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American Atlanta conference edited in September 1992 pancreatitis of serious illness acute definition is: AP companion has local complication (cyst of pancreatic and necrotic, false sex, pancreatic abscess) or the organ fails (function of shock, lung not complete, kidney declines, gastrorrhagia, sow medicinal powder the cruor inside sexual blood-vessel and serious metabolization obstacle), or both holds concurrently.

概要: 急性胰腺炎为临床常见急腹症,是由于胰液和溶蛋白酶漏入到胰腺实质和胰腺四周组织而引起。1963年,法国马赛会议以病理形态学为基础,将AP分为水肿性胰腺炎和出血、坏死性胰腺炎。1992年9月美国亚特兰大会议修订重症急性胰腺炎定义为:AP伴有局部并发症(胰腺坏死、假性囊肿、胰腺脓肿)或器官衰竭(休克、肺功能不全、肾衰、胃出血、播散性血管内凝血和严重的代谢障碍),或二者兼有。

Pancreatic and biliary duct fistula are the most severe and common complication following pancreatoduodenectomy. To prevent this complication, anastomosis should be appropriately performed and drainage of the pancreatic and bile duct is crucial.

胰瘘和胆瘘是胰十二指肠切除术后最常见和最严重的并发症,为预防其发生,除吻合方法的不断改进外,胰液及胆汁的引流是预防胰、胆瘘的关键。

Results Intravenous 5-HT caused obvious exciting effect on spontaneous afferent discharge of subdiaphragmatic vagus nerve.

结论5-HT可能是作用于与迷走传入相关的5-HT3受体兴奋了膈下迷走传入神经自发放电;促胰液素可以增强5-HT对迷走神经传入自发放电的兴奋作用。

The effects of 5-HT, 5-HT3 receptor antagonist and 5-HT + SEC on the discharge of subdiaphragmatic vagus nerve were investigated by intravenous injection of different dosage of5-HT(3, 10, 30 μg/kg), Granisetron (1 mg/kg) and 5-HT + SEC.

结果5-HT对膈下迷走神经传入自发放电具有兴奋作用,给予5-HT3受体拮抗剂后神经放电的效应被抑制,大剂量5-羟色胺+促胰液素可增强由单独的大剂量5-HT所致的迷走神经传入放电的兴奋作用。

Fat tissue is hydrolyzed and released fatty acid as well triolein by lipase; on the other hand, high fat diet can't be directly absorbed across the intestinal mucosa.it must be firstly decomposed into one 2-monoglyceride and two free fatty acids by pancreatic lipase ,which is delivered into the lumen of the gut as a constituent of pancreatic juice.

脂肪酶能够使体内的多余脂肪组织分解成三酸甘油酯,另一方面,人体摄入高脂肪食物,并不能直接被小肠粘膜吸收,而是先被小肠内的胰脂肪酶分解成2-单酸甘油酯和两个游离脂肪酸,然后和胰液一起被运输到肠道的内腔被吸收。

Results In pancreatic cancer patients the positive rate of plasma K - ms gene mutation was 73 %, without false positivity, and higher than that in the pancreatic juice and duodenal juice, but lower than that in the fine - needle aspirates. Plasma K - ras gene mutation was not found in patients with benign pancreatic disease and healthy subjects.

结果 胰腺癌患者血浆K-Fas基因的突变阳性率为73%,无假阳性率,高于胰液、十二指肠液的检测率,低于细针穿刺的检测率;胰腺良性病变患者及健康对照者血浆中未检测到K-ras基因的突变;胰腺癌组K-ras基因突变与非胰腺癌组及健康对照组相比具有显著性差别。

The acute hemorrhage necrosis pancreatitis is one kind critically ill, the condition is heavy, the prognosis bad pancreatic gland acute inflammation pathological change, as a result of the ductus pancreaticus obstruction, is accompanied by the duodenum either the biliary duct contents regurgitation adds on the pancreatic gland blood circulation insufficiency or the nervous reason, causes the pancreatic juice secretion evacuation barrier, the reversion permeating pancreatic gland essence, causes this sickness primary cause.

急性出血坏死型胰腺炎是一种起病急、病情重、预后差的胰腺急性炎症病变,由于胰管梗阻,伴有十二指肠或胆道内容物反流加上胰腺血运不足或神经性原因,导致胰液分泌排空障碍,逆向渗入胰腺实质,是引起本病的主要原因。

The acute hemorrhage necrosis pancreatitis is one kind critically ill, the condition is heavy, the prognosis bad pancreatic gland acute inflammation pathological change, as a result of the ductus pancreaticus obstruction, is accompanied by the duodenum either the biliary duct contents regurgitation adds on the pancreatic gland blood circulation insufficiency or the nervous reason, causes the pancreatic juice secretion evacuation barrier, the reversion permeating pancreatic gland essence, causes this sickness primary cause.

本文章共2291字,分2页,当前第1页,快速翻页:摘要:急性出血坏死型胰腺炎是一种起病急、病情重、预后差的胰腺急性炎症病变,由于胰管梗阻,伴有十二指肠或胆道内容物反流加上胰腺血运不足或神经性原因,导致胰液分泌排空障碍,逆向渗入胰腺实质,是引起本病的主要原因。

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