肥厚

Objective To investigate the correlation between the functional genetic variations of angiotensin-converting enzyme and Chymase genes and the risk of atrial fibrillation in hypertensive patients with left ventricular hypertrophy. Meth- ods From Dec.

目的 探讨高血压左心室肥厚患者血管紧张素转换酶基因和Chymase基因的多态性与心房颤动的相关性。

Iso could induce myocardial hypertrophy and change levels of correlational factors in rats.

Iso能促进大鼠心肌肥厚和相关因子的变化。

The major etiology of cubital tunnel syndrome is chronic injury with sustained compression of ulnar nerve around elbow joint.

结论］肘部的创伤及慢性劳损可以导致肘管弓状韧带出现肥厚增生，引起尺神经卡压磨损，这是肘管综合征最常见的病因；其他病因还包括慢性骨关节炎，占位病变和先天异常；尺神经可被机械性卡压和磨损，出现慢性缺血缺氧，导致肘管综合征的发生；详细的体格检查和术前的肌电图检查是诊断肘管综合征的主要手段，在诊断时应注意该病与其他部位迟发性尺神经麻痹的鉴别。

Results The patients with cardiac amyloidosis had characteristics of large atriums and hypertrophic ventricular walls in echocardiography. And they also had low limb lead voltage in ECG. The ratio of (SV5+RV1) and left ventricle crossing area of all patients were obviously lower than 1.5. Biopsy behaved staining of congo red. And most patients had renal, dermatic and gastrointestinal involvements.

结果 心脏超声心动图表现为患者左、右心房增大，心室肥厚，心腔正常；心电图表现为肢体导联低电压，心电图电压(SV5+RV1)与左心室横截面积CSA比值均明显小于1.5；心外活检表现为刚果红染色阳性，且大部分患者合并肾脏、皮肤及消化系统损害。

However, none of the 3 athletes had any other phenotypic features of HCM on further noninvasive testing and none had first-degree relatives with features of HCM. One of the 3 athletes agreed to detrain for 12 weeks, which showed resolution of electrocardiography and echocardiographic changes confirming physiologic left ventricular hypertrophy.

然而这3例非侵入检查未发现其他HCM表型特征，且他们无一级亲属有HCM特征。3例中有1例同意脱离锻炼12周，其心电图和超声心动图变化消失，证实左室肥厚为生理性改变。

Hypertensive, left ventricular fleshy, hidebound, severe anemia, dialytic it is critical factor inadequately.

高血压、左心室肥厚、营养不良、严重贫血、透析不充分是危险因素。

Next, as the extension of dialytic time, oedema, hypertensive, left ventricular fleshy and bloodshot sex heart decline with each passing day heighten.

其次，随着透析时间的延长，水肿、高血压、左心室肥厚及充血性心衰的发生率日趋增高。

Results: All cases have classical electrocardiographic manifestation.

结果： 15例均具有心尖肥厚性心肌病典型心电图表现。

When AT1 receptors are eliminated from the kidney, the residual repertoire of systemic, extrarenal AT1 receptors is not sufficient to induce hypertension or cardiac hypertrophy.

当从肾脏中去除血管紧张素Ⅰ受体，肾外的周身其余部分的血管紧张素Ⅰ受体不足以导致高血压及心脏肥厚。

Main changes of ECG were arrhythmia, Left ventricular high-voltage, Ventricular hypertrophy, bundle branch block and fascicular block, and ST-T abnormal, etc. Long-term exposure to high temperature has adverse effect on cardiovascular system.

结果 高温组的心电图异常率高于对照组(χ^2=12.00， P.01)，主要改变为心律失常，左室高电压、心室肥厚、束支与分支传导阻滞、ST-T异常等。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。