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VOICE: I remember from anatomy and physiology classes, that the pancreas secretes

我记得在解剖学和生理学上,腺分泌高血糖素和岛素。

The injured gill cells had a different ultrastructure, with endoplasmic reticulum dilated and vesiculated, mitochondrial cristaes partially disintegrated, gill fibre cuticles denatured and dropped. The ultrastructural damages to hepatopancreatic cells were characterized by the decrease of microvilli, swelling and breaking up of mitochondria, expansion of endoplasmic reticulum, disruption and vacuolization of nucleus, tumefaction of nuclear membrane and increase of lipid droplets. As for the toxicological changes of myocardial cells, the mitochondria were swelling, having a lot of vacuolus and disintegrating, while the myofibrils were irregular with endoplasmic reticulum dissolved.

鳃细胞的损害表现为鳃丝水肿,细胞器溶解,角质层变薄,线粒体、内质网的肿胀、解体;肝脏细胞的主要损害特征为肝管微绒毛减少,线粒体水肿解体,内质网扩张,细胞核空泡化,核膜水肿和脂肪滴增加;心脏细胞的毒理变化为线粒体内嵴肿胀、瓦解,肌原纤维不规则,内质网溶解。

The result shew, Chromatin broke into pieces which dispersed around the irregular nucleus, nuclear outer member disintegrated; Mitochondria condensed, cristae of which disintegrated partly; The endoplasmic reticulum dilated, vesiculated, gradually disintegrated; A great number of dense granules appeared in the cell. To sum up: Enrichment of heavy metals in Eriocheir sinensis, Macrobrachium nipponease, Cipangopaludina cathayensis is obviou""

综上所述:我们首次证明上海地区沼虾、河蟹、园田螺对重金属元素富集能力要明显强于淡水鱼类;肝、鳃是鱼、虾、蟹体内重金属富集的主要部位;Hg和Cr为高富集元素,Cd是中富集元素,Cu和Pb为低富集元素;罗氏沼虾对镉离子和锌离子的96小时的半致死浓度(LC50)分别为0.115mg/L和0.68mg/L;首次证明锌离子、GSH在一定范围内可以抵抗镉离子对罗氏沼虾肝脏破坏。

The acute hemorrhage necrosis pancreatitis is one kind critically ill, the condition is heavy, the prognosis bad pancreatic gland acute inflammation pathological change, as a result of the ductus pancreaticus obstruction, is accompanied by the duodenum either the biliary duct contents regurgitation adds on the pancreatic gland blood circulation insufficiency or the nervous reason, causes the pancreatic juice secretion evacuation barrier, the reversion permeating pancreatic gland essence, causes this sickness primary cause.

急性出血坏死型腺炎是一种起病急、病情重、预后差的腺急性炎症病变,由于管梗阻,伴有十二指肠或胆道内容物反流加上腺血运不足或神经性原因,导致液分泌排空障碍,逆向渗入腺实质,是引起本病的主要原因。

The acute hemorrhage necrosis pancreatitis is one kind critically ill, the condition is heavy, the prognosis bad pancreatic gland acute inflammation pathological change, as a result of the ductus pancreaticus obstruction, is accompanied by the duodenum either the biliary duct contents regurgitation adds on the pancreatic gland blood circulation insufficiency or the nervous reason, causes the pancreatic juice secretion evacuation barrier, the reversion permeating pancreatic gland essence, causes this sickness primary cause.

本文章共2291字,分2页,当前第1页,快速翻页:摘要:急性出血坏死型腺炎是一种起病急、病情重、预后差的腺急性炎症病变,由于管梗阻,伴有十二指肠或胆道内容物反流加上腺血运不足或神经性原因,导致液分泌排空障碍,逆向渗入腺实质,是引起本病的主要原因。

The patien ts of SAP combined with acute respiratory distress syndrome, shock, pancr eatic brain diseases, abdominal cavity hemorrhage and systemic infections had hi gh mortality of 52%, 40%, 40.63%, 66.67% and 73.68 respectively. Conclusions The incidence of complications of SAP in perioperat ive period is high and the complications are closely related to pateints' death, especially ARDS, shock, pancrestic brain diseases, abdominal cavity hemorrhage and systemic infections.

急性重症腺炎病情复杂、外损害严重,手术前后并发症多,是导致急性重症腺炎患者手术后病情加重,死亡率增加,预后不佳的主要原因,现将西南医院全军肝胆外科研究所1994年1月~2004年1月10年间总共行了外科手术治疗的116例SAP回顾分析,分析SAP围手术期并发症与预后的关系,以提高外科手术治疗的效果。1资料和方法

Compared to the respectively alone stress, cold + 5 km hypoxia (4 hours/day, continuation for 2 days) and restraint + 5 km hypoxia combinated stresses significantly inhibited body weight gain of rats, and the five stresses also markedly decreased hepatic glycogen contents, and increased plasma glucagon levels. 2. Effect of continual hypoxia on the level of insulin secretion Compared to the sea level normoxia control group, simulated continual 2 km hypoxia for 1-25 days (1, 2, 5, 10,15 and 25 days) did not significantly influence rats body weight, food intake and plasma insulin level.

连续性低氧对岛素分泌水平的影响与海平面常氧对照组相比,模拟2km连续低氧1-25天(1、2、5、10、15和25天)没有明显影响大鼠体重变化、摄食量和血浆岛素水平,但在低氧第2天显著升高肝糖原含量和血糖水平,在第10天明显升高血浆高血糖素水平,在第1天血浆皮质酮水平显著升高,在第1和第2天瘦素水平明显上升,然而在第5天瘦素水平显著下降。

Four goats (42.4±3.5 kg) surgically fitted with cannulas in common bile duct and abomasal were used in 4×4 Latin square design and randomly fed diets contained 20%, 30%, 40% or 50% of starch to determine the effects of rumen escape starch, by altering dietary starch concentration, onα-amylase secretion in goats. Increasing rumen escape starch results in a quadratic increase in pancreaticα-amylase secretion.

活体分泌试验表明,日喂3次情况下,胆汁分泌量和α-淀粉酶分泌活性呈非匀速分泌,在饲喂前后1h内最高(P.05);腺α-淀粉酶总分泌活性随随过瘤胃淀粉数量的提高呈二次曲线变化(P.05),过瘤胃淀粉为102g/d时腺α-淀粉酶总分泌量达到最大。

METHODS: Umbilical cord blood samples were sterilely isolated using Percoll density gradient centrifugation to harvest intermediate layer cells. DMEM medium containing fetal bovine serum, penicillin, streptomycin and L-glutamine was added. Following several adherences and purification, the floating cells were discarded. Thus, many adherent cells with a confluence were collected. When cells were 60%-80% confluent, cells were digested by trypsin for subculture.

无菌条件下,应用Percoll密度梯度离心法分离脐血标本,收获中间层细胞,加入含胎牛血清、青霉素和链霉素、L-谷氨酰胺的DMEM基础培养液,再经反复贴壁纯化,除去悬浮生长细胞,得到较多呈融合状态的贴壁细胞,待细胞达60%~80%融合时酶消化传代。

Objective The preventive effect of cimetidine on post- ERCP pancreatitis and hyperamylasemia was investigated.

目的评价西咪替丁对经内镜逆行胆管造影术后高淀粉酶血症及急性腺炎的预防作用。

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