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肝脏

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Through all this test analyse Pathogenicity and Immunological status of ALV-J Inner Mongolia strain, the result showed the infected chickens began to have pathological change of Myelcytomatosis at 3-week: the main pathological changes of acidophile myelocyte were found in bone marrow and between cardiac muscle fibers,and there were many granules in the hyalotome.This kind of oncocyte proliferated in every tissue at different amount, but the cells that proliferated in bone marrow, cardiac muscle fiber, oarium were much more than other tissue, and they often developed neoplasma. At same condition,there were no abnormalty advent in control group, In immunological aspect: the increase of lymphokine and ANAE positive cells showed cell-mediated immunity was strengthend during antineoplastic infection; the emergence of ALV-J antibody of ELISA suggest there was humoral immunity exist at the same time.

结果显示:脾脏PCR检测结果表明1、2两实验组均有阳性病例出现,而对照组全部为阴性,说明实验鸡体内已有ALV-J内蒙株感染;接种病毒后3周龄开始出现骨髓细胞瘤病的病理变化:肝脏、脾脏有程度不同的肿大,镜下观察,骨髓内最早出现了与骨髓细胞相似、形态基本一致的大型圆形或椭圆形、胞核较大多为椭圆形、胞浆内含有大量嗜酸性圆形颗粒的髓细胞样瘤细胞增生,随后通过血流瘤细胞可转移到肝脏、心肌、卵巢或睾丸等组织并形成肿瘤性病灶,而对照组没有出现明显病变;免疫状态方面:以脾脏淋巴因子和ANAE阳性细胞增多说明细胞免疫在抗肿瘤感染过程中增强,ELISA检验实验鸡抗体出现阳性的结果表明同时也存在着体液免疫。

Can be used to treat heart disease, liver disease, agranulocytosis, thrombocytopenia, optic atrophy, prevention and lifting of schistosomiasis from drug-induced liver or heart side effects.

可用于治疗心脏病、肝脏疾患、白血球减少症、血小板减少症、视神经萎缩,能预防及解除由血防药物所引起的对心脏或肝脏的副作用。

Objective To investigate the differentiation of bone marrow derived Thy-1+β2M-cells into liver cells in allyl alcohol induced liver injury micro-environment.

研究表明,除了肝脏、胰腺中存在肝干细胞外,移植骨髓来源的干细胞也可在受者肝脏内分化为肝细胞和胆管上皮细胞[l,2]。

Portal vein arterialization has no obvious influence on liver hemodynamics and microvessels.

门静脉动脉化后1 mo对肝脏的血流动力学和肝脏的微血管不会造成明显的不良影响。

Methods Experimental model of partial portal vein arterialization was established in rats. The changes of hepatic microvasculature and histology in the rat model were observed.

建立大鼠部分门静脉动脉化重建肝脏血流的实验模型,观察该模型大鼠肝脏微血管和组织学的变化。

Hepatitis viruses, alcohol, drugs, poisons, blood fluke,and so on, may irritate liver long term, which then activates hepatic stellate cells living in hepatic sinusoid to excrete excess extracellular matrix.

肝纤维化的形成主要是由于肝炎病毒、酒精、药物、毒物、血吸虫等多种损伤因素长期刺激肝脏,使肝窦内肝星状细胞活化,肝组织内细胞外基质成分过度增生与异常沉积,导致肝脏结构或功能异常。

Objective To study on chance and regularity of doing harm to thr liver of chronic Brucellosis patients.

目的 探讨慢性布氏菌病是否可以造成肝脏损害及肝脏损害在慢性布病中发生的几率和规律。

Objective To investigate the contribution of bone marrow cells transplantation in the treatment of acute liver injury induced by carbon tetrachloride,and the differentiation of transplanted bone marrow cells into hepatocytes in vivo.

目的观察骨髓细胞移植对急性肝损伤小鼠肝脏的保护作用,以及移植的骨髓细胞在小鼠肝脏内的转归。

HEPATIC VEIN - Main outflow vein of liver connecting liver to vena cava and then to heart.

肝静脉——肝脏主要的流出静脉,连接肝脏和腔静脉,然后到心脏。

TLR4 participate in the signal transduction of inflammatory cell of SAP. Sandostation can down regulate the expression of TLR4 level, which may influence on the signal transduction of inflammatory cell of the SAP rats.

TLR4影响SAP大鼠肝脏细胞炎症信号转导过程,善宁可能通过影响TLR4的表达,干预SAP大鼠肝脏细胞炎症信号转导过程。

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