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胶原

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Objective To research the influence of sun-exposure and ages to elastics and collagens of aging skins.

目的:探讨光曝露情况及年龄因素对皮肤中弹性组织、Ⅰ/Ⅲ型胶原及基底膜Ⅳ型胶原的影响,进而探索皱纹形成的组织学原因。

Patented Matrixyl 盿nd type 3 of collagen of skin. Activate fibrocytes to create more collagen that will make your skin look younger and suppler.

专利成分 Matrixyl 可强化第一型、第三型胶原蛋白,可激活纤维母细胞合成更多的胶原蛋白,使肌肤有弹性。

Patented Matrixyl formula could strengthen the type1and type 3 of collagen of skin. Activate fibrocytes to create more collagen that will make your skin look younger and suppler.

专利成分 Matrixyl 可强化第一型、第三型胶原蛋白,可激活纤维母细胞合成更多的胶原蛋白,使肌肤有弹性。

Matrixyl could strengthen the type1 and type3 of collagen of skin, and then activate fibrocytes by creating more collagen. Therefore, Matrixyl will improve the loose skin and wrinkles at the same time. Make your skin look younger and suppler.

Matrixyl 是专门针对抗皱所设计的专利配方,可使第一型、第三型的胶原蛋白更健全具有支撑力,并激活真皮层中纤维母细胞,促进胶原蛋白增生,改善细纹与肌肤松弛的问题。

The wound surface formates ability to reduce collagenically and decompose collagenically excessively inside, become the difficult important reason to heal of wound surface.

创面内胶原合成能力降低和胶原分解过度,成为创面难愈合的重要原因。

The freezing temperature and crosslinking process are two important factors to form collagen scaffolds.

交联处理以及胶原蛋白支架形成过程中的温度控制,是制造胶原蛋白支架的重要控制因素。

Results The expression of Collagen Ⅳ can be seen in the basement membrane of glomeruli, renal tubules and collecting ducts.

国内外学者对Ⅳ型胶原在肾组织中表达的研究显示,在肾脏发生发育过程中Ⅳ型胶原以一种复杂的动态方式表达于基膜[4]。

The reaction mechanism of tetrakis hydroxymethyl phosphonium chloride and raw collagen were researched by the simulated materials of collagen including glycin,ethylenediamine and hide powder.

通过研究四羟甲基氯化鏻与皮胶原模拟物反应,间接证明THPC主要与胶原蛋白的氨基结合,可与氨基以n∶n=1∶2.2形成交联,THPC与羟基和酰胺基均有少量结合,并在pH=8.0时结合量最高。

Taiwan fans Ya salt to green collagen skin care products, and create local brand goods dependent; in recent years, collagen, skin care products has always been a popular domestic topic.

台塩以绿迷雅胶原蛋白保养品,创造本土养品的第一品牌;近年来胶原蛋白,一直是国内保养品流行的话题。

Some cell dropped into the cavity and became free. Thrombosis or part organization could be seen. The internal elastic layer became thin, disappear or broken. In internal and middle layer existed fibroblasts, fibrocytes and collagen. Some of the wall indicated hyaline change, soomth muscle cell decreased greatly. The massive inflammatory cells invaded the middle and external layer. There were many foam cells in the capsule tissue. Cytoplasm was filled with fatty tissue and cholesterol. some cavities were full of thrombosis. Some thrombosis was fibrosis, the bottom was organization. The surface of the thrombosis existed red blood cell and librae.(2)Elatic fibrila staining: the internal elastic menbrane almost completely disappeared, the intact internal elastic menbran could be seen in the new small vessels.

动脉瘤管壁厚薄明显不均,全层或局部区域显著变薄向外膨出,内皮细胞空泡变性或坏死脱落,部分内皮细胞剥离并突入管腔成游离状,可见血栓形成及部分血栓机化;内弹力板变薄、消失或突然中断;在内膜及中膜部位主要为纤维母细胞、纤维细胞和大片胶原;部分动脉瘤壁呈均质状玻璃样变,平滑肌细胞明显减少;中膜和外膜可见大量的炎性细胞浸润;瘤壁组织有纤维母细胞、纤维细胞、大片胶原成分及较多泡沫细胞,胞浆内充满脂类物质及胆固醇结晶;部分动脉瘤腔内充满血栓,有的血拴已经纤维化,血栓基部机化,血栓表面有红细胞和纤维素。

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