胶质细胞

There was an obvious increase of insulin receptor gene and protein in the cerebral ischemia reperfusion tissues in rats. The positive cells such as neuron, neuroglia cells, vascular endothelium cells possibly participated in regulating the processes of energy metabolism of cells and neovascularization. It implied that the high expressions of insulin receptors in vascular endothelium cells in ischemic zones possibly participated in regulating the pathophysiological changes of the blood vessels in ischemic zones; The higher expressions of insulin receptors in different cells were possibly one of the self-protective machanisms in which the ischemic tissues confront the anoxemia injury.

脑缺血再灌注大鼠脑组织胰岛素受体基因和蛋白水平表达增加；半暗带区神经元、部分神经胶质细胞及血管内皮细胞等阳染细胞可能参与脑缺血后组织细胞的能量代谢、促新生细胞形成等病理生理调节过程；胰岛素受体在缺血区血管内皮细胞高表达提示其可能在缺血区血管的病理生理变化中起某种调节作用；胰岛素受体在不同细胞中表达增高可能是缺血脑组织对抗缺血缺氧损伤的自身保护机制之一。

Following brain stem injury,the number of GFAP-positive astrocytes in cerebral cortex and pervent-ricular area,dorsomedial and ventro-medial pafts of midbrain at 30 min antemortem,and in periventricular area,dorsomedial part of pens,dorso-medial and ventro-medial part of medulla oblongata at 60 min antemortem.

采取针刺法造成实验大白鼠脑干损伤，用免疫组织化学LSAB法检测大脑、中脑、桥脑及延髓等不同部位脑组织中胶质原纤维酸性蛋白的改变。结果发现，生前损伤30min，大脑顶部灰质及中脑腹例中央部的GFAP阳性星形胶质细胞数目增多；生前损伤60min，除大脑顶部灰质及中脑腹侧中央部外。

Objective To explore influence of the push-pull G on glial fibrillary acidic proteinexpression in rat brain astrocytes.

目的观察推拉动作后大鼠脑星形胶质细胞中胶质纤维酸性蛋白表达的改变。

Methods The rat embryonic spinal cord astrocytes were cultured and identified by cellular morphology and glial fibrillary acidic protein.

体外原代培养大鼠脊髓星形胶质细胞并鉴定（胶质原纤维酸性蛋白GFAP阳性）。

Central nervous system ; Astrocyte ; Glial fibrillary acidic protein ; Brain injured

中枢神经系统；星形胶质细胞；胶质纤维酸性蛋白；脑损伤

Objective: To study the expressions of microtubule associated protein-2 (MAP-2) and glial fibrillary acidic protein in hippocampus of rats with depression after burn.

目的：探讨烧伤后慢性应激大鼠海马神经元微管相关蛋白－2(MAP-2)和神经胶质细胞胶质纤维酸性蛋白表达的变化。

Against glioma in support of the central nervous system of glial cells.

神经胶质瘤侵害支持中枢神经系统的神经胶质细胞。

If want to changed the stat, must be operatived decrease secondary injury, limited proliferation rate of neuroglial cell, promoted neuron regeneration,after spinal cord injury.

只有有效的降低脊髓损伤后的继发性损害、限制神经胶质细胞的增生速度、促经神经元的再生、才能防止脊髓坏死空洞和胶质瘢痕的形成。

In the experimental study section, the rats were divided into four groups, including the normal control, the model, the Piracetam, the QPC, and copied to AD model. After the treatment, we observed the effects on the AD rats' ability of leaning and memorizing, detecting the TChE activity in whole blood and the expression levels of the related biochemical indicators of As includeing GFAP、NGF and BDNF of the AD rats' brain hippocampus, then compared the QPC with other groups to reveal the mechanism of action of the QPC treating AD to bright.

在动物实验部分，我们通过塑造AD动物模型，设立正常对照组、模型组、脑复康组和强脾益智胶囊组，观察强脾益智胶囊对AD模型大鼠空间学习记忆能力的影响，检测AD大鼠全血、海马匀浆液中乙酰胆碱酯酶活性和通过免疫组化的方法检测胶质纤维酸性蛋白、神经生长因子、和脑源性神经营养因子的表达情况来反映星形胶质细胞的增值情况，以期揭示强脾益智胶囊对AD的治疗作用机理。

Statistical data of VEGF grading scores showed that its expression was not significantly related to the size and location of glioma, sex, and age of patients, but closely related to the glioma gradings. The expression of TF was positively correlated with that of VEGF, suggesting that TF might participate in the anigogenesis of gliomas, and provide proper environment for the growth of gliomas through its coagulant effect.

根据VEGF分级计数统计学分析，显示VEGF表达与肿瘤的大小、部位及患者性别、发病年龄等临床因素无显著相关性，而与肿瘤的组织学分级密切相关。3、胶质细胞瘤中TF与VEGF的表达正相关，TF可能参与了肿瘤血管的生成过程，并通过其促凝血活性作用为胶质瘤的生长创造适宜的内环境。

In the negative and interrogative forms, of course, this is identical to the non-emphatic forms.

。但是，在否定句或疑问句里，这种带有"do"的方法表达的效果却没有什么强调的意思。

Go down on one's knees;kneel down

屈膝跪下。。。下跪祈祷