肺泡的

Machine of concealed source sex turns a sex pneumonic is one kind a qualitative sex that names with clinical pathology is pneumonic, COP was 1985 Epler [the pathology performance that 1] offers is pulmonary alveolus inside, fine bronchus of sex of alveolar canal, breath and eventually the independent disease that end fine bronchus has granulation organization to form inside antrum is planted, say to send a gender especially again machine of fine bronchitic companion turns out-of-the-way sex the gender is pneumonic , will nearly 20 years report gradually grow in quantity, easy infection, n/med tuberculosis, tumour, as nodal as lung disease, other a disease of qualitative sex lung promiscuous, reached my courtyard to received vivid check of treated bronchus lens lung 2008 2007 now (8 case that TBLB) wins pathology to confirm are analysed as follows.

概要： 隐源性机化性肺炎是一类以临床病理命名的间质性肺炎，COP是1985年Epler等[1]提出的病理表现为肺泡内、肺泡管、呼吸性细支气管及终末细支气管腔内有肉芽组织形成的独立病种，又称特发性闭塞性细支气管炎伴机化性肺炎，近20年来报道逐渐增多，易和肺部感染、结核、肿瘤、结节病、其他间质性肺疾病等混淆，现将我院2007年至2008年收治的支气管镜肺活检获得病理证实的8例病例分析如下。

Methods Cultivation of alveolar type Ⅱ cells were mode from normal adult rat and of silicotic animal model of rat was established.

分离培养正常成年大鼠肺泡Ⅱ型细胞，建立大鼠矽肺模型，用原位杂交和免疫组化技术检测体外培养的和矽肺病变中的肺泡Ⅱ型细胞TGFβ1和PDGF BmRNA和蛋白的表达。

Methods Cultivation of alveolar type Ⅱ cells were mode from normal adult rat and of silicotic animal model of rat was established. In situ hybridization and irnmunohistochemieal staining were applied to detect the gene expression of TGFβ1 and PDGF-B in the alveolar type Ⅱ cells of primary culture and silicotic tissue.

分离培养正常成年大鼠肺泡Ⅱ型细胞，建立大鼠矽肺模型，用原位杂交和免疫组化技术检测体外培养的和矽肺病变中的肺泡Ⅱ型细胞TGFβ1和PDGF-B mRNA和蛋白的表达。

Results For patients who had trouble in preoperative intubation, the success rate was 100% using fiberoptic bronchoscopy.（2）The success rate of taking out the heterobjectives under induction of paediatric and adult bronchoscopy was 100%.（3）Postoperative and burned patients who went through sputum suction and alveolus lavement with fiberoptic bronchoscopy healed quicker.（4）The application of fiberoptic bronchoscopy had saved dyspnea patients＇ life through sputum suction and alveolus lavement.

结果 （1）对于外科手术前插管困难者，利用气管镜进行插管成功率为100％；（2）利用小儿气管镜及成人气管镜进行气管内异物取出成功率为100％；（3）在外科手术后病人及烧伤病人吸痰、肺泡灌洗对病人的愈合缩短了时间；（4）对呼吸困难的病人吸痰、肺泡灌洗解决呼吸道通畅，挽救了病人的生命。

Compared with control group,the ADM level in the left lung increased(P＜0.001),but the ADM levels in the heart,liver,kidney decreased.The ratio of wet to dry weight of the left lung in experiment group was 6.491±0.248,and 5.937±0.361 in control group,there was no significant difference（P＞0.05）.No alveolar wall thicken,alveolar and mesenchyma edema were found in two groups.

实验组兔左肺的湿/干重量比为6.491±0.248，右肺的湿/干重量比为5.937±0.361，双肺的湿/干重量比差异无显著性（P＞0.05）；HE染色结果显示实验组与对照组均未见肺泡壁增厚、间质及肺泡水肿等改变。

In conclusion, the protocol proposed by the Acute Respiratory Distress Syndrome Network, lacking solid physiologic basis, frequently fails to induce alveolar recruitment and may increase the risk of alveolar overinflation.

总之，ARDS Network的实验设计中，缺少有力的生理学依据，不能有效地促进起肺泡复张，甚至增加肺泡过渡通气的风险。

By scanningelectron microscopy,the trachea mucosa infected with PRRSV BJ-4 showed conventionalmorphology but the lung showed marked morphological changes.Alveolar septa were thickenedand pseudopodia were far less evident or absent. Many of PAMs had completely collapsed.

扫描电镜观察发现，PRRSV BJ-4毒株感染的仔猪气管粘膜上皮没有明显的形态学改变，而肺脏则看到肺泡间质明显增宽及肺泡巨噬细胞表面伪足的消失和细胞萎陷。

Methods: Twenty-four rabbits were randomly divided into three groups:① the control group(group C, n=8): received sternotomy only and no ischemia;② the ischemia/reperfusion group(group I/R, n=8): the left lungs of rabbits were rendered ischemia by ligating the left pulmonary hili for 60 minutes followed by 60 minutes reperfusion;③ the edaravone group(group E, n=8): the left lungs were rendered ischemia for 60 minutes followed by 60 minutes reperfusion, and 10 mg/kg edaravone was administered intravenously 5 minutes prior to ischemia. Blood MDA and SOD were measured. Protein concentrations, WBC count, and PMN percentage in broncho-alveolar lavage fluid were determined. Samples of left lung tissue were sent for determining the left lung dry-to-wet weight ratio and evaluating the pathologic changes.

采用兔肺原位热缺血再灌注损伤模型进行研究。24只大白兔随机分为三组：①对照组(C组， n=8)，开胸后不阻断肺门，静脉缓慢推注生理盐水5ml/kg；②缺血再灌注组(I/R组， n=8)，左肺接受60min的缺血，然后接受60min再灌注，于缺血前5min静脉缓慢推注生理盐水5ml/kg；③依达拉奉干预组(E组， n=8)，于缺血前5min静脉缓慢推注依达拉奉10mg/kg（5ml/kg），左肺接受60min的缺血，然后接受60min再灌注。120min实验结束时，留取各组动物血液标本，测定血浆丙二醛含量及超氧化物歧化酶活性；收集支气管肺泡灌洗液，检测支气管肺泡灌洗液中白细胞计数、中性粒细胞百分比及肺通透性指数；留取肺组织标本，测定肺组织湿、干重量，计算干／湿重比及行肺组织病理检查。

Toxic effect of exposure to VTM dust was lower than that to vanadium oxide and silica, but higher than that to titanium dioxide, which had slight toxic effect.

钒钛尘对肺泡巨噬细胞的毒性作用低于V2O5和SiO2，但高于TiO2.TiO2对肺泡巨噬细胞亦有轻微的毒性作用。

VEGF can promote synthesis and secret of pulmonary surfactant protein B; antagonises under-regulation of expression of AEC II receptors by Dexamethasone.

VEGF能促进肺泡发育和肺表面活性物质结合蛋白B的合成、分泌，拮抗地塞米松下调肺泡Ⅱ型细胞受体的表达。

The reasons of iron ions content overproof in grade Ⅱ desalting water system,such as variation water quality,contamination of regenerant , operation adjustment of pretreatment system and switching operation of bed were discussed.

对二级脱盐水系统中铁离子含量超标的原因，如来水水质发生波动、再生剂受到污染、预处理系统操作调整、床体运行切换等进行了论述。

You were hired to drum up new business, so go and do it.

公司雇你招徕新业务，你就做你的事好了。