic lesions, the right side of the pons was hyperintense on long TR/TE images that did not cross the midline, suggestive of an arterial lesion (Fig 3). In two patients with pontine tegmental lesions, there was an associated middle cerebellar peduncle and deep cerebellar white matter involvement (Fig 4). Two others had associated cervical lesions.

在26处脑桥延髓区病灶中，3处病灶仅限于脑桥延髓区，其它部位无病灶（图2）；12处病灶还同时伴有背盖部和小脑上脚/结合臂处病灶；6处病灶为与中脑-间脑接合处病灶相连的皮质脊髓束受累；还有1例患者除端脑见病灶外，右侧脑桥于T2像也见高信号病灶，但未超越正中线，提示可能系动脉病变所致（图3）；2例脑桥背盖部病灶的患者同时伴有小脑中脚/小脑脑桥脚和小脑深部白质受累（图4）；还有2处脑桥延髓区病灶还同时伴有颈髓病灶。

ABSTRACT AIM: To study the effects of dl-, l- and d-3-n-butylphthalide on pial arteriole diameter and blood flow velocity in focal ischemia rats. METHODS: Urethane-anesthetized rats were instrumented for monitoring pial AD and BFV in the cranial window preparation.

经多年研究表明，本室研制开发的一类新型抗脑缺血药物丁基苯酞，有调节脑缺血状态下脑能量代谢［1］、增加缺血区脑血流量［2］、缩小脑梗塞面积、减轻脑水肿［3］、改善脑缺血后神经功能缺损症状的作用。

There were no significant differences for the firing rates in the site of contralateral TNC neurons among during pre-CSD,CSD,and post-CSD (P＞0.05).For flunarizine group,the firing rates in the site of ipsilateral TNC neurons during pre-CSD were higher as compared with during CSD(P＜0.05).2.1 There were statistical differences on palasma levels of CGRP and SP among the three groups(P＜0.05).The levels of CGRP and SP in CSD group were higher than control group(P＜0.05).No significant differences on the levels of CGRP and SP in ipsilateral trigeminal ganglia were found among the three groups(P＞0.05).2 The number of neurons with positive CGRP and SP immunoreactivity was statistically different in right-sided trigeminal ganglia among the three groups (P＜0.05).The number in fight-sided trigeminal ganglia in CSD group was higher as compared with control group(P＜0.05).The number in right-sided trigeminal ganglia was statistically higher than that in left-sided trigeminal ganglion in CSD group(P＜0.05).3.1 Altered ReHo in ipsilateral pons and other brain regions response to pain such as basal nuclei,thalamus,cingulated gyms and prefrontal cortex was detected during the acute spontaneous attack as compared with during headache remission(P＜0.05,corrected by Monte Carlo simulation). 2 Positive functional connectivity was detected between ipsilateral pons and other brain regions related to pain within pain state and within non-pain state (P＜0.05,corrected by false discovery rate,FDR).Increased functional correlation between ipsilateral pons and other pain-related brain regions such as ipsilateral prefrontal cortex and contralateral subcallosal gyrus was detected during the acute spontaneous attack as compared with during headache remission(P＜0.05,corrected by Monte Carlo simulation).

结果1。对照组未发现CSD；同侧TNC放电频率，CSD中＞CSD后＞CSD前P＜0.05对侧TNC放电频率，CSD前、中、后无统计学差异(P＞0.05氟桂利嗪组同侧TNC放电频率，CSD前＞CSD中(P＜0.05)，CSD前与CSD后及CSD中与CSD后之间无统计学差异(P＞0.05)。2.1关于放免测定，各组血浆CGRP、SP水平有统计学差异(P＜0.05)，CSD组高于对照组(P＜0.05)，CSD组与氟桂利嗪组、对照组与氟桂利嗪组之间均无统计学差异P＞0.05各组之间同侧三叉神经节中CGRP、SP水平未见变化(P＞0.05.2关于免疫组化研究，右侧三叉神经节CGRP、SP免疫阳性细胞数三组之间有统计学差异(P＜0.05)，多重两两比较结果CSD组大于对照组(P＜0.05)，CSD组与氟桂利嗪组之间、对照组与氟桂利嗪组之间无统计学差异P＞0.05左侧三叉神经节CGRP、SP免疫阳性细胞数三组之间无统计学差异(P＞0.05CSD组中右侧三叉神经节CGRP、SP免疫反应阳性细胞数大于左侧(P＜0.05)。3.1局部一致性分析发现两组患者头痛疼痛状态较非疼痛状态脑活动发生变化的脑区有同侧脑桥以及其他疼痛相关脑区如基底节区、丘脑、扣带回、前额叶皮层等(P＜0.05，蒙特卡罗模拟校正)。2功能连接分析发现疼痛状态与非疼痛状态下主要疼痛相关脑区均与同侧脑桥有功能联系P＜0.05，false discovery rate，FDR校正疼痛状态与非疼痛状态比较，同侧前额叶皮层、对侧胼胝下回等疼痛相关脑区与同侧脑桥之间功能联系增强(P＜0.05，蒙特卡罗模拟校正。

