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For these years, residua of metanephric blastema and delay of differentiation in the development of kidney have been considered the cause of nephroblastoma.

近年来的研究发现,肾发育过程中的胚基残留,分化障碍可能是肾母细胞瘤发病的原因之一。

Symptoms inquring,nasal respiratory,X ray,CTand MRI images were no valuable for the diagosis of chondroma.Their diagnosis was made based on pathohistology.The chondroma were often damaged in the most of the cases.Suryery is an effective method on chondroma.

鼻窦鼻咽部软骨瘤病因不明,多数学者认为可能是来源于异位的软骨胚芽及头颅软骨遗基的残余,如果只靠症状及鼻腔检查、X线摄片、 CT及MRI扫描一般不能诊断本病,病理是诊断软骨瘤的金指标,治疗则以手术为主。

With tile ovary maturing, the orifice was narrowed because of tile ovary growth at last fused completely. The gynoecium is composed of a single carpel.(1) In tile series developmental sections of ovary, the ovular primordium was initiated on the adaxial meristem when tile mouth of ovary was formed.

子房由1枚心皮构成;(4)从子房发育过程的切片看,该植物的胚珠是在子房发生后不久发生的,子房上的圆孔形成时,从近轴侧的分生组织发生胚珠原基,由胚珠原基分化出珠被与珠心。

It also indicates that the expression of WT1 is regulated strictly in time and space, and the aberrant expression of WT1 may result in retarded differentiation of blastemal cell leading NR or nephroblastoma.

提示在散发性肾母细胞瘤发生中除WT1突变外,WT1表达延迟可能是其中起重要作用的机制之一;WT1基因与肾母细胞瘤的组织分化有关,可能诱导胚基细胞向上皮细胞方向分化。

In the normal uterus, Cytokeratins immunolabelling were detected in glandular cell, luminal epithelial cell, Vimentin immunolabelling were detected in stromal cell and endoblastic cell; CK7 immunolabelling were not detected in any tissue of the yak utenus.

研究结果显示:未妊娠时,泛角蛋白在子宫内膜腺上皮细胞、腔上皮细胞内表达,波形蛋白在子宫内膜基质细胞内表达,平滑肌肌动蛋白在子宫平滑肌和血管平滑肌内表达,牦牛子宫任何部位均不表达角蛋白7;妊娠30天左右时,泛角蛋白在子宫内膜腺上皮细胞、子宫内膜腔上皮细胞、滋养层细胞、内胚层细胞和尿囊细胞内表达,波形蛋白在子宫内膜基质细胞和内胚层细胞内表达,平滑肌肌动蛋白在子宫平滑肌和血管平滑肌内表达,角蛋白7在尿囊细胞内表达,偶尔在腔上皮细胞的细胞核边缘表达;消化法进行原代培养时,组织经胶原酶消化并通过100目和400目筛网组合可以有效地分离原代子宫内膜基质细胞和子宫内膜腺上皮细胞;分离得到的子宫内膜基质细胞活率达90%以上,并可在体外传代7次以上;分离得到的子宫内膜腺上皮细胞活率可达85%以上,并可在体外传代5次以上;RPMI1640培养基最适合子宫内膜基质细胞和子宫内膜腺上皮细胞的生长,维持子宫内膜基质细胞正常生长的FBS添加量为20%,维持子宫内膜腺上皮细胞正常生长的胎牛血清添加量为30%。

Leaves several, lorate or linear, cross section semiterete or terete, solid or fistulose, base usually not narrowed into a petiole; ovules 2 to several per locule; if leaf base narrowed into a petiole or ovules 1 per locule then bulb tunic never distinctl

叶数个,带状或线形,横断面半圆柱形或圆柱状,中空的固体或,基部通常不缩小成为叶柄;胚珠2到数枚每室;如果叶基部狭窄成为叶柄或胚珠1每室那么鳞茎皮从未是distinctl 7

Results Only unstable random mutations happened in 29 sites with nonsynonymous vs synonymous mutation ratio of 1.42 during 50 passages in 2 serials without maternal antibodies.

LG1株H9N2在没有抗体的鸡胚的传代过程中,仅发生少数碱基的不稳定随机变异,且多为无义突变。

These were raised underinducing condition to test if any morphology and structure changes. Twenty two Arabidopsismutants had various changes in morphology and anatomical structures. Twenty mutants hadlow germination rate from 10% to 60%, of which 2 had survival rates at 0% and 50%. Fivemutants occurred structure changes in the hypocotyls or stem. One of these lines, namedarris-stem, showed some unique changes: slower growth rate in comparison with the wildtype from germination to florescence; serrated margin of leaf blades, spiral rosette; morebranch in the bottom part of the stem, shorter nodes, twisted stem and branch. There were oneor several arrises along the stem. Across sections of the arrises showed one or several compactcells lumps which were round, made up of several layers of cells, looked like vascular-bundle.

