肺

Results Of 177 cases,contusion of lung(n=115),traumatic wet lung (n=17),laceration of lung and hematomas of lung (n=16),atelectasis and consolidation (n=24)(includingthedelayed11),hemothorax(n=85)(includingthedelayed21)pneumothorax(n=17),pneumohemothorax (n=15),hemomediastinum(n=7),peumomediastinum(n=9),fracture of rib (n=94) and other organs' injuries.

结果：177例中肺挫伤115例，湿肺17例，肺撕裂伤及肺内血肿16例，肺不张及肺实变24例，其中11例为迟发性肺不张，血胸85例，其中21例为迟发性血胸，气胸17例，血气胸15例，纵隔血肿7例，纵隔积气9例，肋骨骨折94例，以及合并其他脏器损伤。

There were high density images shaped as rope and slip in lungs of model control group rats and the pleura near the images were a little thickening. In MTX treated group, there were high density images shaped as slice along pulmonary veins. The veins of the blood vessels were obscure and the edges were unclear. In TPT treated group,there were multiple high density images shaped as tubercle and slice and the boundary were not clear. In XFC treated group, the lungs' outlines were clear and the veins were welldistributed. High density images shaped as spot were found in part lungs.

结果：HRCT表现为模型对照组大鼠肺轮廓欠清晰，肺部血管影扩张，边缘模糊且不规则，两肺有索条状密度增高影，邻近胸膜轻度肥厚；甲氨喋呤组大鼠肺部可见沿着肺纹理分布的斑片状密度增高影，肺血管纹理模糊，边缘不清楚；雷公藤多苷片组大鼠两肺可见多发性结节状及斑片状密度增高影，境界欠清晰；新风胶囊组大鼠肺轮廓清晰，肺纹理分布均匀，部分肺组织显示斑点状密度增高影。

The results of respiratory mechanics, homodynamic mechanics,aterial blood gaS, and inflaInInatory factors [TNF- Q, IL-l fl, IL-6,IL-l0, and adrenomedulin] which detected in animal serumand bronchial alveolar lavage fluid obtained from tiP lObe,heart lobe, and diaPhragIn lobe of right lung seParate1y in the mongreldogs were measured before and after lung injury (including receivinglung protective ventilation).

本实验证明了 loS肺内／肺外不同源性 ARDS动物模型的差异性，而且AnDS的肺部病变存在不均一性和多形性的特点，对有关ARDS进行性低氧血症的发生机制提出了肺动静脉可能存在类似于右一左分流的肺内分流现象，以及因肺内分流而导致严重通气／血流比例失调的解释。发现小潮气十PEEP通气对肺内源性 ARDS疗效不佳，而俯卧位＋小潮气－I－ PEEP通气对肺内／肺外不同源性ARDS均有较好的疗效。。

X displays right pulmonary tuberculosis accounted for 30.4% the left lung accounted for 18.8% the double lung accounted for 50.8% between the lung nature venereal diseases changes the performance is two lung textures increases thickly the edge is fuzzy the lung hilus increases the disorganization accounted for 62.3%; The lobulous lung essence infiltration pathological change performance is in two lungs retires the inner tube spot laminated shape and the cloud cotton wool shape shadow the focus of infection fusion may assume the ground glass density to account for 30.5%; The lung section substantive infiltration pathological change displays for the boundary clear triangle or the fan-shaped big laminated high density was 7.2%.

胸部X线表现右肺病变者占30.4%，左肺占18.8%，双肺占50.8%，肺间质性病变表现为两肺纹理增粗，边缘模糊，肺门增大，结构紊乱占62.3%；小叶性肺实质浸润病变表现为两肺中、下野内带斑片状及云絮状阴影，病灶融合可呈磨玻璃密度占30.5%；肺段实质性浸润病变表现为边界清楚的三角形或扇形大片状高密度阴影占7.2%。

X displays right pulmonary tuberculosis accounted for 30.4% the left lung accounted for 18.8% the double lung accounted for 50.8% between the lung nature venereal diseases changes the performance is two lung textures increases thickly the edge is fuzzy the lung hilus increases the disorganization accounted for 62.3%; The lobulous lung essence infiltration pathological change performance is in two lungs retires the inner tube spot laminated shape and the cloud cotton wool shape shadow the focus of infection fusion may assume the ground glass density to account for 30.5%; The lung section substantive infiltration pathological change displays for the boundary clear triangle or the fan-shaped big laminated high density was 7.2%.

结果 7个月~1岁发病例数占4.3%；1~3岁占17.4%；4~7岁占46.4%；8~13岁占31.9%。咳嗽占100%；发热占63.8%；56.5%无肺部体征。胸部X线表现右肺病变者占30.4%，左肺占18.8%，双肺占50.8%，肺间质性病变表现为两肺纹理增粗，边缘模糊，肺门增大，结构紊乱占62.3%；小叶性肺实质浸润病变表现为两肺中、下野内带斑片状及云絮状阴影，病灶融合可呈磨玻璃密度占30.5%；肺段实质性浸润病变表现为边界清楚的三角形或扇形大片状高密度阴影占7.2%。

Histomorphometric studies were done to measure the following indexes: the IOD values of pulmonary, skeletal muscular and pleural small vascular walls from staining of AT1 and AT2 receptors, the pulmonary vascular density, muscularization of the nonmuscular vessels, percent wall thickness and proliferation of pulmonary small vascular walls.

