胶原质

Immunohistochemical staining with an anti-type II collagen antibody demonstrated rich production of the type II collagen in the pericellular matrix from the chondrocytes.

用抗II型胶原抗体免疫组织化学染色证明在软骨细胞周围的基质中有丰富的II型胶原产生。

Collagen Filler Lip smoothes away lines around the lips with a daily boost of collagen and plumps the lips with hyaluronic micro-spheres.

胶原填充唇部平滑线以外各地的嘴唇每日增加胶原和plumps嘴唇与透明质酸微球。

We have researched distribution of TGF β and its receptor in various age group and discs of various degeneration grade, found that positive ratio of TGF β and its receptor is higher in below 40 year old group and discs of MRI Ⅰ and Ⅱ grade, mainly occur in fibrous cartilagous cells of fibrous annulus and nucleus pulpous; In group of 40-60 years old and grade Ⅱ and Ⅲ discs, TGF β decreased and TGF β R decreased not as much as TGF β; In above 60 years old group, TGF β and its receptor almost disappear.

生长因子包括TGF-β对细胞功能有重要调节作用，它是一种相对分子质量为2.5×10〓的二聚体多肽，几乎参与了哺乳动物所有细胞的病理，生理过程，尤其是在细胞外基质的产生及调控方面意义重大，且TGF-β已被公认为是与胶原代谢关系最密切的细胞因子。胡有谷、陈宏等，应用杂交技术检测了TGF-β对椎间盘Ⅰ、Ⅲ型胶原MRA的调节作用。

In carcinomatous mucosa, immunoreaction of the carinomatous cell nest was negative, or only remnants of positively were seen at the site of the former membrane by collagen type Ⅳ.

结果 正常组织的基底膜呈现很强的Ⅳ型胶原抗体阳性反应，在基底膜形成较粗的环形染色带；Ⅲ型胶原抗体阳性反应在细胞外间质呈连续的细丝纤维样。

Results In increasing dose group, the death rate and the heart weight were increased but the cardiac function were decreased.

结果增量感染组小鼠死亡率持续增高，心脏重量增加，心功能下降，心肌胶原容积分数和血清sIL-2R含量高于其它各组（P〈0.05），病理学特征为基质胶原明显增生重建。

Some cell dropped into the cavity and became free. Thrombosis or part organization could be seen. The internal elastic layer became thin, disappear or broken. In internal and middle layer existed fibroblasts, fibrocytes and collagen. Some of the wall indicated hyaline change, soomth muscle cell decreased greatly. The massive inflammatory cells invaded the middle and external layer. There were many foam cells in the capsule tissue. Cytoplasm was filled with fatty tissue and cholesterol. some cavities were full of thrombosis. Some thrombosis was fibrosis, the bottom was organization. The surface of the thrombosis existed red blood cell and librae.(2)Elatic fibrila staining: the internal elastic menbrane almost completely disappeared, the intact internal elastic menbran could be seen in the new small vessels.

动脉瘤管壁厚薄明显不均，全层或局部区域显著变薄向外膨出，内皮细胞空泡变性或坏死脱落，部分内皮细胞剥离并突入管腔成游离状，可见血栓形成及部分血栓机化；内弹力板变薄、消失或突然中断；在内膜及中膜部位主要为纤维母细胞、纤维细胞和大片胶原；部分动脉瘤壁呈均质状玻璃样变，平滑肌细胞明显减少；中膜和外膜可见大量的炎性细胞浸润；瘤壁组织有纤维母细胞、纤维细胞、大片胶原成分及较多泡沫细胞，胞浆内充满脂类物质及胆固醇结晶；部分动脉瘤腔内充满血栓，有的血拴已经纤维化，血栓基部机化，血栓表面有红细胞和纤维素。

Methods:Serum levels of hyaluronan, laminin, type Ⅲ procollage n and type Ⅳ collagen were assayed in 83 patients with viral hepa titis and liver biopsy was carried out in 62 of them.

病毒性肝炎患者83例，测定其血清透明质酸、层粘连蛋白、Ⅲ型前胶原和Ⅳ型胶原含量，其中62例同时作肝组织病理检查。

We show that the matrix-modifying hormone relaxin increased tumor-associated fibroblast interaction with collagen fibers by stimulating b1-integrin activity, which is necessary for fiber remodeling by matrix metalloproteinases.

我们指出基质修改激素松弛肽通过分泌活性b1-integrin ，从而增加了瘤相关纤维原细胞与胶原纤维的相互作用。这对于基质金属蛋白酶对纤维的重塑很重要。

The results demonstrated that exogenous type II collagen indeed induced the re-expression of type II collagen and aggrecan mRNAs, and glycosaminoglycan levels.

研究结果证明，细胞外间质分子之第二型胶原蛋白可刺激已去分化之软骨细胞再度分泌第二型胶原蛋白及聚葡萄糖胺。

The major feature of fibrosis is the excess deposition of ECM, which includes collagen, non-collagen glucoprotein and proteoglycans.

当纤维化发生时，ECM的量和质均发生显著变化，其中胶原量可较正常增加3—8倍，并且主要以Ⅰ型胶原增加为主。

They weren't aggressive, but I yelled and threw a rock in their direction to get them off the trail and away from me, just in case.

他们没有侵略性，但我大喊，并在他们的方向扔石头让他们过的线索，远离我，以防万一。

In slot 2 in your bag put wrapping paper, quantity does not matter in this case.

在你的书包里槽2把包装纸、数量无关紧要。