肥大

It has been shown that in the design of hull forms for such high block coefficient ships, the attention must be payed to resistance performance both in the full load and ballast conditions.

在此基础上，探讨肥大船型的权衡设计，并认为在肥大船型设计时应同时兼顾满载和压载航行状态的阻力性能。

The results showed that mast cell of health rabbit distributed in many tissues, but mainly located in the superficial layer of epithelial tissues or in a location easy to reveal to antigen near blood vascular, gland tube and neuron.

结果表明，健康家兔肥大细胞分布于多种组织器官中，各种组织中肥大细胞多分布于浅层的上皮组织或靠近血管、腺管或神经等易表露于环境抗原的位点。

Apoenzyme enriching in mast cell can offer material basis for mast cell taking part in allergic reaction of Ⅰtype.

肥大细胞内酶蛋白的富集可为肥大细胞参与Ⅰ型变态反应提供物质基础。

RESULTS:(1) ET-1 could increase total protein production, surface area, ERKs activity and ［Ca2+］i in cultured cardiomyocyte in dose-dependent manner at concentrations ranging from 10-9 to 10-7 mol/L. And this effect could be abolished by BQ123, an antagonist of ETA receptor, partly inhibited by PTX, but not by BQ788, an antagonist of ETB receptor.（2）The activation of ERKs and the increase of ［Ca2+］i induced by ET-1 were obviously inhibited by PD98059, a selective ERKs kinase inhibitor, and nifedipine, a calcium channel blocker, respectively. Both antagonists partially inhibited ET-1-stimulated cardiomyocyte hypertrophic response.(3) Staurosporine, a selective PKC inhibitor, could inhibit ET-1-stimulated cardiomyocyte hypertrophic response and increase of ［Ca2+］i, but not affect the activation of ERKs.

结果： ①ET-1浓度依赖性增加新生大鼠心肌细胞蛋白质含量和心肌细胞表面积、ERKs活性及［Ca2+］i浓度，以上作用可被ETA受体拮抗剂BQ123所完全抑制，被百日咳毒素部分抑制，而ETB受体拮抗剂BQ788则无效；②ERKs激酶特异性抑制剂PD98059可完全抑制ET-1激活ERKs的作用，钙通道拮抗剂硝苯地平可明显抑制ET-1介导的［Ca2+］i浓度增加，但二者皆仅部分抑制ET-1介导的心肌细胞肥大反应；③蛋白激酶C选择性抑制剂staurosporine并不能明显抑制ET-1介导的ERKs激活，但可抑制ET-1介导的的［Ca2+］i浓度增加及心肌细胞肥大反应。

It seemed that the staining properties of grass fish mast cells were similar to mammalian mucosal mast cells in some aspects.

草鱼肥大细胞的染色性质在某些方面似乎类似于哺乳动物的粘膜肥大细胞。

Our experiments proved that VEGF have relationship with proliferation and involuting of hemangiomas.

结果显示，增生期血管瘤组织中P53着色的肥大细胞数目为29.64±2.67/HPF，消退期为14.87±4.43/HPF，血管畸形组织中为1.33±0.6/HPF.F检验结果表明：在增生期、消退期血管瘤及血管畸形组织中P53着色的肥大细胞数目有显著性差异（P.05）。

OBJECTIVE: To investigate the effect of mitogen activated protein kinase on the role of cardiac hypertrophy induced by Ang Ⅱ in cardiac myocytes so as to provide theoretical basis for clinical prevention and cure of cardiac hypertrophy.

目的：探讨丝裂素活化蛋白激酶在AngⅡ致心肌细胞肥大的作用，为心肌肥大的防治提供理论依据。

Results Average weights ofthe thymi and adrenal glands were45.38±14.13g and4.75±1.65g respectiveˉly;There were8adrenal gland atrophy cases and4lymph tissue hyperplasia cases in8decedents.

结果 胸腺肥大猝死者平均胸腺重45.38±14.13g，双侧肾上腺重4.75±1.65g，8例胸腺肥大猝死者肾上腺均发生不同程度萎缩、皮质细胞减少；4例伴有淋巴组织增生。

Common pulmonary signs have joint pain or hypertrophy, clubbing, multiple neuritis, myasthenia gravis, Cushing's disease, male breast hypertrophy, hypercalcemia, mental disorders.

肺外体征常见有四肢关节疼痛或肥大、杵状指，多发性神经炎，重症肌无力，库欣病、男性乳房增生肥大、高钙血症、精神异常等。

Extrapulmonary signs common with limb joint pain or enlargement, clubbing, multiple neuritis, myasthenia gravis, Cushing's disease, male breast hypertrophy, hypercalcemia, mental disorders.

肺外体征常见有四肢关节疼痛或肥大、杵状指，多发性神经炎，重症肌无力，库欣病、男性乳房增生肥大、高钙血症、精神异常等。

On the other hand, the more important thing is because the urban housing is a kind of heterogeneity products.

另一方面，更重要的是由于城市住房是一种异质性产品。

Climate histogram is the fall that collects place measure calm value, cent serves as cross axle for a few equal interval, the area that the frequency that the value appears according to place is accumulated and becomes will be determined inside each interval, discharge the graph that rise with post, also be called histogram.

气候直方图是将所收集的降水量测定值，分为几个相等的区间作为横轴，并将各区间内所测定值依所出现的次数累积而成的面积，用柱子排起来的图形，也叫做柱状图。