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胍丁胺

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Agmatine has many functions in central nervous system.

胍丁胺对中枢神经元有多种作用。

Agmatine was found to increase the proliferation of neural stem cell and efaroxan can block the proliferation effect.

胍丁胺可以促进体外培养的新生大鼠海马神经前体细胞的增殖,其促增殖作用可能与咪唑啉Ⅰ(Ⅰ1)受体有关。

Agmatine co-treated with morphine could reverse morphine's regulation on NMDA receptors' expression in these two regions.

胍丁胺与吗啡伴随给药能逆转吗啡对两脑区NMDA受体蛋白表达的调节作用。

The effects of agmatine on the discharges of neurons in CA1 area of hippocampal slices were examined by using extracellular recording technique.

应用细胞外记录单位放电技术,在大鼠海马脑片上观察了胍丁胺对CA1区神经元放电的影响。

AIMTo further elucidate the role of agmatine on the pharmacological effects of opioids.

目的进一步阐明胍丁胺对阿片药理作用的影响。

The mechanism of regulation of agmatine on opioid dependence is related to the reverse effect on the NMDA receptors' level and construction.

胍丁胺调节阿片依赖可能与其逆转吗啡对NMDA受体亚基数量和构成的调节有关。

Efaroxan 10 μmol· L^-1 inhibit proliferation effect of agmatine on neural stem cell.

结果从新生大鼠海马分离得到的细胞可以持续分裂增殖形成细胞克隆;胍丁胺1、10μmol·L^-1可以显著促进神经前体细胞的增殖,并且这种促增殖作用可以被依法克生所抑制。

The effect of agmatine on vascular tension and the underlying receptor mechanism were investigated in the isolated aortic artery of rats.

采用离体血管环灌流方法,观察了胍丁胺(agmatine, Agm)对大鼠胸主动脉张力的影响,并探讨其受体机制。

Present study, therefore, is to directly demonstrate whether I1-imidazoline receptor participates in the modulation to morphine dependence by agmatine, and to discuss its molecular mechanism in signal transduction aspect.

本文 旨在首次提供直接证据以阐明I1-眯唑啉受体是否介导了胍丁胺对吗啡依赖的调节,并探讨I1-咪唑啉受体在受体后水平抑制吗啡依赖的可能分子机制。

In 66 anaesthetized rats, the effects of local injection of agmatine on femoral, renal, and mesenteric vascular beds were investigated respectively by constant flow perfusion method.

在66只麻醉大鼠,分别采用后肢、肾脏和肠系膜动脉在体恒流灌注法,观察了向灌流环路中直接注射胍丁胺(agmatine, AGM)的血管效应,以所引起的灌流压增减反映血管的收缩和舒张。

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交际能力和语法的关系应该是目标与途径的关系。

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这不是纸上谈兵式的交易,这是人与人的业务,而且涉及金额巨大。