结果：先心病患儿肺小血管壁AT1、AT2受体染色的IOD值均高于对照组(P=0.000)；先心病患儿骨骼肌和胸膜小血管壁AT1、AT2受体染色的IOD值均高于肺小血管(P＜0.05)；先心病患儿肺小血管壁AT1受体染色的IOD值与肺血管密度、非肌性肺血管肌化程度、肺小血管壁厚百分比和肺小血管壁细胞的增殖度相关(r分别为-0.783、0.742、0.521、0.657)，AT2受体仅与肺血管密度和血管壁厚百分比相关(r分别为-0.487、0.516)。

The contents of mineral elements, like Fe, in LMTCMs were higher than that of nor-LMTCM. The result of analysis of mineral element of 14 LMTCMs by ICP-MS method also showed the content of Fe have something to do with lung meridian tropism effects. The result of the study of platycodon radix approved the laws too. These laws were used to predict if the marine medicines have the lung meridian tropism effects.2. The Compatibility laws of Medicines and their chemical composition in LSP were studied by mining the data of the constituent herbs and their chemical composition. The results showed that the most constituent medicines of lung system prescriptions have lung meridian tropism. Radix glycyrrhizae, herba ephedrae, semen armeniacae amarum, fructus schisandrae Chinese and their compatibility are common in lung system prescriptions. Terpene, alkaloid, steroid, and flavanoid compounds and their compatibility are common in lung system prescriptions.3. The marine phycophyta medicines of LSP were studied by the same method.

对比归肺经中药及肺系方剂主要化学成分构成，萜类、生物碱、甾体类成分可能是归肺经中药及肺系方剂的主要药效成分；结合君臣佐使药的化学成分构成特点及肺系方剂组成中药的归经构成情况，肺系方剂组方设计应该考虑以下几个方面：一、优先考虑归肺经中药，在应用不归肺经中药时要配伍归肺经中药；二、组方时应考虑含萜类、甾体及生物碱类化合物的中药；治疗肺咳病及咳嗽症状时可以考虑含萜类化合物的中药，治疗气喘症状时可以考虑含生物碱类化合物的中药；三、在确定组方中药或化学成分配伍关系时，君药多考虑含生物碱、黄酮类化合物的中药；臣药、佐药可以考虑含萜类及甾体类化合物中药；使药可以考虑含萜类、甾体类及黄酮类化合物的中药。

The therapies for pulmonary disease include promoting and descending lung-qi, removing heat from the lung and relieving asthma, warming the lung to reduce watery phlegm, sending down lung-qi and resolving the phlegm, moistening the lung and resolving the phlegm, clearing away the dissolving phlegm, replenishing and restoring the lung-qi, astringing the lung-qi and nourishing the lung-yin.

肺病治法包括宣降肺气，泻肺平喘，温肺化饮，肃肺化痰，润肺化痰，清肺泻热，清肺化痰，补益肺气，收敛肺气，滋补肺阴；配伍用药包括配伍健脾药，配伍疏肝药，配伍补肾药，配伍开窍药，配伍补血药，配伍活血药，配伍解表药，配伍利湿药，配伍软坚药；辨治用方例举过敏性哮喘、肺气肿，喉源性咳嗽、慢性支气管炎，肺气肿、肺源性心脏病，肺间质纤维化，过敏性哮喘，矽肺、气胸。

Methods: Twenty-four rabbits were randomly divided into three groups:① the control group(group C, n=8): received sternotomy only and no ischemia;② the ischemia/reperfusion group(group I/R, n=8): the left lungs of rabbits were rendered ischemia by ligating the left pulmonary hili for 60 minutes followed by 60 minutes reperfusion;③ the edaravone group(group E, n=8): the left lungs were rendered ischemia for 60 minutes followed by 60 minutes reperfusion, and 10 mg/kg edaravone was administered intravenously 5 minutes prior to ischemia. Blood MDA and SOD were measured. Protein concentrations, WBC count, and PMN percentage in broncho-alveolar lavage fluid were determined. Samples of left lung tissue were sent for determining the left lung dry-to-wet weight ratio and evaluating the pathologic changes.

采用兔肺原位热缺血再灌注损伤模型进行研究。24只大白兔随机分为三组：①对照组(C组， n=8)，开胸后不阻断肺门，静脉缓慢推注生理盐水5ml/kg；②缺血再灌注组(I/R组， n=8)，左肺接受60min的缺血，然后接受60min再灌注，于缺血前5min静脉缓慢推注生理盐水5ml/kg；③依达拉奉干预组(E组， n=8)，于缺血前5min静脉缓慢推注依达拉奉10mg/kg（5ml/kg），左肺接受60min的缺血，然后接受60min再灌注。120min实验结束时，留取各组动物血液标本，测定血浆丙二醛含量及超氧化物歧化酶活性；收集支气管肺泡灌洗液，检测支气管肺泡灌洗液中白细胞计数、中性粒细胞百分比及肺通透性指数；留取肺组织标本，测定肺组织湿、干重量，计算干／湿重比及行肺组织病理检查。

Lung 肺 strong-lunged.

肺活力强的。

Neither the killing of Mr Zarqawi nor any breakthrough on the political front will stop the insurgency and the fratricidal murders in their tracks.

在对危险的南部地区访问时，他斥责什叶派民兵领导人对中央集权的挑衅行为。

In fact,I've got him on the satellite mobile right now.

实际上 我们已接通卫星可视电话了