脊髓运动神经元

Results: Current of 1 mA, 210 s could cause necrosis and cavitation in the dorsal funiculus, resulting in bilateral corticospinal tract injury; rats with electrolytic lesions exhibited declined motor function with decreased Tarlov score, increased Ashworth score and reduced inclined plane angle, the HRP labeled neuron counts in motor cortex were also significantly lower in the lesioned group than sham-operation group.

结果：1 mA， 210 s的损伤电流，可在脊髓后索造成神经组织坏死，形成空腔，使双侧皮质脊髓束纤维受损；术后1周、2周、3周观察，电损毁组大鼠运动功能显著低于假手术组，表现为后肢运动评分下降，Ashworth 肌张力评分增大，斜板实验角度下降；电损毁组运动皮质标记神经元计数也显著低于假手术组。

SCI can cause hematomyel ia, dropsy, necrosis, capsular space, gummosis and so on, which can lead to neuron deprivation, demyelination of neurite. Clinical symptoms mainly manifest funct ion disorder of movement, sense, reflectance, and musculus sphincter below injury plan.

SCI后出现脊髓出血、水肿、坏死、囊腔形成、退变胶质化等一系列病理改变，以致神经元丧失、轴突脱髓鞘，临床表现为受损平面以下的运动、感觉、反射及括约肌功能的障碍。

After treatment of CCl4 through oral route for four weeks, we have observed an interesting phenomenon in the animal model. That is the significantly decreased condition of nitric oxide synthase. The locations are sympathetic T7-T10 dorsal root ganglion, T7-T10 intermediolateral cell column, celiac ganglion, parasympathetic nodose ganglion, solitary tract nucleus, and dorsal motor vagal nucleus.

结果显示：在餵食四氯化碳四周起，其交感神经回路上的T7-T10背根神经节、T7-T10脊髓侧角中间外侧细胞柱之交感节前神经元以及位於腹腔神经节的交感节后神经元所含之一氧化氮合成下降；而副交感神经回路上的小结神经节、孤立径核以及迷走神经背侧运动核所含之一氧化氮合成亦下降。

NRR publishes original contributions written by experts and leaders in the fields of neuroanatomy, pathology, physiology, biochemistry, pharmacology, immunology, and auxanology approaches to examine neural regeneration research; it publishes experimental studies dealing with the structure and function of the nervous system, such as cerebral cortex, Hippocampus, pineal, glial cell, spinal cord neurons, peripheral neurons and sensory nerve injury and regeneration research.

NRR非常感兴趣来自神经解剖、病理、生理、生物化学、药理、免疫、发育等多学科、多层面的题材，感兴趣发表以基础实验性研究为主的揭示大脑皮质、海马、松果体、神经胶质细胞、脊髓神经元、周围神经元以及运动、感觉乃至植物神经损伤与再生的研究原著，对有助于认识神经再生正常和异常机制的临床类文章，如罕见病例报告、调查分析等也可纳入范围。

He is the first of four patients with spinal cord(6)injuries,muscular dystrophy(7), stroke or motor neurone(8) diseasetestingthe brain-to-movement system developed in Massachusetts.

四个分别患有脊髓损伤，肌肉失调，中风和运动神经元疾病的病人参与测试了这个在马萨诸塞州研制成功的大脑支配行动的装置。雷是他们中的第一个。

Especially for severe extremities disabled such as Injury to spinal cord, Motor neuron disease and so on, who are paralyses in a bed for a long-term. The people who have communication and action disabled, they couldn't do them well, so their lives are full of inconvenience. If they want to associate with the normal people, the most easy method is to make some word cards of reading. With these word cards to guess what he need from his health or psychology, such as the weathers turns cold,"cold","hot","wearing clothes" and so on, But it couldn't express all the thought from patients. Therefore, it often caused that we can't know what the patients want or their condition. It's too inconvenient for their lives.

尤其是重度肢体障碍患者，如脊髓损伤、运动神经元疾病患者等长期瘫痪在床的病人，对於沟通和行动障碍两者并存的患者而言，其日常生活起居处处充满著不便，一般与此类患者沟通最简单的方式就是制作一些简易读字卡，依著基本的身体或心理层面去推敲他的需要，如天气转凉了，「冷」「热」「穿衣服」等…，但这并不能完全表达患者的需求，因此常造成无法满足患者需求的情况，造成生活上非常的不方便。

Cases 1-5 who presented with spastic paraparesis and other prominent upper motor neurone signs showed high signal on T1-weighted images in the centrum semiovale, corona radiata and posterior limb of the internal capsule which corresponded to the large myelinated fibres of the corticospinal tract Fig.

病例1~5患者表现为下肢痉挛性轻瘫和其他一些明显的上运动神经元症状体征，在T1像上于半卵圆中心、放射冠、内囊后肢处的皮质脊髓束大有髓纤维走行区均可见高信号病灶（图1），在T2、PdWI像上也可见自放射冠延伸至大脑脚的高信号病灶。

Results We found firstly that the SCVM was innervated by both somatic motor neurons of ventral group and Onuf's nucleus in ventral horn of the spinal cord,which is similar as perineal muscles,that means double innervation.

我们的实验发现了骶尾腹内侧肌的神经分布与会阴肌相同，即受脊髓腹侧角运动神经元腹侧群和Onuf核的双重神经支配，至今尚未见国内外有报道。

Spinal muscular atrophy is the name given to a group of inherited disease characterized by muscle weakness and wasting cause of motor neurones defect.

脊髓性肌肉萎缩症是一群因运动神经元退化而造成肌肉萎缩、无力的遗传性疾病。

No p ulp fibers with poisoning and extend significantly reduced. Spinal motor neurons can be seen Golgi body expansion accompanied by Schwann cell swelling.

无髓纤维数量随中毒时间延长而明显减少；脊髓前角运动神经元可见高尔基氏体扩张，伴有雪旺氏细胞肿胀。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。