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AIM: To investigate the effects of potassium-magnesium aspartate injection on hepatic cells metabolism and its correlative mechanism.

目的:探讨门冬氨酸钾镁溶液对人肝细胞代谢的影响及其相关机制。

Objective To evaluate the effects of L-ornithin-L-aspartate treatment on the quality of life in patients with minimal hepatic encephalopathy.

目的 探讨左旋门冬氨酸鸟氨酸治疗对亚临床肝性脑病患者生命质量的影响。

Portal hypertensive enteropathy,whose fundamental pathologic change is a vasculopathy characterized histopathologically by mucosal and submucosal vessel dilatation,oedema and congestion,mainly results from a combination of increased intrahepatic vascular resistance and increased blood flow through the portal venous system.

肝硬化门静脉高压性肠病(portal hypertensive enteropathy,PHE)的2大主要发病机制是肝内门脉血流阻力增加和流经门脉血流增多。PHE的基本特征是组织病理学上的特征性血管病变。临床表现无特异性,但却是下消化道出血的主要原因。

To the best of our knowledge, this is the first report of portal cavernoma caused by a hydatid cyst of the liver.

据我们所知,这是第一例由肝钙化囊造成的门脉海绵样瘤。

Gate of Life: In the middle of the waist line, after the first two lumbar spinous process in the next depression, with replenishing liver and kidney, the role of Wenshen impotence, one can treat men's impotence, nocturnal emission, women's vaginal discharge, enuresis, frequent urination, irregular menstruation , as well as Yao Ji strong pain, hand, foot and cold.

命门:在腰部后正中线上,第2腰椎棘突下凹陷中,具有补益肝肾、温肾壮阳的作用,可以治疗男子阳痿、遗精,妇女带下、遗尿、尿频、月经不调,以及腰脊强痛、手足发冷。

The indication of treatment of splenomegaly in cirrhotic patients is presence of thrombocytopenia or complications from portal hypertension.

因此对肝疾患者而言,脾肿大的处理主要是针对血小板低下症及降低门脉高压而作治疗。

Results The common manifestations of Kala-azar misdiagonosed were irregular fever, poor appetite, acratia, hepatosplenomegaly, anemia et al. Kala-azar were easily misdiagnosed to be malnutritional anemia, lymphoid tuberculosis in pedo hilus pulmonis, hepatitis and nephritis et al.

结果 被误诊的黑热病常表现为不规则发热、食欲不振、乏力、肝肿大、脾肿大、贫血貌,易被误诊为营养不良性贫血、小儿肺门淋巴结结核、肝炎和肾炎等疾病。

Labeled perforin cDNA probe in liver biopsy tissues. RESULTS: Perforin positive cells were seen in the whole intralobular areas, especially in inflammatory areas. The amount of positive cells was different, 71.9% in spotty necrosis areas, 31.0% in focal necrosis areas, 9.2% in portal areas and only 4.8% in non????

结果: Perforin阳性细胞在整个肝小叶内均可检见,有炎性细胞浸润区均有perforin阳性细胞分布,浸润阳性细胞数量因病变部位的不同而有较大差异,慢性丙肝点状坏死区阳性率最高达71.9%,其次是片状坏死区(31.0%),门管区阳性率为9.2%,小叶内非坏死区仅为4.8%。

RESULTS: Perforin positive cells were seen in the whole intralobular areas, especially in inflammatory areas. The amount of positive cells was different, 71.9% in spotty necrosis areas, 31.0% in focal necrosis areas, 9.2% in portal areas and only 4.8% in non necrotic areas. The average amount of perforin cells in the liver lobules of 18 patients had no linear correlation with the ALT level or Knodell's histology activity index.

结果: Perforin阳性细胞在整个肝小叶内均可检见,有炎性细胞浸润区均有perforin阳性细胞分布,浸润阳性细胞数量因病变部位的不同而有较大差异,慢性丙肝点状坏死区阳性率最高达71.9%,其次是片状坏死区(31.0%),门管区阳性率为9.2%,小叶内非坏死区仅为4.8%。

Portal hypertension occurs as a consequence of structural changes within the liver in cirrhosis and increased splanchnic blood flow.

门脉高压是硬化肝内结构性变化及内脏血流增加的结果。

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