胶质

Neural stem cells are capable of both self-renewal and differentiation into neurons and glial cells.

神经干细胞具有分化为神经元、星形胶质细胞、少突胶质细胞的能力。

Finally, when the gliomatous component is very bland and I cannot decide, I would just diagnose it as a grade I~II ganglioglioma.

最后，如果这些胶质瘤成分非常温和而我不能判定时，我一般诊断为Ⅰ-Ⅱ级节细胞胶质瘤。

Certainly, one has to see a gliomatous component to call a neuronal tumor a ganglioglioma.

当然，必须看到胶质瘤成分才能诊断一个神经肿瘤为节细胞胶质瘤，这一判断非常主观--&亡斧疑邻&。

Certainly, one has to see a gliomatous component to call a neuronal tumor a ganglioglioma. This judgment can be very subjective -"all in the eyes of the beholder".

当然，必须看到胶质瘤成分才能诊断一个神经肿瘤为节细胞胶质瘤，这一判断非常主观--&亡斧疑邻&。

In addition, the inventive compounds can reduce the inflammatory component of diseases that affect the nervous system, for example, by reducing activation of the microglia and/or astrocytes and/or by reducing reactive gliosis.

另外，根据本发明的化合物特别通过减少小胶质细胞和/或星形细胞的活化和/或通过减少反应性的神经胶质增生而能够减少影响神经系统的疾病的炎性物质。

Gliosis plays a critical role in post-traumatic epileptogenesis. The inhibition of the proliferation of astrocytes with 7β OHCH could influence synaptic plasticity, modulate the imbalance of excitatory and inhibitory systems in hippocampus and postpone the latency between injury and the occurrence of chronic spontaneous seizures, furthermore to some extent, reduce the occurrence of post-traumatic epilepsy.

星形胶质细胞增生在创伤性癫痫发病机制中起重要作用，抑制胶质细胞增生，可影响海马突触可塑性，有利于调整海马内兴奋性和抑制性功能系统之间作用的失衡，可延长损伤与发生慢性自发性抽搐之间的间歇期，在一定程度上降低创伤性癫痫的发病率。

Result bFGF immunoreactivity was found in neuron and gli al cell in corte x,hippocampus habenular nucleus,and found in macrophage surrounding hematoma; Ln immunoreactivity was found in neurons,glial cells and blood vessles in front a l cortex,hippocampus,thalamus hypothalamus.

结果 脑出血后脑内bFGF主要在扣带皮质、海马、缰核及其他区域表达，阳性细胞骨折主要有神经哮喘元、神经哮喘胶质、吞噬细胞骨折、室管膜细胞骨折、伸长细胞骨折等；Ln不仅在大脑皮质、海马、缰核、下丘脑等区域的神经哮喘元表达，且在这些区域的神经哮喘胶质细胞骨折也表达，血管也有阳性反应。

RESULTS A variable degree of vascular hyalinization and glioma intercellular with enhanced tenascin immunostaining was observed.Expression of tenascin and cell proliferation in gliomas correlated positively with each other and increased with the degree of tumor malignancy.

结果 胶质瘤与其增生血管内皮细胞均表达Tenascin，Tenascin基因表达与肿瘤细胞增殖指数呈显著正相关，随胶质瘤恶性程度提高，Tenascin和Ki-67表达均增强。

Combined detection of VEGF、COX-2 and iNOS may helpful to evaluate prognosis and infiltrative capability of glioma.

联合检测VEGF、COX-2和iNOS可能有助于胶质瘤侵袭发展能力的评估，也可作为判断人脑胶质瘤病理生物学行为的客观指标。

Tumor mass could express CD133 and nestin. The karyoplasmic ratio of CD133+ brain glioma stem cells reached 2/3-3/4, and there were a few processes, a few organelles and a plenty of ribosome. These were accorded with the characteristics of stem cells.

肿瘤球能同时表达干细胞标志物CD133和巢蛋白，CD133+脑胶质瘤干细胞核浆比例达2/3~3/4，突起少，胞浆中线粒体等细胞器较少，核糖体丰富，未见胶质丝等分化结构，符合干细胞超微结构特点。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。