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Portal hypertensive enteropathy,whose fundamental pathologic change is a vasculopathy characterized histopathologically by mucosal and submucosal vessel dilatation,oedema and congestion,mainly results from a combination of increased intrahepatic vascular resistance and increased blood flow through the portal venous system.

肝硬化门静脉高压性肠病(portal hypertensive enteropathy,PHE)的2大主要发病机制是肝内门脉血流阻力增加和流经门脉血流增多。PHE的基本特征是组织病理学上的特征性血管病变。临床表现无特异性,但却是下消化道出血的主要原因。

In addition, HCC tissues with peplos encroachment showed lower expression rates than others without peplos encroachment (P<0.05). There was no other significant correlations of Plk1 expression with either age, tumor diameter and number, lymphoid node metastasis, extrahepatic metastasis, or portal vein embolus could be established.

高分化肝癌中Plk1蛋白的表达明显高于中低分化肝癌(P<0.01);无包膜侵犯的肝癌组织中Plk1蛋白的表达高于有包膜侵犯的肝癌组织(P<0.05);Plk1的表达与患者年龄、肿瘤直径、肿瘤数目、淋巴结转移、肝外转移、门静脉有无癌栓无关。

Results The change on morphous,size,liver lobe proportion,parenchyma density and portal vein in decompensation phase of liver cirrhosis was often observed,while splenectasis,collateral circulation construction,abnormality perfuse,ascitic fluid and pleural fluid also found in spiral CT scanning of liver cirrhosis.

结果 肝硬化在失代偿期CT表现有明显异常,肝脏有不同程度的形态、大小及肝叶比例改变,肝脏表面的实质密度、门静脉也有改变,并且常伴有侧支循环的建立、肝脏异常灌注、脾增大、胸腹水等。

Results: Different features were shown during the arterial phase, portal venous phase and lag phase, which were important for the diagnosis and differential diagnosis of focal hepatic lesions.

结果:不同类型的肝占位性病变在超声造影检查中显示出小同的动脉相、门静脉相和延迟相的变化。

Our results showed that the PPCC was always torturously worm-like in appearance on real-time US, and the hepatic artery usually had a linear channel appearance on real-time US.

我们的结果显示:在实时间超音波上,傍门静脉侧枝循环总是呈现扭曲虫样的型态外观,而肝动脉经常是直线形的管状构造。

The tissue samples of small intestine, liver and blood samples from peripheral and portal vein of 4 macaques of 6-month fetus, 2-day neonate, 45-day neonate and adult were obtained after anesthetization.

通过手术途径获得胚胎6月、新生2 d、新生45 d和成年猕猴的回肠、肝脏、门静脉和外周血等标本,应用放射免疫分析法测定各标本中的VIP含量;通过免疫组化方法观察VIP在肠、肝组织内的分布;利用原位杂交法检测VIP受体1(VIP receptor 1,VIPR1)的表达。

Among them, 18 lesions were lobar or segmental. The major causes of the filling defects were portal vein invasions or thromboses (10), laminar flows (11), and arterioportal shunts (8). Nine patients had diffuse and mottled filling defects.

楔形灌流缺损的主要原因为肝门脉侵犯或栓塞(10处),薄层血流(11处),和动脉门静脉交流(8处)。9位病人则呈现散在斑驳状的灌流缺损,其中有7位病人与肝硬化有关。

The research team from Switzerland and Germany investigated whether IP may also improve the blood flow in the hepatic artery and the portal vein, i.e., the macrocirculation during liver resections in humans.

来自瑞士和德国的研究团队研究了在人肝脏切除术中缺血预处理是否对肝动脉和门静脉即肝脏大血管的血流供应有积极的影响。

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