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胸腺细胞

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AIM: To study the changes of mitochondrial membrane potential in mouse thymocytes stimulated by dexamethasone.

目的:研究地塞米松刺激小鼠胸腺细胞过程中线粒体膜电势的变化。

Purpose To set up the model for thymocytes development study in vitro.

目的 建立胸腺细胞体外研究模型,为T细胞早期发育及相关实验提供有效技术平台。

After 36 hours of transportation, the expression rate of GFP in thymocytes reachedmore than 90%, while in liver cells and spleen cells the rate was less than 10%.

体外转染后36h,GFP在胸腺细胞中的表达率达到90%以上,脾、肝细胞中的表达率则低于10%。

Objective To investigate the effect of FK506 on cell-surface antigen expression in porcine thymocytes.

目的 探讨他克莫司(FK506)对猪胸腺细胞表面抗原表达的影响。

CONCLUSION摘要: The supplementation of nucleotides can accelerate the repair of damaged DNA in mouse thymocytes in vitro.

结论摘要:外源核苷酸能明显促进受损小鼠胸腺细胞DNA的修复,且核苷酸的功能和其添加水平有关。

The supplementation of nucleotides can accelerate the repair of damaged DNA in mouse thymocytes in vitro.

外源核苷酸能明显促进受损小鼠胸腺细胞DNA的修复,且核苷酸的作用与其添加水平有关。

The changes of caspase- 3 protein and the expression of p38MAPK proteins were detected by western blotting at 0,30,60 and 180 min after dexamethasone- induced thymocytes apoptosis.

运用 Western blotting技术检测地塞米松处理的 Balb/c小鼠胸腺细胞 0, 30, 60, 180 min时相点 caspase- 3蛋白与 p38MAPK的表达变化。

Results The results showed that Cd might cause time-and dose-dependent apoptosis in primary cultured thymocytes.

结果镉能引起原代培养的胸腺细胞发生凋亡,并呈时间-反应和剂量-效应关系。

The immunoreactive nerve fibers for every peptide were seen, and all of them contacted closely with the thymocytes.

结果:各神经肽的免疫反应性主要位于髓质,均可见到阳性神经纤维,并与胸腺细胞密切接触。

Aim To investigate the gene regulation effect during the thymocytes' apoptosis induced by candida albicans.

目的 探讨白色念珠菌诱导小鼠胸腺细胞凋亡过程中的基因调控作用。

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This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。

There is no differences of cell proliferation vitality between labeled and unlabeled NSCs.

双标记神经干细胞的增殖、分化活力与未标记神经干细胞相比无改变。