肺的

Results The displayed number of the segmental and sub-segmental bronchia with 2.5 mm thick slices was more than that of 5 mm and 3.75 mm. There were no differences between 5 mm and 3.75 mm, or 2.5 mm and 1.25 mm slices in the display of them.

结果 2.5mm层厚MDCT显示的肺段及肺亚段支气管数均高于5及3.75mmMDCT；5和3.75mm、2.5和1.25mm层厚MDCT对肺段及肺亚段支气管的显示数无统计学差异。

ASCL2 gene escapes genomic imprinting in skeletal muscle,fat, heart,liver,spleen,lung,kidney,stomach,small intestine,uterus,ovary and pituitary.(2) Both of the transcripts of NNAT gene is paternally expressed in skeletal muscle,fat,heart, liver,spleen,lung,kidney,stomach,small intestine,uterus,ovary and pituitary.(3) DIRAS3 gene is paternally expressed in skeletal muscle,fat,heart,liver,spleen,lung, kidney,stomach,small intestine,uterus,ovary and pituitary.(4) DLX5 gene is maternally expressed in skeletal muscle,fat,spleen,lung,stomach,small intestine,but escapes imprinting in heart,liver,kidney,uterus,ovary,testicle and pituitary.

1ASCL2基因在肌肉、脂肪、心、肝、脾、肺、肾、胃、小肠、子宫、卵巢和垂体中均逃脱印记；(2)NNAT基因两个转录本在肌肉、脂肪、心、肝、脾、肺、肾、胃、小肠、子宫、卵巢和垂体中均为母系印记：(3)DIRAS3基因在肌肉、脂肪、心、肝、脾、肺、肾、胃、小肠、子宫、卵巢和垂体中母系印记：(4)DLX5基因在骨骼肌、脂肪、肺、脾、胃和小肠中表达母本来源的等位基因，即表现为父系印记；而在心、肝、肾、子宫、卵巢、睾丸和垂体中表达双亲来源的等位基因，即逃脱印记。3。

Real time PCR was undertaken to quantitate the AQPs gene mRNA expression.

本文的研究目的为： 1判定AQPs基因是否在羊胎肺中表达以及在其胎肺发育中的表达模式； 2确定AQPs基因在羊肺中的表达分布； 3研究在引起胎肺液体动力改变的生理模型中胎肺AQPs基因表达的变化。

These results suggested that the lung Fib were activated and produced excess HA, resulting in the excessive accumulation of HA within the lung at the stage of alveolitis during the process of pulmonary fibrosis. However, increased HA synthesis of lung Fib and increased HA levels in BALF merely reflect the intensity of alveolitis and the disease activity rather than the degree of pulmonary fibrosis.

以上结果提示：肺Fib在肺纤维化的早期肺泡炎阶段就处于活化状态，产生HA增多，导致肺内HA的过多聚积，肺Fib产生HA增多和BALF中HA水平的增高可反映肺泡炎的强度和疾病的活动性，但并不能反映肺胶原代谢或纤维化的程度。

Rats in LPS group (n = 12) were intratrachealy administered 200 μg of LPS dissolved in 100 μl saline after anesthetization, rats in control group(n = 12 ) were given intratracheal 100 μl saline.

通过检测LPS组大鼠气管注入LPS后第2、4、7天肺组织炎症指标包括肺炎症积分、支气管肺泡灌洗液中性粒细胞计数、巨噬细胞计数、TNF-α、IL-1β及IL-8含量和气道黏液分泌指标包括肺组织阿先蓝—过碘酸雪夫阳染面积、免疫组化黏蛋白Muc5ac及逆转录-聚合酶链反应Muc5ac mRNA的表达，对LPS诱发大鼠肺炎症和气道黏液高分泌的改变进行动态观察。

Objectives To study the expressions of STAT1 and PDGF in lung tissue of pulmonary fibrosis model induced by bleomycin in rats, and discuss the possible mechanism.Methods Thirty Wistar rats were randomly divided into two groups (15 in each group), they were bleomycin group and control group.①The BLM group was intratracheally instilled with belomycin (5mg/kg);② NS group was intratracheally instilled with saline (5mg/kg).

目的 观察肺纤维化鼠肺组织信号转导和转录活化因子1（STAT1）和血小板源性生长因子表达的变化，探讨其在肺纤维化的作用机制方法 30只Wistar大鼠随机分博莱霉素组和对照组，①BLM组：气管内灌注BLM（5mg/kg）诱导肺纤维化；②NS组：气管内灌注NS。

Malondialdehyde production and total superoxide dismutase activity in samples of nondependent lung and dependent lung were detected and analysed in both groups on time of OLV 3hr and OLV 6hr to determine whether long-term OLV led to injury of the nondependent lung and whether the injury would be deteriorated by low-dose ALM.

通过对OLV 3hr及OLV 6hr时两组实验犬无通气肺与通气肺组织标本中丙二醛含量及总超氧化物歧化酶活力的测定，观察长时间OLV是否引起无通气肺组织损伤，并同时探讨小剂量ALM对无通气肺组织损伤的影响。

Model of animals were established by putting rats in a container with slippy inner wall and half-full of water in it, letting them swimming freely till died of being drowned. Each rat from different groups was killed by breaking its neck except drowning group, then BW and SO_2 was detected, and its chest was cut open rapidly, lung was taken out and weight was measured accurately to count lung coefficient. Its lung appearance was observed and lung tissue was fixed with formalin, which was made into pathology slices with HE staining, and pathology change of the lung tissue was observed;Expression of IL-8 and IL-10 in lung tissue was detected with Immunocytochemistry, and correlation between them was analyzed .

动物模型制作采用将大鼠放入盛有黄河水的内壁光滑的容器内，让其自由泳动自然溺死，除溺水致死组外，各组动物分别在规定的时间点断头处死，行体重和血氧饱和度等指标检测，并在冰盘中迅速开胸取肺，准确称重以计算肺系数，观察各组大鼠肺脏外观，取肺组织福尔马林固定，制成病理切片HE染色，光镜下观察肺组织病理改变；用免疫组化的方法检测肺内IL-8、IL-10的表达，并对其相关性进行分析研究。

The prevalence of lung functional impairment in simple pneumoconiosis varies with the parameter measured, and DLco determination may be helpful in case of normal spirometric measurement. Chin J Public Health.

煤矿工单纯尘肺症肺功能障害的盛行率视参数的种类而有所不同，肺弥散量测定可以补充肺量测定之不足。

Result:among 45 patients with AIDS complicated by pulmonary tuberculosis,10 cases with one lung segmental infiltrations,6 cases with 2 lung segmental infiltrations, 4 cases with 3～5 lung segmental infiltrations, 20 cases with diffused lung infiltration , 5 cases with negative lung presentation, among these, 10 cases complicated with PCP , 8 cases with fungus infection, 5cases with bacteria infection confirmed by pathogen test.

结果：45例艾滋病合并肺结核患者，病灶累及1个肺段的10例，2个肺段的6例，3～5个肺段的4例，两肺弥漫性分布的20例，肺部无阳性表现者5例，其中病理证实同时合并卡氏肺孢子虫肺炎10例，合并细菌感染8例，合并真菌感染5例。

In the negative and interrogative forms, of course, this is identical to the non-emphatic forms.

。但是，在否定句或疑问句里，这种带有"do"的方法表达的效果却没有什么强调的意思。

Go down on one's knees;kneel down

屈膝跪下。。。下跪祈祷