脑死亡的
- 与 脑死亡的 相关的网络例句 [注:此内容来源于网络,仅供参考]
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Among them, because the diseases such as artery sclerosis and diabetes, kidney trouble, heart attack and head stroke caused, etc., will produce great agony after not having illness coming on, might cause half-length paralysis, old age such as being dull-witted, even die, the threat to the life is great, can't really regard as unimportant . As the saying goes bad something.
其中,因为动脉硬化所引发的病、肾脏病、心肌梗和脑中风等疾病,但会在发病后产生极大的痛苦,还有可能造成半身麻痹、痴呆等后遗症,甚至死亡,对於生命的威胁极大,实在可等闲视之。
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Results The incidence of multiple organ disorder syndrome was 8.2%which mainly occurred in encephalon cardio lumpliver renal and acute gastroduodenal ulcers.
结果:本组多器官功能失常的发生率占重症胸外伤的比例为8.2%,主要为脑、心、肺、肝、肾、和消化应激性溃疡出血等多器官功能失常,在一系列积极的抢救措施下仍有5例死亡,死亡率为41.6%。
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Results The incidence of multiple organ disorder syndrome was 8.2%, which mainly occurred in encephalon, cardio, lump, liver, renal and acute gastroduodenal ulcers. Although with series hard treatment, five cases died, the total mortality is 41.6%.
结果 该组多器官功能失常的发生率占重症胸外伤的比例为8.2%,主要为脑、心、肺、肝、肾和消化应激性溃疡出血等多器官功能失常,在一系列积极的抢救措施下仍有5例死亡,死亡率为41.6%。
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Results The incidence of multiple organ disorder syndrome was 8.2%,which mainly occurred in encephalon, cardio, lump,liver, renal and acute gastroduodenal ulcers.
结果:本组多器官功能失常的发生率占重症胸外伤的比例为8.2%,主要为脑、心、肺、肝、肾、和消化应激性溃疡出血等多器官功能失常,在一系列积极的抢救措施下仍有5例死亡,死亡率为41.6%。
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Results The incidence of multiple organ disfunction syndrome was 8.2% following severe thoracic injuries,which mainly occurred in encephalon,cardio,lump,liver,renal and acute gastroduodenal ulcers.Although with series hard treatment,five cases died,the total mortality was 41.6%.
结果 本组多器官功能失常的发生率占重症胸外伤的比例为8.2%,主要为脑、心、肺、肝、肾和消化道应激性溃疡出血等多器官功能失常,在一系列积极的抢救措施下仍有5例死亡,死亡率为41.6%。
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Dr Gros and his colleagues are examining the second part of this process because it is after this stage that infected blood cells can become trapped in small blood vessels in the brain, causing cerebral malaria and, potentially, death.
Gros博士和他的同事们正在研究这个过程的第二部分。因为正是是经过这个阶段,被感染的血细胞可能滞留在大脑的小血管中,从而引起脑型疟疾,并可能最终导致死亡。
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Stroke, a major cause of death and disability to the elderly, often results in delayed neurological morbility or mortality. Potassium cyanide intoxication has often been used as a model of chemical hypoxia.
1脑中风,造成老年人口的主要死亡原因,因此研究一有效药物以对抗中风后造成的严重损伤进而治愈其造成之后遗症,一直是从事研究工作者努力的目标。
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Results there were unconsciousness, respiratory rhythm disturbance and hyporeflexia of pupil light reflex immediately after injury, and reactiveness decrease and activity retardation still existed even after resuming consciousness. at anatomical scene, subarachnoid hemorrhage or cerebroventricular haemorrhage were widespread. at an early stage, there were swelling, collapse, and axonal retraction ball formation at cortico-medulla junction, callosum, brainstem, and cerebellar white matter under microscope. but at the later stage, gitter cell proliferation and nest-like aggregation were major pathophysiological changes at focal brain tissue.
结果 伤后大鼠均即刻出现原发昏迷,其中2只于损伤后20 min内死亡,余持续时间1-30 min不等;伤后大鼠呼吸节律紊乱,瞳孔对光反射减弱或消失,醒后均有程度不等的反应性下降,肢体活动迟缓;肉眼可见广泛蛛网膜下腔出血或脑室出血;光镜下可见皮髓交界区、胼胝体区、脑干、小脑白质等部位的神经轴索有不同程度的肿胀、断裂、轴索球形成,后期有小胶质细胞增生,局部呈巢样聚集。
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These growth factors have the effects of neurogenesis, angiogenesis, neurotrophy, inhibitory excitoxity induced by glutamate and NMDA, and anti-oxidative, by these ways they can prevent neural cells from death.
这些生长因数均有保护神经细胞避免因为脑损伤而造成的死亡,其作用有神经生长性、血管生长性、神经滋养性、抑制麸氨酸盐与NMDA所引发的兴奋性毒性及抗氧化作用,但是这些仅限於动物模式,应用於临床还有一段艰辛的历程。
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Microglia cell, an important resident immunoreactive cells in the CNS, its activation is not only responsible for the cleavage of dead cell debris, but also involved in the initiation and propagation of the neuron injury.
小胶质细胞是脑内重要的免疫反应活性细胞,它的激活不是对死亡细胞进行清除,而是参与到神经元损伤的启动和放大过程。
- 推荐网络例句
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This one mode pays close attention to network credence foundation of the businessman very much.
这一模式非常关注商人的网络信用基础。
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Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.
扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。
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There is no differences of cell proliferation vitality between labeled and unlabeled NSCs.
双标记神经干细胞的增殖、分化活力与未标记神经干细胞相比无改变。