肺泡内的

Machine of concealed source sex turns a sex pneumonic is one kind a qualitative sex that names with clinical pathology is pneumonic, COP was 1985 Epler [the pathology performance that 1] offers is pulmonary alveolus inside, fine bronchus of sex of alveolar canal, breath and eventually the independent disease that end fine bronchus has granulation organization to form inside antrum is planted, say to send a gender especially again machine of fine bronchitic companion turns out-of-the-way sex the gender is pneumonic , will nearly 20 years report gradually grow in quantity, easy infection, n/med tuberculosis, tumour, as nodal as lung disease, other a disease of qualitative sex lung promiscuous, reached my courtyard to received vivid check of treated bronchus lens lung 2008 2007 now (8 case that TBLB) wins pathology to confirm are analysed as follows.

概要： 隐源性机化性肺炎是一类以临床病理命名的间质性肺炎，COP是1985年Epler等[1]提出的病理表现为肺泡内、肺泡管、呼吸性细支气管及终末细支气管腔内有肉芽组织形成的独立病种，又称特发性闭塞性细支气管炎伴机化性肺炎，近20年来报道逐渐增多，易和肺部感染、结核、肿瘤、结节病、其他间质性肺疾病等混淆，现将我院2007年至2008年收治的支气管镜肺活检获得病理证实的8例病例分析如下。

The honeycomb vascular networks were various in size and form at a low magnification. The venule connected with the pulmonary capillary networks directly. In some fields of view, the pulmonary alveolar networks were plate-like and sieve-like.

低倍镜下肺实质内的微血管网呈现大小不等的蜂窝状，肺实质内微静脉与肺泡毛细血管之间联系紧密；在局部区域，肺泡毛细血管网可呈现板块状或筛网状。

Intrapulmonary pressure is the pressure in pulmonary alveoli.

肺内压是指肺泡内的压力。

These results suggested that the lung Fib were activated and produced excess HA, resulting in the excessive accumulation of HA within the lung at the stage of alveolitis during the process of pulmonary fibrosis. However, increased HA synthesis of lung Fib and increased HA levels in BALF merely reflect the intensity of alveolitis and the disease activity rather than the degree of pulmonary fibrosis.

以上结果提示：肺Fib在肺纤维化的早期肺泡炎阶段就处于活化状态，产生HA增多，导致肺内HA的过多聚积，肺Fib产生HA增多和BALF中HA水平的增高可反映肺泡炎的强度和疾病的活动性，但并不能反映肺胶原代谢或纤维化的程度。

The main pathological findings include a desquamative interstitial pneumonia-like reaction with intra-alveolar macrophages and numerous large multinucleated histiocytes that ingested inflammatory cells were admixed with macrophages.

GIP主要病理表现有脱屑性间质性肺炎样反应，即在肺泡腔内有巨噬细胞和大量的多核巨细胞聚集，位于肺泡腔内的多核巨细胞内可见被吞噬的炎性细胞，是GIP的特点。

Because of little and not well-distributed suction power of gas molecule to liquid molecule, there is surface tension between the liquid and gas in alveoli(a kind of suction power or concentric force souced from surface).

肺泡内液气界面存在着表面张力（一种表面能来源与分子间的吸引力或内聚力），是由于气体分子对肺泡内面表面液气分子吸引力很小，液体分子各向受力不匀的表现。

Some exudation and bleedings happen in the pulmonary alveoli, some inflammatory cells infiltrate into lung tissues; in the serum AMY, ALP, TNF-α, IL-6 increases obviously high, COX-2 expression increase; The expression of NF-κB in big rat lung of SAP increase, and the nucleus is thick dyed, it explain that the NF-κ moved into the nucleus; Stimulate the inflamatory reaction.

我们的实验中见到SAP组的大鼠肺脏组织细胞腔扩大、肺泡壁断裂，有的出现肺大泡，肺泡内伴有渗出及少量非出血，肺组织可见咽细胞浸润；血清中AMY、ALP、TNF-α、IL-6明显增高；在SAP组大鼠肝细胞、肾小管上皮细胞、肺脏小气管及肺泡上皮上COX-2表达增加；NF-κB在SAP大鼠肺脏中表达增加，并且出现核浓染，说明NF-κB前移入核；启动炎症反应等一系列反应SAP大鼠受损伤的肺组织中AQP-1无论在mRNA水平上、蛋白水平上还是免疫组化染色病理学检测上都明显下降。

The alveolar cavities were made of single-layer capillary networks and the alveolar cavities varied in size. The alveolar cavities were half-hollow spheres, concave to outside and base to inside.

单层毛细血管网围成肺泡腔，腔体大小不一，伸展状态较好的肺泡呈现出凹面向外，底面朝内的半空心球状结构。

The causes of pneumomediastinum in DKA patients may be a result of vomiting or Kussmaul's respiration, where increased intra-alveolar pressure permits air to dissect along the perivascular sheath into the mediastinum.

造成气纵膈的可能原因，酮酸中毒病人会发生呕吐或库氏呼吸(Kussmaul's rcspiration)，此会增加肺泡内压力，使得空气可剥离肺泡血管周围鞘进入纵膈腔内。

The cytochemistrialstaining was used to detect the activities of PAMs.The results showed that the activities of ACPaseat the early stage of postinoculation,the activities of glycogen or glycoprotein,ATPase and ANAEin PRRSV-infected PAMs were markedly decreased.

用酶组织化学染色法对感染PRRSV的仔猪肺泡巨噬细胞内的细胞化学成分进行了动态研究，发现PRRSV急性感染早期肺泡巨噬细胞ACPase活性减弱，感染后各时期肺泡巨噬细胞内糖类、ATPase和ANAE活性均减弱。

The reasons of iron ions content overproof in grade Ⅱ desalting water system,such as variation water quality,contamination of regenerant , operation adjustment of pretreatment system and switching operation of bed were discussed.

对二级脱盐水系统中铁离子含量超标的原因，如来水水质发生波动、再生剂受到污染、预处理系统操作调整、床体运行切换等进行了论述。

You were hired to drum up new business, so go and do it.

公司雇你招徕新业务，你就做你的事好了。