肺泡的

It were found by comprehensive analysis, the precisive diagnosis rates of the markers from bronchoalveolar lavage fluid were much higher than that from blood, and the markers from alveolar macrophage were much higher than that from broncholaveolar lavage fluid. The precisive diagnosis rates of fibronectin secreted by alveolar macrophage, and fibronectin secreted by alveolar macrophage inhibited by cort and inhibitory rate were 96. 2%, 92. 3%, and 92. 3%, with the diagnosis markers of higher sensibitity and specificity.

综合分析发现，支气管肺灌洗液中指标的正确诊断率高于外周血，而肺泡巨噬细胞中诊断指标又优于支气管肺灌洗液，其中肺泡巨噬细胞产生的纤维结合蛋白、皮质激素抑制肺泡巨噬细胞产生纤维结合蛋白和纤维结合蛋白抑制率均为敏感性和特异性较高的诊断指标，其正确诊断率分别为96.2%、92.3%和92.3%。

The experimental group has various changes in the different density of potentill anserine extract.

实验组不同浓度的蕨麻提取物对小鼠肺组织的影响不同，Ⅰ组小鼠肺组织表面肉眼见不同程度的散在点状出血、显微镜下肺泡腔内见散在红细胞；Ⅱ组小鼠肺组织表面肉眼无明显异常，显微镜下肺泡腔内见散在红细胞及淡伊红色液体，两组均见肺泡壁毛细血管扩张充血及毛细血管内皮细胞呈不同程度的增生肥大。

The experimental group has various changes in the different density of potentill anserine extract. The small rat pulmonary tissue surface ofⅠgroup can see sporadic petechia of different degree; pulmonary alveolus cavity can see sporadic erythrocyte under the microscope. The small rat pulmonary tissue surface of Ⅱ group is no obvious abnormality by naked eye; pulmonary alveolus cavity can see sporadic erythrocyte and thin powder color liquid under the microscope. Two groups all have the capillary vessel of alveolar wall dilation and the endothelial cell hyperplasia and hypertrophy in different degree.

实验组不同浓度的蕨麻提取物对小鼠肺组织的影响不同，Ⅰ组小鼠肺组织表面肉眼见不同程度的散在点状出血、显微镜下肺泡腔内见散在红细胞；Ⅱ组小鼠肺组织表面肉眼无明显异常，显微镜下肺泡腔内见散在红细胞及淡伊红色液体，两组均见肺泡壁毛细血管扩张充血及毛细血管内皮细胞呈不同程度的增生肥。

The experimental group has various changes in the different density of potentill anserine extract. The small rat pulmonary tissue surface ofⅠgroup can see sporadic petechia of different degree; pulmonary alveolus cavity can see sporadic erythrocyte under the microscope.

实验组不同浓度的蕨麻提取物对小鼠肺组织的影响不同，Ⅰ组小鼠肺组织表面肉眼见不同程度的散在点状出血、显微镜下肺泡腔内见散在红细胞；Ⅱ组小鼠肺组织表面肉眼无明显异常，显微镜下肺泡腔内见散在红细胞及淡伊红色液体，两组均见肺泡壁毛细血管扩张充血及毛细血管内皮细胞呈不同程度的增生肥大。

The experimental group has various changes in the different density of potentill anserine extract. The small rat pulmonary tissue surface ofⅠgroup can see sporadic petechia of different degree; pulmonary alveolus cavity can see sporadic erythrocyte under the microscope. The small rat pulmonary tissue surface of Ⅱ group is no obvious abnormality by naked eye; pulmonary alveolus cavity can see sporadic erythrocyte and thin powder color liquid under the microscope. Two groups all have the capillary vessel of alveolar wall dilation and the endothelial cell hyperplasia and hypertrophy in different degree.

实验组不同浓度的蕨麻提取物对小鼠肺组织的影响不同，Ⅰ组小鼠肺组织表面肉眼见不同程度的散在点状出血、显微镜下肺泡腔内见散在红细胞；Ⅱ组小鼠肺组织表面肉眼无明显异常，显微镜下肺泡腔内见散在红细胞及淡伊红色液体，两组均见肺泡壁毛细血管扩张充血及毛细血管内皮细胞呈不同程度的增生肥大。

The results showed that:(1) In COPD group the alveolar walls were broken. The average area of alveolars was 2.46 times as big as the control group, and the average diameter was 1.42 times as compared with the control group. As the diameter of bronchioles was less than 100μm and 100～200μm respectively, the average area of smooth muscle in bronchioles increased to 3.06-fold and 2.13-fold respectively. By electron microscopic, the proportion of Clara cells of respiratory bronchioles in COPD group decreased, the Clara cells were denaturalized, the proportion of Clara cells containing low density secretory granules increased. The airway resistance was 1.84 times while the dynamic compliance was only 70.45% comparing with the control group.