在诱导条件下,这些突变体表现出不同的形态和结构的变化,共有22个表型和结构发生变化。20个突变系发芽率或存活率较低,发芽率由10%—60%,其中2个突变系存活率分别为0%和50%。5个突变系在茎或下胚轴的结构上发生变化,其中一个突变系arris-stem发生如下表型变化:从真叶出现至开花结实的整个生长期,生长速度要明显比野生型拟南芥缓慢;叶缘有明显锯齿,莲座叶呈螺旋状排列;茎的基部有较多侧枝,侧枝间距明显缩短,茎有明显扭曲;突变体茎侧面有1至数条棱形突起,内部存在一至数个排列紧密的细胞团,由多层细胞呈环形排列,细胞壁明显加厚,内部存在管状分子,推测为维管组织。

On the termination date, the cultured explants were all examined by Western blot, HE and transmission electron microscope. Our results showed that after 12-days in culture, the cultivation treated with AS-ODN reduced the synthesis of AMBN and had a deformed dental cusp with thinner enamel matrix. Ultrastructure analyses showed that there was hardly any cisternae of the rough endoplasmic reticulum in the ameloblasts at the tip of the cusp of AS-ODN treatedexplants. However, on average the enamel matrix was thinner compared with that in the control group. Furthermore, the collagen fibers in extracellular matrix were found disorganized. These findings seemed to provide a direct experimental evidence that tended to indicate that the arrested AMBN translation in cultured tooth germs might result in the delay of the tooth development.

经用Western蛋白印迹检测表明,所设计的反义核酸对AMBN InRNA具有良好的封闭效果并成功阻断了牙胚对AMBN的表达;在缺乏AMBN情况下,与对照组相比,实验组牙胚在体外可以继续生长发育至钟状晚期,出现成釉细胞和成牙本质细胞的分化,成釉细胞可以分化成为分泌期型成釉细胞,胞浆中缺少合成蛋白质所必需的粗面内质网和高尔基氏体,缺乏溶酶体,表明对蛋白合成和脚的能力降低;实验组牙胚有牙尖形成和基质分泌,但牙尖形态异常,基质形成减少,牙尖周围基质最厚处为O.6卜m,明显薄于对照组的5.spin,基质中胶原纤维粗细不等,排列稀疏, 3 第四军医大学硕士学位论文未见钙化现象,充分证明了AMBN在牙胚发育中参与釉质基质形成和矿化过程,影响胶原纤维和牙本质基质的合成,促进成釉细胞对蛋白质的合成和釉质基质蛋白降解。

Results The expression of endogenous LN was found in the basement membrane of epithelium, glands and blood vessels in uterine endometrium of both estrus and early pregnancy. And its was faint that LN present in basement membrane as the implantation occurs, but the LN immunostaining in the epithelial cells of endometrium was stronger. The exogenous LN has no obvious influence on blastocyst attachment and implantation. LN-antibody intra-uterine injection showed significant repressing effect on mouse blastocyst implantation.

结果 动情期及妊娠早期小鼠子宫内膜内源性LN在上皮和腺基膜及血管内皮基膜均有表达,并随着植入的发生基膜的LN表达减弱,而内膜上皮中LN免疫染色逐渐增强;外源性LN对小鼠胚泡粘附与植入子宫内膜不产生明显影响,层粘连蛋白抗体明显抑制小鼠胚泡着床。

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Finally, according to market conditions and market products this article paper analyzes the trends in the development of camera technology, and designs a color night vision camera.

最后根据市场情况和市面上产品的情况分析了摄像机技术的发展趋势,并设计了一款彩色夜视摄像机。

Only person height weeds and the fierce looks stone idles were there.

只有半人深的荒草和龇牙咧嘴的神像。

This dramatic range, steeper than the Himalayas, is the upturned rim of the eastern edge of Tibet, a plateau that has risen to 5 km in response to the slow but un stoppable collision of India with Asia that began about 55 million years ago and which continues unabated today.

这一引人注目的地域范围,比喜马拉雅山更加陡峭,是处于西藏东部边缘的朝上翻的边框地带。响应启始于约5500万年前的、缓慢的但却不可阻挡的印度与亚洲地壳板块碰撞,高原已上升至五千米,这种碰撞持续至今,毫无衰退。