肺出血

Methods Potential related risk factors of ROP, such as age, sex, birth weight, FiO 2, duration of inspiring ox ygen, hyaline membrane dzsease, Apgar score, intracranial hemorrhage , recurrent apnea, broncho pulmonary dysplasia, patent ductus arteriosus were studied prospectively by univariate analysis and Logistic regression a nalysis in 135 preterm infants.

方法以 13 5例早产儿患儿为研究对象，对胎龄、性别、出生体重、吸氧时间、吸氧浓度、肺透明膜病、Apgar评分、颅内出血、支气管肺发育不良、反复呼吸暂停、动脉导管开放、眼底等进行检查。同时用单因素χ2 检验和多因素logistic回归分析筛选和判定早产儿ROP发生的危险因素。

Methods 1.52 patients with cryptogenic hemoptysis were selected,including following two group:① routine image examinations (including chest plain film,tomogram,computed tomography and bronchialgraphy) were negative.② one lobe or segment showed a few lesions,but they were not the ones for causing hemoptysis proved by BAG,while another lobe or segment had signs of bleeding (17 cases).

1。选择隐源性咯血病人52例，包括以下两种情况：(1)常规影像学检查阴性(胸部平片、断层阴性，部分病人同期CT及支气管碘油造影阴性)(2)一个肺叶或一侧肺内有少量病变，但BAG证实为非咯血病灶，而另叶或另段肺内有出血征象，计17例。

The tachypnea immediately occurred for most of the rats, then bradypnea and dyspnea.

肺大体观及病理证实为肺损伤，且4h时出血明显，6h时水肿达高峰，24h仍未恢复。

American Atlanta conference edited in September 1992 pancreatitis of serious illness acute definition is: AP companion has local complication (cyst of pancreatic and necrotic, false sex, pancreatic abscess) or the organ fails (function of shock, lung not complete, kidney declines, gastrorrhagia, sow medicinal powder the cruor inside sexual blood-vessel and serious metabolization obstacle), or both holds concurrently.

概要： 急性胰腺炎为临床常见急腹症，是由于胰液和溶蛋白酶漏入到胰腺实质和胰腺四周组织而引起。1963年，法国马赛会议以病理形态学为基础，将AP分为水肿性胰腺炎和出血、坏死性胰腺炎。1992年9月美国亚特兰大会议修订重症急性胰腺炎定义为：AP伴有局部并发症(胰腺坏死、假性囊肿、胰腺脓肿)或器官衰竭(休克、肺功能不全、肾衰、胃出血、播散性血管内凝血和严重的代谢障碍)，或二者兼有。

Results （1） Observation by light microscope:Parts of lung were consolidation in 3rd day and 7th day groups of pneumonia, and macrophagus were prevailing in alveolus.

BLM第3天及第7天组大鼠肺组织实变、出血，其中炎细胞浸润情况与同期肺炎组类似；第14天及第28天组大鼠肺间质内持续存在单核巨噬细胞浸润，其阳性面积比与正常对照组比较差异有统计学意义（P〈0.05）。

Acute diffuse lung injury were identified in toxication control group, intra-pul

结果 中毒组大鼠肺的病理学改变为急性弥漫性肺损伤，表现为肺泡腔内出血、渗出、炎性细胞浸润，肺泡隔炎性细胞浸润及纤维组织增生，⌒秃廷⑿蜕掀は赴坏死脱落。

Acute diffuse lung injury were identified in toxication control group, intra-pulmonary alveoli hemorrage, effusion, inflammotory cell infiltration and inter-pulmonary alveoli, fibroplasia were seen; necrosis and abbscission were seen in of typeⅠand Ⅱ epithelial cells.

结果 中毒组大鼠肺的病理学改变为急性弥漫性肺损伤，表现为肺泡腔内出血、渗出、炎性细胞浸润，肺泡隔炎性细胞浸润及纤维组织增生，Ⅰ型和Ⅱ型上皮细胞坏死脱落。

The second, many coccus in kidney, heart and pancreas were found besides liver cell degeneration, congestion of liver, epithelium cell degeneration, necrosis of tubuli tenales, kidney hemorrhage, cardiac muscle cell degeneration, heart hemorrhage.

病例2，肝细胞变性，肺淤血，肾小管上皮细胞变性、坏死，间质出血，心肌纤维变性，间质出血，肾、心、胰脏均可见到散在分布的球菌。

The results showed that the sheep had fibrinous pneumonia and lymphocyte interstitial pneumonia, which were typical pathologic changes of this disease. Remarkable changes were observed in the lung: fibrinous exudates, infiltration of inflammatory cells, pleura thickening, interstitial edema, hemorrhage and formation of thrombiand lymph clot. The heart, liver and kidney had different degeneration degree. Reactive hyperplasia presented in bronchales lymphonodi.

结果发现该病例具有胸膜肺炎的典型病理变化，呈现纤维素性肺炎和淋巴细胞性间质肺炎，纤维素性物质的渗出和炎性细胞的浸润很明显，肺胸膜增厚，同时在肺间质还可见水肿、出血、血栓和淋巴栓的形成；另外心、肝、肾也均有不同程度的变性，淋巴结呈反应性增生。

Changes of structure of pulmonary tissue in each group: Twelve-hour LPS attack later, acute pulmonary injury was observed in the LPS group, and the histological examination was expressed as pulmonary edema, hemorrhage and inflammatory cell infiltration.

各组小鼠肺组织结构变化情况：脂多糖攻击12h后，脂多糖组出现急性肺损伤，组织学检查表现为肺水肿、出血和炎症细胞浸润。

I didn't watch TV last night, because it .

昨晚我没有看电视，因为电视机坏了。

Since this year, in a lot of villages of Beijing, TV of elevator liquid crystal was removed.

今年以来，在北京的很多小区里，电梯液晶电视被撤了下来。