结果表明：(1)光镜下，COPD组大鼠与正常对照组比较，肺泡壁破坏、肺泡腔扩大，平均肺泡面积和平均肺泡直径分别为正常对照组的2.46倍和1.42倍，在直径＜100μm及直径100～200μm的小支气管，COPD组大鼠支气管平滑肌面积分别为正常对照组的3.06倍和2.13倍；电镜下可见，在呼吸性细支气管，Clara细胞占上皮细胞的比例下降，细胞变性，含低电子密度颗粒的Clara细胞比例增加；呼吸功能测定可见COPD组大鼠呼气相气道阻力是正常对照组的1.84倍、动态呼吸系统总顺应性为正常对照组的70.45％。

After the stimulation of LPS, the expression of CXCR4 and the concentrations of TNF-α and IL-6 in culture fluid were detected through the methods of Western Blot, immunohistochemistry and RT-PCR respectively.

在LPS刺激肺泡巨噬细胞后，分别应用ELISA、Western Blot及RT-PCR法检测肺泡巨噬细胞培养液中TNF-a和IL-6浓度以及肺泡巨噬细胞CXCR4蛋白和mRNA表达的变化。

Some exudation and bleedings happen in the pulmonary alveoli, some inflammatory cells infiltrate into lung tissues; in the serum AMY, ALP, TNF-α, IL-6 increases obviously high, COX-2 expression increase; The expression of NF-κB in big rat lung of SAP increase, and the nucleus is thick dyed, it explain that the NF-κ moved into the nucleus; Stimulate the inflamatory reaction.

我们的实验中见到SAP组的大鼠肺脏组织细胞腔扩大、肺泡壁断裂，有的出现肺大泡，肺泡内伴有渗出及少量非出血，肺组织可见咽细胞浸润；血清中AMY、ALP、TNF-α、IL-6明显增高；在SAP组大鼠肝细胞、肾小管上皮细胞、肺脏小气管及肺泡上皮上COX-2表达增加；NF-κB在SAP大鼠肺脏中表达增加，并且出现核浓染，说明NF-κB前移入核；启动炎症反应等一系列反应SAP大鼠受损伤的肺组织中AQP-1无论在mRNA水平上、蛋白水平上还是免疫组化染色病理学检测上都明显下降。

In histopathology sections showed that alveoli were complete and clear with walls being thin and brittle. In experimental group, some petechia were observed in lung samples , the area of fibroblast and fibrinogen in histopathology sections were found , and the ground-glass predominance and reticulation structure in Micro-CT findings were increased during the period from the 1st week to the 3rd week, and degrade in the 4th week after the administration.

结果显示，控制组之肺脏检体色泽正常，没有出血，组织病理学则以I型肺泡细胞为主，无间质性肺炎且肺泡完整清净，肺泡壁薄而完整；实验组方面，第一~三周肺脏检体外观及组织病理的呈现出血点及出血病灶且越趋严重，第四周则消退，且Micro- CT影像评估亦相同。

The cytochemistrialstaining was used to detect the activities of PAMs.The results showed that the activities of ACPaseat the early stage of postinoculation,the activities of glycogen or glycoprotein,ATPase and ANAEin PRRSV-infected PAMs were markedly decreased.

用酶组织化学染色法对感染PRRSV的仔猪肺泡巨噬细胞内的细胞化学成分进行了动态研究，发现PRRSV急性感染早期肺泡巨噬细胞ACPase活性减弱，感染后各时期肺泡巨噬细胞内糖类、ATPase和ANAE活性均减弱。

The reasons of iron ions content overproof in grade Ⅱ desalting water system,such as variation water quality,contamination of regenerant , operation adjustment of pretreatment system and switching operation of bed were discussed.

对二级脱盐水系统中铁离子含量超标的原因，如来水水质发生波动、再生剂受到污染、预处理系统操作调整、床体运行切换等进行了论述。

You were hired to drum up new business, so go and do it.

公司雇你招徕新业务，你就做你的事好了。