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A pulmonary acinus is defined as that portion of lung distal to the terminal bronchiole, and up to 12 acini can make up one secondary pulmonary lobule.

肺腺泡是指肺终末支气管远端的部分,12个腺泡组成一个次级肺小叶。

Unusual findings included lobar consolidation, hilar adenopathy, cystic lesions, pleural effusion and atelectasis, etc.

少见的表现有大叶实变、肺门淋巴结增大、肺囊性病变、胸腔积液及肺不张等。

M{rX*o0 OBJECTIVE: To investigate the effects of neuronal nitric oxide synthase inhibitor 7-nitroindazole (7-NI) on smoke inhalation pulmonary injury in rats. METHODS: Forty healthy male SD rats were randomly divided into three groups: control group (n=8), smoke inhalation model group (n=16) and 7-NI treatment group (n=16). After reproducing the smoke inhalation model, to the 7-NI treatment group rats 7-NI (20 mg/kg in 2 ml arachis oil) was administered by intraperitoneal injection, while in the control group and the model group, 2 ml arachis oil was administered by intraperitoneal injection.

探讨神经型一氧化氮合酶抑制剂7-硝基吲唑(7-NI)在烟雾吸入性肺损伤中的作用。40只SD雄性大鼠被随机分为正常对照组(n=8)、烟雾吸入性肺损伤模型组(n=16)和7-NI治疗组(n=16),建立烟雾吸入性肺损伤模型。7-NI治疗组在致伤后立即腹腔注射7-NI 20 mg/kg(溶于2 ml花生油中);正常对照组及模型组腹腔注射2 ml花生油。

After reproducing the smoke inhalation model, to the 7-NI treatment group rats 7-NI (20 mg/kg in 2 ml arachis oil) was administered by intraperitoneal injection, while in the control group and the model group, 2 ml arachis oil was administered by intraperitoneal injection.

探讨神经型一氧化氮合酶抑制剂7-硝基吲唑(7-NI)在烟雾吸入性肺损伤中的作用。40只SD雄性大鼠被随机分为正常对照组(n=8)、烟雾吸入性肺损伤模型组(n=16)和7-NI治疗组(n=16),建立烟雾吸入性肺损伤模型。7-NI治疗组在致伤后立即腹腔注射7-NI 20 mg/kg(溶于2 ml花生油中);正常对照组及模型组腹腔注射2 ml花生油。

Objective Oxygen toxicity plays a major role in the pathogenesis of bronchopulmonary dysplasiain neonates requiring ventilation.

支气管肺发育不良(bronchopulmonary dysplasia, BPD)是一种因长期用氧,氧毒性使肺组织纤维化及肺萎陷的病变[1]。

The level of serum amylase, the pulmonary dry/wet weight ratio and histological changes were measured at different time points in each group. The expression of pulmonary CINC protein and mRNA were detected by immunohistochemistry and semi-quantitative reverse transcriptase polymerase chain reaction, respectively.

分别检测各组不同时间点血清淀粉酶、肺干湿重比和肺组织病理学改变,用免疫组织化学法和半定量逆转录聚合酶链反应法检测肺组织中CINC蛋白和CINC mRNA表达的变化情况。

Methods Twenty-four patients scheduled for undergoing thoracoscope operation were divided into two groups of 12 each: desflurane group and isoflurane group. Breathe desflurane or isoflurane before and after one-lung ventilation. And change the I/E to 1:1.5. Recorde inhalation concentration, pulmonum concentration, lung compliance and peak of air tube.

24例拟施胸腔镜手术的病人,随机分为两组,地氟醚组(D组n=14)和异氟醚组(I组n=14),分别在单肺通气前后吸入地氟醚或异氟醚15min,其中单肺通气时吸呼比调整为1.0:1.5,连续纪录吸入浓度、肺泡浓度、肺顺应性和气道峰压。

Methods 39 nonencephalophathic and 11 encephalopathic patients with cor pulmonale and 20 controls were assessed simutaneously with VEP and SEP.

5 0例肺源性心脏病患者,其中肺性脑病 11例,非肺性脑病 39例和 2 0例正常者进行了视觉、体表感觉诱发电位的检测。

Pulmonary pneumatocele is a cystic lesion within the lung that usually occurs in associated with the penumonia. It could enlarge to compress the adjacent lung, heart, mediastinum, then to cause respiratory or cardiovascular compromise, and possibly fatal outcome.

肺囊肿是肺内的一个囊状病兆,通常它的发生和肺炎有关,肺囊肿可以慢慢增大到压迫周围的肺、心脏和纵隔腔,造成呼吸或心脏衰竭,甚至可能会致命。

Results Thirty-one(7.64%) of 406 patients showed abnormal ventilatory function, while 165 of 404 patients showed diffusing abnormality. Of the 434 patients who had received HRCT scans,151(34.79%) showed abnormalities including subpleural and basal ground-glass and reticular attenuation,nodular septal thickening and bronchiectasis. Of the 395 patients who had received both lung function test and HRCT scanning,55(13.92%) had abnormalities both in the lung function and HRCT.

结果 在4 0 6例进行肺通气功能检测的患者中,31例(7 6 4 %)异常;4 0 4例进行肺弥散功能检测,其中16 5例(40 84 %)异常;4 34例进行胸部HRCT检查,15 1例(34 79%)异常,表现为磨玻璃密度阴影、网格状阴影和胸膜下弧线影等;395例同时接受肺弥散功能检测和胸部HRCT检查,5 5例(13 92 %)两项均异常,且HRCT显示双肺病变典型者的肺弥散功能较HRCT正常者有显著的下降,两组差异有统计学意义。

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