脂质体的

Objective: This paper aims to verify the curative effect that Jiusuyu cure the acute alcoholism disease by counting the changes of ebriety time, ebriety rate, sober-up time death rate, and discuss the effect of neurotransmitter and oxidative damage on acute alcoholism mice in brain tissue by examining the changes of SOD, MDA, DA, 5-HT, AchE in btain tissue.

目的：本研究旨在通过观察急性酒精中毒小鼠模型醉酒时间、醒酒时间、醉酒率、死亡率来验证酒速愈的药效，并通过检测脑组织中超氧化物歧化酶、丙二醛，纹状体多巴胺、5-羟色胺(5-HT)和海马胆碱脂酶指标的变化探讨该药对急性酒精中毒小鼠脑神经递质和氧化损伤的影响。

As antioxidants, the vitamin E or quercetin (10μg/mL) significantly attenuated the toxic effects of PCBs on the cultured hepatocytes. The above results indicated that vitamin E or quercetin might antagonize the toxic effect of PCBs via antioxidizing effects on in vitro cultured chicken embryo hepatocytes.

结果表明离体培养的鸡胚肝细胞可作为肝损伤的细胞模型，PCB对培养的肝细胞的损伤及抗氧化剂维生素E和槲皮素对它的缓解作用提示， PCB对肝细胞的损伤可能是通过诱导细胞膜发生脂质过氧化而导致细胞的死亡。

In vivo, nuclear Ser-P-STAT1 was found in macrophage-rich regions of advanced murine and human atheromata.

在体研究表明，在小鼠或人的脂质斑块中，核丝氨酸磷酸化的STAT1在富含巨噬细胞的区域被发现。

The effect of various preservative flukd with or without antagonists at 15℃ and 4℃ on the ultrastructure of fetal rat brain tissue in vitro hak been observed. The levels of oxygen free radical and liperoxide were also analysed by measuring SOD and MDA.

观察了室温与4℃条件下，含钙拮抗剂和不含钙拮抗剂的不同保存液对胎鼠脑组织超微结构的影响；并利用超氧物岐化酶和丙二醛为指标，观察了不同保存液保存的离体胎鼠脑组织中自由基和脂质过氧化水平的变化。

M. penetrans can stimulate the production of NO and inhibit mouse macrophages prolification, thus it may be an important etiological factor.

穿透支原体的脂质相关膜蛋白能诱导巨噬细胞产生一氧化氮，且能抑制巨噬细胞的增殖，因而可能与其致病性相关。

Results M. penetrans LAMPs stimulated mouse macrophages to produce nitric oxide in dose-and time-dependent manner. And M. penetrans LAMPs inhibited cell prolification in mouse macrophages.

结果 穿透支原体脂质相关膜蛋白能以时间和剂量依赖方式刺激小鼠巨噬细胞产生NO，并且对小鼠巨噬细胞的增殖有一定的抑制作用。

Aβ at concentration of 10 to 100 μM caused an increase in reactive oxygen species generation and the stimulatory effect was dose dependent.

当贝他类淀粉蛋白浓度由10 μM一直提升到100 μM所产生活性氧化物的量是随著浓度呈现剂量效应，但是却无法在溶小体上产生显著的脂质过氧化效应。

Alcohol, the most common cause, is a hepatotoxin that interferes with mitochondrial and microsomal function in hepatocytes, leading to an accumulation of lipid.

乙醇是最常见的病因，作为肝毒素能阻碍肝细胞内的线粒体和微粒体的功能，导致脂质沉集。

The effects of phenolic acid compounds in vegetables and fruits on metabolicactivation of BaP in rats,as well as 〓 and TCA induced lipid peroxidation inmouse and Salmonella typhimurium TA104 cells were investigated using DNAadducts as a biomarker to assess DNA damage induced by xenobiotic carcinogens.BaP was used to study the genotoxic carcinogenesis,while 〓 and TCA were usedto study the non-genotoxic carcinogenesis.The possible protection mechanisms ofphenolic acid compounds on DNA damage were studied.

本研究根据化学致癌过程的遗传毒性和非遗传毒性机理，以DNA加合物作为机体接触外来化学物而导致DNA损伤的生物学标志物，通过观察酚酸类化合物对大鼠体内BaP活化代谢以及〓和TCA诱导小鼠肝微粒体脂质过氧化的影响，及酚酸类化合物对BaP和TCA诱导的沙门氏菌TA100和TA104形成DNA加合物的影响，探讨蔬菜、水果中酚酸类化合物对外来化学物诱导的DNA损伤的保护机制。

Objective To investigate the inhibitory effects of tea polyphenols and Vitamin C on FeSO4-cysteine induced lipid peroxidation in isolated human plasma and carbon tetrachloride(CCl4) induced liver free radical injury in mice.

目的 观察不同剂量的茶多酚、维生素C对FeSO4-半胱氨酸诱发离体人血浆脂质过氧化及CCl4诱发肝自由基损伤的抑制作用。

The labia have now been sutured together almost completely.The drains and the Foley catheter come out at the top.

此刻阴唇已经几乎完全的缝在一起了，排除多余淤血体液的管子和Foley导管从顶端冒出来。

To get the business done, I suggest we split the difference in price.

为了做成这笔生意，我建议我们在价格上大家各让一半。