脑损伤

The device has been developed with the use of aerodynamical principle, measuring technique for feeble signal, and integration of machine-electricity. The gas percussion, rigid impact, and cell injury can be produced by altering impact tip.

应用空气动力学原理、微弱信号检测技术和机电一体化的方法研制而成，通过更换撞击头可实现柔性撞击、刚性撞击和细胞损伤，通过眼、脑、肺和细胞进行实验研究。

Think at present, cause the main and immediate cause with heart disease and apoplectic head, arteriosclerosis, it is long the result of accumulate over a long period of injury of the film inside artery.

目前认为，造成心脏病和脑中风的主要直接原因—动脉硬化，是长时间动脉内膜损伤日积月累的结果。

Objective To study the protective effects of extract of astragalus against focal cerebral ischemia-reperfusion injury in rats.

目的 探讨黄芪提取物对实验性局脑缺血再灌注损伤的保护作用。

It is now known that seere hypoxia-ischemia may not cause immediate cell death, but may precipitate a complex biochemical cascade leading to the delayed deelopment of neuronal loss. These phases include a latent phase after reperfusion, with initial recoery of cerebral energy metabolism but EEG suppression, followed by a secondary phase characterized by accumulation of cytotoxins, seizures, cytotoxic edema, and failure of cerebral oxidatie metabolism from 6 to 15 h post insult.

现在认识到严重的局部缺氧有可能不立即引起细胞坏死，但是可能促成一种复杂的生化级联反应，这可能导致神经元丢失，进而导致延缓发育，这些时期包括再灌注后的潜伏期，该期出现早期的脑能量代谢复苏但脑电图却呈抑制状态，紧接着第二期表现为损伤后6－15个小时细胞毒素，癫痫，细胞毒性水肿，脑氧化功能衰竭的累积。

Objective: To investigate the brain-protective mechanism of edaravone in gerbils with cerebral ischemia-reperfusion injury.

目的：探讨依达拉奉对沙土鼠全脑缺血再灌注后自由基损伤的脑保护机制。

Objective To explore the effects of hypoxic preconditioning on middle cerebral artery occlusion-induced brain injuries and its possible mechanisms.

目的 探讨低氧预适应对脑中动脉阻塞所致脑缺血性损伤的影响及其可能机制。

TCM may modulate the glial cells through the following mechanisms: inhibiting expression of iNOS, thereby minishing the toxicity of NO and its oxygen free radicals; inhibiting overactivation of astrocytes and overexpression of GFAP in acute cerebral ischemia and cerebral hemorrhage; promoting glial cells' secretion of NGF which can be caused by ischemic injury; inhibiting the nervous system's immune inflammatory response; and so on.

中药调节神经胶质细胞活性的机制可能包括：（1）抑制iNOS表达，从而抑制NO及其产生的氧自由基的毒性；（2 抑制脑缺血和脑出血急性期星形胶质细胞的过度活化和GFAP的过度表达；(3)上调缺血损伤导致的神经胶质细胞分泌NGF；(4)抑制神经系统的免疫炎症反应等各种途径。

36 big mice stochastically divide into the vacation surgery group, castrate the group and castrate the sterandryl substitution group.

线栓法建立大鼠局灶性脑缺血再灌注损伤模型，TTC染色测脑梗死体积及免疫组化染色检测脑组织Bax的表达。

The results indicate that the level of IL-18 is elevated in serum and cerebrospinal fluid of epidemic encephalitis B and it is related to the degree of parenchymal lesions of brain.

血清及脑脊液中IL-18含量增高与脑实质损伤程度密切相关，其含量增高可作为估计乙型脑炎病情及预后的一项重要指标，并对乙脑免疫调节方面的治疗具有一定的参考价值。

Numerous experiments have already demonstrated that GBE has many clinical therapeutic applications not only for cerebral insufficiency, coronary artery disease, hypertension, angina, arteriosclerosis, but also for cognitive speed, dementia, and aging damages.

GBE 不仅对冠心病、高血压、心绞痛、动脉硬化有显著的预防和治疗效果，还用于治疗脑功能减退、老年性痴呆、老年性记忆减退、衰老等与脑内兴奋性损伤有关的疾病。

We are in a real jam.

我们的麻烦大了。

Hey, it's Ahmet from India, that foreign exchange guy.

看，那是印度的阿曼特，国际交换生