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NYA can up-regulate the BDNF、GDNF protein and mRNA expression in brain of ICH injury rats.

（1）脑出血损伤大鼠脑内BDNF、GDNF蛋白及mRNA的表达均增强，脑溢安能上调BDNF、GDNF蛋白及mRNA的表达水平。

It indicated that NYA can enhance absorption of brain edema and hemotoma, alleviate degenerationand necrosis in brain, and quicken the repair.

提示脑溢安可明显促进脑水肿和血肿的吸收，减轻脑组织变性坏死，加快损伤修复。

The patients in NYA group were treated with NYA pills,while the patients in WM group were treated with normal western medicine.

脑溢安组给予脑溢安颗粒加西药基础治疗，西药组给予西药基础治疗，治疗四周后观察疗效。

Apoptosis was induced by hypoxia injury in the NSCs, the amount of apoptosis in the group treated with NYA serum was significantly lower than that of model group and normal-serum group. It suggested that NYA serum can inhibit the NSCs apoptosis.

培养神经干细胞缺氧损伤后发生凋亡，脑溢安血清组凋亡细胞数显著低于模型组和正常血清组，提示脑溢安能减少缺氧损伤神经干细胞凋亡。

The BDNF and GDNF protein expression level of NYA group were higher than model group at 1d、4d after ICH injury (P.05). To sum up, NYA could up-regulated the expression of BDNF and GDNF protein.

脑溢安组BDNF、GDNF蛋白的表达在1d、4d强于模型组，两组阳性细胞计数表明差异有显著性（P.05），提示脑溢安能上调上述两种蛋白的表达。

Cultured NSCs appeared damage after 6h of hypoxia, the amount of survival NSCs in the group treated with NYA serum was significantly higher than that of model group and normal-serum group. It suggested that NYA serum can promote the NSCs survival.

培养神经干细胞缺氧损伤后大量死亡，脑溢安血清组存活细胞数显著高于模型组和正常血清组，提示脑溢安能促进缺氧损伤神经干细胞存活。

It is indicated that the NYA group can effectively decrease the contents of plasmal NE, E and DA, to remove cerebrovascular spasm, to relieve cerebral edema, to improve microcirculation of brain tissue, to increase blood and energy supplies, anticerebral secondary ischemia and hypoxia and improve the clinical effects.

提示脑溢安颗粒剂能有效地降低血浆NE，E和DA含量，解除患者脑血管痉挛，减轻脑水肿，改善脑组织微循环，增加组织血供和能量供应，改善脑组织缺血缺氧状态，提高临床疗效。

Methods Rat models of cerebral edema after cardiac arrest were established by water-airproof bottle obturation methods, and CPR was carried out at once.

脑水肿是指脑组织的含液量增多而引起的脑容量增加，它是中枢神经系统受到各种外源性或内源性有害因素刺激时所产生的一种最常见的非特异性反应。

In most known examples, the affected proteins are enzymes, as in phenylketonuric oligophrenia which affects 1 in 10,000 of the population. It is due to deficiency in the liver cells of phenylalanine hydroxylase, which normally converts phenylalanine to tyrosine, and brain damage apparently results from raised blood and cerebrospinal fluid levels of phenylalanine and its metabolites.

在已知的大部分病例中受损的蛋白质是酶，如苯丙酮尿酸精神幼稚病，患者占人口的万分之一，是由于在正常情况下使苯基氨基酸转变为酪氨酸的肝细胞苯基丙氨酸羟化酶缺乏所致，因此血液和脑脊液的苯基丙氨酸和其代谢产物水平升高而引起脑损伤。

Verbal dyspraxia may result from damage to Broca's area, the supramarginal gyrus, supplementary motor area, frontal operculum, and insula.

口语运用障碍可能是由布洛卡区域损害发生造成的，布洛卡区域是指缘上的脑回，补充性的运动区域、额盖及脑岛。

Lugalbanda was a god and shepherd king of Uruk where he was worshipped for over a thousand years.

Lugalbanda 是神和被崇拜了一千年多 Uruk古埃及喜克索王朝国王。

I am coming just now,' and went on perfuming himself with Hunut, then he came and sat.

我来只是现在，'歼灭战perfuming自己与胡努特，那麼，他来到和SAT 。