脑损伤

There was an obvious increase of insulin receptor gene and protein in the cerebral ischemia reperfusion tissues in rats. The positive cells such as neuron, neuroglia cells, vascular endothelium cells possibly participated in regulating the processes of energy metabolism of cells and neovascularization. It implied that the high expressions of insulin receptors in vascular endothelium cells in ischemic zones possibly participated in regulating the pathophysiological changes of the blood vessels in ischemic zones; The higher expressions of insulin receptors in different cells were possibly one of the self-protective machanisms in which the ischemic tissues confront the anoxemia injury.

脑缺血再灌注大鼠脑组织胰岛素受体基因和蛋白水平表达增加；半暗带区神经元、部分神经胶质细胞及血管内皮细胞等阳染细胞可能参与脑缺血后组织细胞的能量代谢、促新生细胞形成等病理生理调节过程；胰岛素受体在缺血区血管内皮细胞高表达提示其可能在缺血区血管的病理生理变化中起某种调节作用；胰岛素受体在不同细胞中表达增高可能是缺血脑组织对抗缺血缺氧损伤的自身保护机制之一。

The contents of Ca2+, CaM in blood and brain tissue were measured by atonic absorption spectrometry and radioimmunoassay.

颅脑损伤后神经元和神经胶质细胞膜系统的钙通道开放、细胞内钙超载、钙调蛋白激活，是导致继发性脑损害、脑水肿发生的关键［1］。

The rise of CINC was an effect of activiting WBC after the ischemia or ischemia reperfusion and indicated that the change was involving in the damage of ischemia reperfusion.

实验性脑缺血再灌注外周血CINC有表达增高改变，其作用过程可能与白细胞激活过程有关，提示CINC增高可能在脑缺血再灌注损伤中起一定作用。

The peaks were integrated and the relative values might be used to express the metabolism or damnification of brain.

各波峰积分后得到各种物质的相对含量，而这些代谢中间产物的相对含量变化可以反应脑内的代谢状况和脑受损伤情况。

It was shown that the TRH test is closely correlated with the injury types,the shift degree of brain,midline and the volume of EDH respectively on CT.The reduced reactions were most seen in the groups of SDH and midline shift ab...

结果发现：CT所表现的损伤类型、脑中线移位程度及EDH血肿量与TRH兴奋试验的改变有密切关系，反应不良出现率以SDH组及脑中线移位≥10mm组最高，EDH血肿量越大，TRH兴奋试验改变也越明显。

Results: The main reasons for acute encephalocele were delayed intracranial hematoma,acute brain swelling,hypotension or hypoxia and injury of large veins.

结果 导致脑膨出的主要原因有迟发性颅内血肿、急性弥漫性脑肿胀、低血压、低血氧及较大回流静脉的损伤。

The expressions of iNOS mRNA and proteins in VTA and FrA increased less than those in CPu and SNpc.

表达增高的iNOS可能对SNpc和VTA脑区DA能神经元及CPu和FrA脑区的DA能神经元末梢起了损伤作用，从而导致了DA能神经元的减少及其末梢退变的发生。

In traditional Chinese medicine group the VEGF express was obriously higher than the nomal saline group at each of the time points.

缺血再灌注可上调VEGF在大脑海马区域的表达；益气活血中药治疗急性脑缺血能增强VEGF蛋白的表达，从而减少毛细血管的消失，为内皮细胞增殖、迁移、新血管再生提供基础保障，这可能是益气活血中药抵抗脑缺血再灌注损伤的机制之一。

1,2-dichloroethane can damage blood-brain harrier cells, and induce cerebral edema.

结论］1，2-二氯乙烷可造成血脑屏障细胞的损伤，引起血管源性脑水肿。

Objective: To study the mechanism of gastrointestinal injury after brain ischemia reperfusi on in aged rats from the changes of dopamine,noradrenalin,epinephrine and the injury of free radical.

目的：从多巴胺、去甲肾上腺素、肾上腺素变化和自由基损伤方面揭示脑缺血再灌注胃肠损伤的机制。

The X-ray diffract was used to analyze the crystal structure of the composite. Results show that after the talc was treated with blend coupling agent, the crystal structure of talc changed, and effect Intercalation phenomenon.

通过X射线衍射分析可知，用混合偶联剂对滑石粉进行预处理后，滑石粉晶体结构发生改变，产生了插层现象，使PP／滑石粉复合材料为纳米复合材料。

An authentic Maui wedding might require the services of a 'kahu', which is a Hawaiian minister.

毛伊可能需要一个真实婚礼服务'kahu'，这是夏威夷部长。