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Stone recurrence in 3 years was found in 4 out of 9 cases with cholesterosis,and in 5 out of 8 in which preoperative function of contraction was less than 50% evaluated by oral cholecystography.

胆囊切开取石术适于轻症胆囊结石、胆囊无畸形和胆囊收缩功能良好者,效果较好,但必须严格掌握适应证,并注意恰当操作。

Results: Ultrasonographic manifestations of cholecystoduodenal fistula were gas inside the bile duct and gallladder; indistinction of the wall of gallbudder, gallstone disappeared and small intestinal obstruction.

结果:7例胆囊、二指肠瘘的超声检查表现:胆管和胆囊内积气;胆囊结构消失和原有胆囊内结石消失;小肠梗阻。

Cholesterol gallstones formed in C57L mice and fatty livers developed in AKR mice. Conclusions Biliary cholesterol hypersecretion is the key pathophysiological defect of gallstone formation, lith genes have effects on biliary cholesterol hypersecretion and susceptibility to cholesterol gallstone formation in C57L mice. Lithogenic bile is formed at the canalicular membrane and precedes the development of cholesterol gallstones. It is most likely that cholesterol and bile acid hyposecretion make the AKR strain susceptible to the development of fatty livers and resistant to gallstone formation.

结论胆道胆固醇的高分泌是胆囊胆固醇结石形成的主要病理生理基础,结石基因决定了C57L鼠肝内胆汁中胆固醇的高分泌和胆囊胆固醇结石的易患性,成石胆汁形成在肝内胆管,先于胆囊结石的形成;肝内胆汁中胆固醇和胆酸的低分泌可能与AKR鼠脂肪肝的发生和胆囊结石的免患性有关。

The function of gallbladder has been injured evidently of the cystic polyp patients that the polyp are located at the collum of cholecyst or whose cholecyst have a non-inflammatory pachyntic paries,and the number of stone in cholelifhiasis patients is more then three.or the pachyntic paries more than 0.3 centimeter.

单个结石大于 2cm或小于 1cm但多于 3块或囊壁厚度大于 3cm的患者胆囊的功能明显受损。伴胆囊限局性非炎性肥厚或位于颈管部并伴临床症状的胆囊息肉患者胆囊功能明显受损

Then, perfuse iodine tincture on it and use the gauze to wipe the cyst wall.

选择其中980例,采用提拉牵引法显露胆囊三角,电凝切断胆囊动脉分支,4号丝线结扎胆囊管,三孔法完成胆囊切除,未用钛夹。

Methods The gallbladder patients from july 2004 to july 2005 of our hospital were randomizedly divided into MLC and LC groups. MLC with three ports: one 10-mm umbilical port, one 5-mm subxiphoid port, and one 2-mm port in the right subcostal position.

迷你腹腔镜胆囊切除术采用脐孔10mm、剑突下5mm、右肋缘下2mm三孔法,而常规腹腔镜胆囊切除为脐孔10mm、剑突下10mm,右肋缘下2个5mm四孔法。2004年7月至2005年7月行腹腔镜胆囊切除936例,男349例,女587例胆囊病变病人进行研究。

MLC with three ports: one 10-mm umbilical port, one 5-mm subxiphoid port, and one 2-mm port in the right subcostal position. LC with four ports: one 10-mm umbilical port, one 10-mm subxiphoid port, and two 5-mm port in the right subcostal position.

迷你腹腔镜胆囊切除术采用脐孔10mm、剑突下5mm、右肋缘下2mm三孔法,而常规腹腔镜胆囊切除为脐孔10mm、剑突下10mm,右肋缘下2个5mm四孔法。2004年7月至2005年7月行腹腔镜胆囊切除936例,男349例,女587例胆囊病变病人进行研究。

Objective:To evaluate the part-cholecystetoomy used in some disease of vesicae fellea which difficulty with cholecystetoomy.

目的:探讨胆囊疾病行胆囊切除术有困难时采用胆囊部分切除术的疗效方法:将37例行胆囊部分切者列 A组,并与同期37例随机选取的胆囊全切者进行围手术期比较。

The rupture of the branches of venae hepaticae intermediae intruding beds of gallbladders is the important reason causing the bleeding on beds of gallbladders during cholecystectomize.

结论(1)突入胆囊床的肝中静脉属支破裂是引起胆囊切除时胆囊床大出血的重要原因;(2)在胆囊床由左前下向右后上方缝合肝组织是对突入胆囊床肝中静脉属支破裂出血的有效预防方法。

Gallbladder endoscopicmucosalresectionor open cholecystectomy were applied in 28 cases developed with chronic atrophic cholecystitis and cholecystolithiasis.No hemorrhoea resulted from venae hepaticae intermediae on gallbladder bed injury were occured in all the cases.

术前常规彩色多普勒超声波检查,明确胆囊床肝中静脉及属枝位置关系,对于肝中静脉与胆囊床相贴的病例,采用紧靠胆囊壁的浆肌层剥离,或采用胆囊粘膜切除,或直接开腹手术切除,可以避免因损伤胆囊床肝中静脉而导致大出血。

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