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胶质细胞

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The response of GFAP positive-astrocytes peaked at 24 h, and then decreased. Conclusions Neurons and astrocytes in Nucleus Tractus Solitarii were activated sequently after acute cardiac injury, which suggested that the involvement neurons and astrocytes of NTS in the central modulation of cardiac pain after ACI.

急性心肌损伤后,大鼠延髓NTS中神经元和星形胶质细胞被心肌伤害性刺激激活,参与心肌损伤后疼痛的中枢调控。

The response of Fos-positive neurons peaked at 4 h, then decreased.4h after acute cardiac injury, the influoence gray scale of GFAP positive-astrocytes significantly increased in Nucleus Tractus Solitarii.

急性心肌损伤后4 h GFAP阳性星形胶质细胞荧光强度明显增强,24 h达到高峰然后下降。

BHLH repressors suppress bHLH activators and promote maintenance of progenitors and generation of glial cells.

抑制型bHLH转录因子促进视网膜干细胞状态的维持和出生后视网膜胶质细胞的分化。

Following trauma to the brain significant changes occur in both the astroglial and vascular components of the neuropil.

伴随脑损伤会出现星形胶质细胞和神经纤维网血管性成分的显著变化。

The differentiated neurons have dendrites and there are synapses between their dendrites and the processes of others. Growth cones and myelinated fibers can be seen in neuropil.

关于孕期为18d胎鼠大脑皮质的神经元、胶质细胞、血曾和突触的分化发育状况,我们已经做了研究'。,本实验试图通过对El。

Two-chamber shared culture medium can promote NSCs to differentiate into oligodendrocytes.

共培养液培养促进神经干细胞向少突胶质细胞分化。

The conclusions based on the results are as follows:TF was found in gliomas of all gradings and its expression was significantly enhanced with the increase of glioma gradings.

根据实验结果得出如下结论:1、TF在各级胶质细胞瘤中均有表达,随着肿瘤组织学分级的增加,TF的表达明显增强。

Statistical data of TF grading scores showed that its expression was not significantly related to the size and location of glioma, sex, and age of patients, but closely related to the glioma gradings and microvessel quantity of the tumor.VEGF was found in gliomas of all gradings and its expression was significantly enhanced with the increase of glioma gradings.

TF分级计数统计结果显示其表达与肿瘤的大小、部位及患者性别、发病年龄等临床因素无显著相关性,但与肿瘤的分级及肿瘤微血管数量密切相关。2、VEGF在各级胶质细胞瘤组织中均有表达,随着肿瘤组织学分级的增加,VEGF的表达明显增强。

Methods A model of decompression sickness in rat was reproduced by exposure to 1MPa 5.5min followed by rapid decompression (50s).

采用1MPa暴露5.5min、50s快速减压制备SD大鼠中枢减压损伤模型,在减压后6、24、48、72h取材,分别用FITC标记的凝集素B4(IB4)标记小胶质细胞和原位末端TUNEL。

However, the mechanisms and the source of ATP release from astrocytes remain to be established.

然而,星型胶质细胞释放ATP的途径和机理还有待研究。

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