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The microstructure and ultrastructure of heart,liver,spleen,lung,kidney,pancreas,fabricius' burse and cerebrum of Muscovy ducks infected with MPV were observed.

应用光镜和电镜技术研究了人工感染雏番鸭细小病毒病死鸭的心、肝、脾、肺、肾、胰、法氏囊和大脑等8种器官的显微结构和发病鸭的心、肝、脾、肺、肾、胰等6种器官的超微结构变化。

Results According to CT findings,they were divided into edematous type (n=46) and necrotic type (n=42).Forteen cases were complicated with infection.Swelling in 75 cases,exudation in 68 cases,necrosis in 42 cases,splenomegaly in 62 cases.

结果 水肿型46例,坏死型42例,其中合并感染14例,均有较典型的CT表现,胰腺肿胀75例,胰周渗液68例,胰腺坏死42例,脾大62例。

Pancreatic leak was verified in 8 out of 13 cases with pancreatic enucleation, 6 out of 10 cases with distal pancreatectomy with or without splenectomy and I out of 6 cases with central pancreatectomy.

摘除术13例中8例发生胰瘘,胰体尾脾切除术8例中6例发生胰痰,保留脾的胰体尾切除术2例均未发生胰瘘,中段胰腺切除术6例中有1例发生胰瘘。

The histopathological changes of inoculated duckling were observed.The result showed that the death peak was in 24-48 h with the mortality of 80%.The histopathological changes were hemorrhagic necrotic hepatitis,necrotic splenitis,pancreas limitation coagulation necrosis and heterophil infiltrating into renal leaflet,respectively.

结果表明,接毒后 2 4~ 48h为感染鸭死亡高峰,试验发病的死亡率为 80 %;感染雏鸭肝、脾、胰、肾的组织病理变化分别表现为出血性、坏死性肝炎,坏死性脾炎,胰局灶性坏死及肾小叶的异嗜性粒细胞浸润。

Uva Ursi is believed to provide an antiseptic effect on the tissues of the kidneys and urinary tract system.

用於肾及膀胱感染、肾结石、糖尿病及痔疮,强化心肌、减低脾、肝、胰、小肠的病变。

Results 33 cases of CTPV were all showed collateral veins, which were biliary collateral veins of the portal vein (n=33), gastric collateral veins (n=25), pancreaticoduodenal region collateral veins (n=14), parumbilical collateral veins (n=2). The collateral veins were appeared as soft tissue density on precontrast scan, with generally no enhancement on arterial phase, but on venous phase, they were appeared as multiple punctuate, strip, nodous or thin reticulate enhanced vessels, which were with tortuous, abnormally dilated, and cavernous in shape.

结果 33例全部显示侧支静脉,其中显示33例胆支、25例胃支、14例胰十二指肠后上静脉和2例脐旁静脉,侧支静脉CT平扫表现为软组织密度影,动脉期一般无强化,门静脉期显示多发点状、条状、结节状或细网状的强化血管影,走行扭曲,异常扩张,呈海绵样改变;22例显示门静脉栓子,14例显示肝脏灌注异常,12例显示胆系改变,25例显示脾肿大。

The display rate was 100% for peri-pancreatic big veins, including splenic vein, superior mesenteric vein, and portal vein; 85.3% and 77.3% for small veins of pancreatic head, including ante-superior pancreaticoduodenal vein, gasticocolonal trunk and its branches.

对胰腺周围大静脉(脾静脉,肠系膜上静脉,门静脉主干)的显示率为100%;胰头部小静脉(胰十二指肠上前静脉,胃结肠干及其属支)的显示率为85.3%和77.3%。

Results The frequency of multislice CT to display peri-pancreatic big arteries including celiac trunk, common hepatic artery splenic artery, gastroduodenal artery and superior mesenteric artery was 100% while that of small arteries including dorsal pancreatic artery, transverse pancreatic artery, pancreaticomegana artery, caudal pancreatic artery, superior pancreaticoduodenal artery and inferior pancreaticoduodenal artery was

结果 多层螺旋CT对正常胰腺周围大动脉(腹腔干,肝总动脉,脾动脉,胃十二指肠动脉,肠系膜上动脉)的显示率为100%,对小动脉(胰背动脉,胰横动脉,胰大动脉,胰尾动脉,胰十二指肠上动脉,胰十二指肠下动脉)的显示率为

Objective To sum up the etiology and the experience on diagnosis and treatment for pancreatogenic portal hypertension complicated with hemorrhage of upper digestive tract in severe acute pancreatitis.

胰源性门静脉高压症是一种因脾静脉回流受阻所致的&区域性门静脉高压症&,在重症急性胰腺炎后期合并消化道出血中,胰源性门静脉高压是较少见和容易忽视的病因。

Yi TAI Compounded Medication "we used bases on the basic theory of TCM,set up "The treatment of invigoratmg spleen and promoting blood by regulating qi",target defiency in origin and excess in superficiality TCM syndrome nature,and take experiments;our research injecting L-Arg into abdomen to induce Spleeh-asthenis rat model,it is a new model which combine diseases and syndromes;immunohistochemic 、 optical mirror and biochemistry value number index number are used to detect the expression of related constrain factor TGF-β_1 mRNA、α-SMA when textnre fabrosis occurs in pancreatic,as nell as significance and relation of hydroxyproline in the process of pancreatic fibrosis,meanwhile observe the disturbace which "Yi TAI Compounded Medication makes,revel the anti-fibrosis mechanism from organization form and molecular level.

本研究采用L—精氨酸分次腹腔注射诱导脾虚型慢性胰腺炎大鼠模型,是一种病证结合的新模型;用免疫组化、光镜等技术和生化指标检测CP时胰实质纤维化的相关调控因素TGF-β_1、α-SMA的表达和血清中羟脯氨酸在胰腺纤维化过程中的作用及相互关系,同时观察了&胰泰复方&对其干预机理,从组织形态学和分子水平揭示了该方抗纤维化的作用机制。

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