Every group rats were divided into two parts with the same rats. At last the rats were redivided ,then them were killed and their brains were excluded after one hour.4 hours or one week.Results PO2 and cerebral blood flow of E-selectin 2mg/kg group were higher than that of saline and lower than that of l0mg/kggroup(p.01).The volume of cerebral infarct and blood speed of E-selectin 2mg/kg group was smaller than that of saline and larger than that of l0mg/kg group(p.01).At the same time PCh.speeds.flows and the volume of infarct were related with the time (p.01).Conclusion E-selectin can effectively increase PC2 and rCBF after ischemia and reperfusion .reducing infarct volumes and speeds of blood at the same time . Artificial synthetic E-selectin can effectively reduce the reperfusion injury following cerebral ischemia.

结果 生理盐水组，2mg／kg组，10mg／kg组PO_2值依次提高(p＜0.01)，脑皮质血流量依次提高(p＜0.01)；脑血流速度依次减低；脑梗死体积依次减少(p＜0.01)；同时在测定的时间段里PO_2和脑组织血流量值与时间成正相关(p＜0.01)；脑梗死体积和脑组织血流速度值与时间成正相关(p＜0.01)。E一选择素在脑缺血再灌注损伤中表达的实验研究中文摘要结论E一选择素能增加脑缺血再灌注损伤的脑组织氧饱和度，脑血流量，减低脑血流速度；减少脑损伤体积；从而保护了脑缺血再灌注损伤的脑组织。

To begin with the review of human's understanding on the brain, we will explain the brain formation and working principles in simple terms. How the complex and ingenious structure of brain is formed? Why can we see the colorful world? Why can we hear the sweet warble of the bird and touching music? Why do we have intelligence and thoughts? Why do we have pleasure, anger, sorrow and joy? How can we acquire information and form consolidated memory, and what can affect the learning and memory? Why do we need sleep, and what determine the biorhythm? How do we control our movement, thinking and emotion? How do Alzheimer's disease, stroke, depression, anxiety, schizophrenia, and drug addiction happen? Can adult neuron regenerate?

从回顾人类对脑的认识过程开始，我们将一步步深入浅出的告诉你大脑形成和工作的原理：大脑复杂而精妙的结构是如何形成的；我们为什么能看到色彩缤纷、千姿百态的世界；为什么能听到悦耳动听的鸟的啼啭和动人心弦的音乐旋律；我们为什么有智力、能思维；为什么有喜怒哀乐；我们如何获取信息并形成牢固的记忆，哪些因素影响学习记忆的过程；我们为什么需要睡眠，是什么决定了我们的生物节律；我们如何控制自己的运动、思维和情绪；老年痴呆、中风、抑郁、焦虑、精神分裂症、药物成瘾是如何产生的；成年脑神经细胞能再生吗？

Brain developmentcenter provides a series of productsfrom Beijing research institute ofChinese brain medication for preventing Alzeheimer's, children over activation, autism, bloodvessel and nerve soreness and loweringcholesterol level, such as brain development and conformer capsule as well as stress release capsule. Also, we produce themedicines forreducing ache from period, brainfiredness, vibriosis and andro-function strengthening.

养脑中心还将推广北京中研中医脑病研究院的养脑系列产品，其中包括预防老年性痴呆，降低胆固醇，防治小儿多动症，自闭症及血管神经疼痛等系列养脑产品，如脑毒清胶囊，脑络通胶囊，脑立舒胶囊，脑力宝胶囊，脑神健胶囊，不孕不育的女用助孕一号，二号及男用补肾1号，2号胶囊，做到疗效迅速，价格合理。

This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。