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肿瘤

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It is possible that cancer could be cured radically by specifically killing targeted tumor stem cells and prevented from relapsing and metastasizing.

肿瘤干细胞的提出,使得靶向性杀伤肿瘤干细胞从而使根治肿瘤和防止肿瘤复发和转移成为可能。

There is a rare primary liver tumor that has been reported as ossifying stromal-epithelial tumor (3 cases), desmoplastic nested spindle cell tumor (4 cases), and nested stromal-epithelial tumor (6 cases).

肝脏有一种罕见的原发性肿瘤,先前分别有文献将其报道为骨化性间质-上皮肿瘤(3例)、促纤维组织增生性巢状梭形细胞肿瘤(4例)和巢状间质-上皮肿瘤(6例)。

Among the changes: creating more sub-stages for tumor size, reassigning some large tumors to a more advanced stage, reclassifying tumors that have spread into the fluid surrounding the lung, and recognizing that spread to certain lymph nodes is more dangerous than its spread to others.

方案变动包括,对肿瘤体积进一步分级,将一些大型肿瘤列入晚期,对已扩散到肺部周围液体的肿瘤重新分类,并认定肿瘤扩散至某些特定淋巴结比扩散至其他淋巴结更危险。

Results The red cell C3b receptor rosette(RBCC3bR),natural tumor erythrocyte rosette,direct tumor erythro cyte rosette,enhace tumor erythrocyte rosette,associated tumor erythrocyte rosette and red cell immune adherence enhance factor were sig...

结果宫颈癌患者红细胞C3b受体花环(RBCC3bR)、自然肿瘤红细胞花环、直向肿瘤红细胞花环、促肿瘤红细胞花环、协同肿瘤红细胞花环及红细胞免疫粘附促进因子显著降低(P.001),红细胞免疫复合物花环和红细胞免疫粘附抑制因子显著升高(P.001)。

The result showed HSV seropositive mice was really induced to produce tumor special or non-special factors, possibly included natural killer cell and lymphokine activated killer cell, which cooperated with mtHSV to kill tumor cells in course of mtHSV mediated tumor therapy. The effect in wtHSV seropositive mice was better than in mtHSV seropositive mice.

实验证明HSV免疫小鼠确实诱导产生了肿瘤特异性或非特异性因子,可能包括NK细胞和LAK细胞(lymphokine activated killer cell),这些肿瘤特异性或非特异性因子在mtHSV介导的肿瘤治疗中与mtHSV一起协同杀死了肿瘤细胞,HSV-1F免疫小鼠的这种作用强于mtHSV免疫小鼠。

Much more prolongation ofsurvival term and improvement of survival rate,as well as appearance of a great dealof lymphocytes around pulmonary metastatic nodes and less pulmonary metastaseswere also observed in murines treated with activated、tumor antigen-pulsedmacrophages contrast to those treated with IL-2 gene-transfected macrophageswithout prior pulse of tumor antigen in vitro.

肿瘤抗原冲击的IL-2基因转染巨噬细胞治疗组小鼠期存活明显延长,肺转移结节明显减少,局部病理显示,肺肿瘤周围出现大量淋巴细胞浸润,我们推测这可能是因为抗原冲击的活化巨噬细胞除了增强对肿瘤细胞直接的杀伤效应外,还对其进行处理加工,将免疫信息传递给周围的淋巴细胞,从而引起其增殖活化,并浸润肿瘤组织中。

The expression of vegf in xenograft tumor cells was analyzed by indirect immunofluorescence.

近40年来在肿瘤领域研究的重大进展之一是确立了肿瘤血管生成在肿瘤发展中的重要地位与抗血管生成治疗肿瘤的意义[1,2]。

To investigate the expressions of Fas and FasL on tumor cells and TILs in patients with so as to understand the potential role of Fas system in immune escape and counteratteck involved in HCC,the expression levels of Fas and FasL were detected on tumor cells,noncancorous liver cells,TILs and PBLs in patients with HCC by means of indirect fluorescent immunoassay flow cytometry.

为研究肝细胞癌患者肿瘤细胞、肿瘤浸润淋巴细胞Fas抗原与FasL配体的表达水平,探讨Fas系统在肝细胞癌免疫逃逸及免疫反击中的作用,采用间接免疫荧光流式细胞术检测20例肝细胞癌患者肿瘤细胞、肿瘤浸润淋巴细胞和外周血淋巴细胞Fas与FasL的表达。

These studies would provide theoretical basis and experimental proof in the treatment of tumors with TAMs as therapeutic targets , and would be meaningful for the study of tumor immunity and tumor therapeutics .

本研究为以肿瘤组织巨噬细胞为靶点的肿瘤治疗提供理论基础和实验依据,以期对深入研究肿瘤免疫和肿瘤治疗有一定的意义。

After selecting a highly potent strain of poxvirus that was able to traffic to tumors when administered intravenously to mice the authors engineered the virus such that it would target only specific cancer cells -- those with increased expression of a protein known as E2F and/or activation of signaling downstream of a protein known as EGFR.

以毒攻毒:设计病毒,攻击肿瘤一种新的治疗癌症的方法正在开发中:用病毒感染肿瘤杀死癌细胞留下正常细胞不被伤害。这些病毒以病毒疗法而著称。在一项新的研究当中,David Kirn 和他在旧金山Jennerex生物疗法同事已经描述出利用新病毒疗法后老鼠和兔子抗肿瘤效应的发展。在选择高度特异性痘状病毒后才能有效的到达肿瘤生长地方,然后通过静脉途径给老鼠。

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