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Methods mRNA of placenta from normal pregnancy and PIH were extracted and labeled with flourescent Cy3 and Cy5 in reverse transcription,respectively.

方法从正常妊娠胎盘和PIH胎盘中抽提mRNA,反转录后,分别用Cy3、Cy5荧光标记,获得两组胎盘来源的cDNA探针。

Methods mRNA of placenta from normal pregnancy and PIH were extracted and labeled with flourescent Cy3 and Cy5 in reverse transcription,respectively.The Biodoor chip was hybridized with labeled cDNA.

方法从正常妊娠胎盘和PIH胎盘中抽提mRNA,反转录后,分别用Cy3、Cy5荧光标记,获得两组胎盘来源的cDNA探针。cDNA探针与含8 190条人cDNA点样的BioDoor基因芯片杂交,结果由激光共聚焦扫描仪扫描,并用软件进行统计分析。

The literature was searched for data on the sex ratios of offspring associated with : abruptio placenta , placenta praevia , placenta accreta , extrauterine pregnancy , acute fatty liver of pregnancy or toxaemia of pregnancy .

被搜查的文献资料对后代性别比例的联系:胎盘早期剥离,前置胎盘,胎盘植入,宫外孕,妊娠急性脂肪肝或妊娠毒血症。

The NO content and the NOS activity were down in the PIH pregnant women placenta.This showed that the ghrelin regulated the VEGF and influenced the the NO down.And the placenta angiospasm,blood pressure highten, and the PIH were caused also by this. The ghrelin,VEGF,Flt-1,NO and NOS were detected by the SP-immunochemistry, RQ-PCR,cell culture in vitro,ASODN and nitrate reductase in the normal pregnancy,PIH placenta and the cultured HUVEC antisense blocking ghrelin in vitro.

结果证实:PIH孕妇胎盘组织中Ghrelin,VEGF、Flt-1蛋白和mRNA水平下降与正常孕妇比较有显著性差异p<0.05提示在PIH发病过程中Ghrelin可能对VEGF,Flt-1蛋白和mRNA的表达具有下调作用,进而影响胎盘血管网的形成和发育;NO含量与NOS活力水平在PIH孕妇胎盘组织中下降,提示Ghrelin可能通过下调VEGF的表达进一步影响血管舒张因子NO水平下降,进而引起胎盘血管痉挛,血压升高,最终导致PIH的发生。

The thickening of endangium angiostenosis, fibronoid necrosis and acute arterial atherosis were found at the sites of positive expressions in SPIH.

结果 妊高征组胎盘绒毛和子宫胎盘床血管壁IgA、IgG、IgM和C3的阳性表达率胎盘绒毛:20.1%,24.1%,72.4%和65.5%;胎盘床:20.0%,20.0%,70.0%和60.0%)均分别显著高于对照组(P<0.05,而对照组均呈阴性表达。

P-gp is expressed in normal biliary canaliculi of the liver, the adrenal cortex and proximal tubules of the kidney, and intestinal epithelia including the columnar cells of the large and small intestines; capillary endothelial cells of brain, testis, and placenta; and in the hematopoietic stem cells of bone marrow.

P-gp表达于正常的肝脏胆小管,肾脏的,肾上腺皮质和近端小管,以及包括大肠和小肠的柱状细胞在内的肠上皮细胞;脑、睾丸和胎盘的毛细血管内皮细胞;以及骨髓的造血干细胞。

Their relative molecular weights were 4785 and 4386 respectively. And their absorbance peaks were 210.5 nm and 225.4 nm respectively. The bovine and caprine placental peptides could improve immunosuppressive T lymphocyte transformation rate. The immunosuppressive lymphocyte transformation rates in groups with the bovine placental peptide of 1:1×10^3 and 1:1×10^4 (P.05)and with the caprine placental peptides of 1:1×10^2, 1:1×10^3 and 1:1×10^4 (P.01 )increased more remarkably than that in immunosuppression group.

结果 从牛和羊胎盘中成功提取出了胎盘肽,其相对分子质量分别为4785和4386;其最大吸收峰分别为210.5和225.4nm;牛和羊胎盘肽均可使顺铂抑制的兔外周血T淋巴细胞的转化率显著提高,其中牛胎盘肽以1:l×10^3、1:l×10^4 2组吸收度明显高于抑制组(P.05);羊胎盘肽以1:1×10^2、1:1×10^3和1:1×10^3 3组效果最为明显(P.01)。

MethodsThe distilled bovine and caprine placental peptide s were prepared by ultrafiltering and their relative molecular weights were mensurated by improved Tricine-SDS-PAGE eletrophoresis with urea.

结果从牛和羊胎盘中成功提取出了胎盘肽,其相对分子质量分别为4 785和4 386;其最大吸收峰分别为210.5和225.4 nm;牛和羊胎盘肽均可使顺铂抑制的兔外周血T淋巴细胞的转化率显著提高,其中牛胎盘肽以1∶1×103、1∶1×1042组吸收度明显高于抑制组(P.05);羊胎盘肽以1∶1×102、1∶1×103和1∶1×1043组效果最为明显(P.01)。

Placental factors, such as the placenta dysplasia, a smaller placenta, placental formation Duocheng lobulated, therefore, the fetus through the placenta supplies nutrients bound to be affected.

胎盘方面因素如胎盘发育不良,胎盘较小,胎盘形成多呈分叶状,因此,通过胎盘供应胎儿的营养物质必然受到影响。

Twins: If twin-twin transfusion syndrome, acardius,(4) gestational diabetes mellitus (5) maternal-fetal Rh blood group incompatibility (6) placental factors: such as increasing the placenta, placental chorioangioma 3, B supramaximal amniotic cavity of the vertical depth of the dark zone as the standard.

3双胎:如双胎输血综合症,无心畸形,(4)妊娠糖尿病(5)母儿Rh血型不合(6)胎盘因素:如胎盘增大,胎盘绒毛膜血管瘤3、B超最大羊水腔暗区的垂直深度为标准。

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另一方面,更重要的是由于城市住房是一种异质性产品。

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You rap, you know we are not so good at rapping, huh?

你唱吧,你也知道我们并不那么擅长说唱,对吧?