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胶原

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Desalinate the wrinkle, collagen induction, stimulate the growth of cellular tissue, skin thickness problems, improve skin scar and pregnancy veins.

消除皱纹,诱发胶原蛋白的产生,刺激组织细胞生长,皮肤粗糙的问题,改善皮肤伤疤和妊娠纹。

Storage temperature of 22°C exclusively enhancedadenosine diphosphate- and collagen-induced aggregation comparedwith storage at 37°C.

存储温度22°C 相较37°C 而言是唯一可以增强二磷酸腺苷和胶原诱导的凝集反应。

Results The eyeball distorted, intraocular structure disordered and vision lost severely in 3 eyes. Epithelial plugs were found in superficial layer of stroma and Bowman's layer broke and displaced in three cases. Healing and gap of incision presented 29 days after RK in 1 case. There were incision scar, collagen disarrangement and epithelial island in deep incision more than 2 years following RK in two cases, descent's layer broke in 1 case.

结果3例眼球严重变形,眼内结构紊乱,视力丧失。3例角膜浅层切口有上皮栓形成,前弹力层断裂错位;1例术后29d的切口部分愈合部分缝隙存在;2例术后2a以上的切口瘢痕化,胶原纤维排列紊乱,深层切口有上皮岛;1例后弹力层断裂。

In lens-induced experimental myopias of guinea pigs, increasing of MMP-2 and decreasing of TIMP-2 occur in posterior scleral matrix. This pair of disbalance of MMP-2 and TIMP-2 can cause abnormal growth of guinea pig's eyeball.

透镜诱导豚鼠实验性近视眼巩膜基质中MMP-2活性增强,TIMP-2活性降低,二者平衡失调可导致巩膜胶原的降解,巩膜壁变薄,引起眼球扩张,眼轴延长。

Efficacy: it can effectively discompose melanin, inhibit tyrosinase activity, cut off root of melanin generation, deep whiten and nourish skin, remove sallow and dull skin, present whitening and crystalline skin, resist oxidation and remove free radical, promote substrate cells'regeneration and splitting, promote elastic fiber and collagen fiber growth, and resume skin elasticity and luster.

功效:有效分解黑色素,抑制酪氨酸酶的活性,切断黑色素生成的根源,深层美白营养肌肤,去除黄气暗哑,绽放肌肤净白莹采,抗氧化清除自由基,刺激基底层细胞再生分裂,促进弹力纤维和胶原纤维生长,重现肌肤弹性和光泽,玫瑰精油源源不断供给肌肤充足的水分和营养,深度改善细纹和干燥现象,改善肤色,给肌肤前所未有的白皙亮泽。

Objective: To investigate in vitro release characteristics of collagen double layer minipellet of erythropoietin.

目的: 对红细胞生成素胶原蛋白双层微条的体外释放特性进行研究。

Cortical neurons had well-preserved morphological state and normal electrophysiological properties after dura mater was treated by the enzyme.

从细胞内记录和胞内染色的结果可以看出,用胶原酶软化硬脑膜对皮层神经元的功能和形态均无影响。

Dystrophic epidermolysis bullosa is a group of genetic disease caused by loss-of-function mutations in the type Ⅶ collagen gene. The clinical phenotype is characterized by the blistering and scaring of the skin from trauma and there is increased complication of squamous cell carcinoma, which is the major cause of death in patients with DEB.

营养不良性大疱性表皮松解症是一种由于人体Ⅶ型胶原基因突变或缺失所引起的遗传性疾病,临床特征为创伤诱发的水疱和瘢痕,并发症以恶性鳞状细胞癌多见,是导致患者死亡的主要因素,目前仍无有效的治疗方法。

All 7 cases were characterized by deposition of dystrophic or psammatous calcifications accompanied with chronic inflammatory cells infiltration in the stroma. In some cases, there was formation of germinal centers.

特徵性形态学表现为在胶原化的纤维组织间可见散在的钙化灶或砂砾小体,间质内伴有多少不等的淋巴细胞和浆细胞浸润灶,部分病例中可见生发中心形成。

Here, we report on the first systematic long-term study of fibroblast therapy in a mouse model for recessive dystrophic epidermolysis bullosa, a severe skin-blistering disorder caused by loss-of-function of collagen VII.

我们报道了第一例长期性系统性的关于成纤维细胞治疗隐性营养不良性大疱性表皮松解症小鼠模型的研究。隐性营养不良性大疱性表皮松解症是一种严重的由于VII-胶原蛋白功能丧失引起的出疱性皮肤病。

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In the negative and interrogative forms, of course, this is identical to the non-emphatic forms.

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