肝的 - 网络例句 - 英语人，englisher

Abstract］ objective to study the pathological features and histopathological type and differential diagnosis of hepatic focal nodular hyperplasia.methods the clinicopathological characteristics of 40 cases of fnh were studied.all were evaluted by use of paraffin embedded sections and he staining before light microcope observation.results there were 28 females and 12 males fnh patients whose age were from 16 to 62 years（median 41.3）,all alpha-fetoprotein was negative and had no hepatitis history.25 cases were classic type showed characteristic central stellate fibrotic scar,composed of fibrous connestive tissue and tortuous blood vessels.8 cases were telangiectic type,the left were mixed type and adenomatoid type.conclusion fnh is a reactive proliferation of hepatic cells to local blood vessel anomalies,it is not realy a tumor.its differential diagnosis includes hepatic adenomatous hyperplasia nodule,hepatic anaplasia nodular hyperplasia,fibrolamellar hepatocellular carcinoma and hepatocellular adenoma.

目的探讨肝局灶性结节性增生的病理形态特点、组织分型及鉴别诊断。方法分析40例肝局灶性结节性增生的临床资料，并采用石蜡包埋he染色光镜下观察其组织学特点。结果 40例肝局灶性结节性增生患者中，女28例，男12例，年龄18~62岁，平均年龄41.3岁，所有病例术前均无肝炎病史，甲胎蛋白阴性，组织学上25例为经典型，有特征性的中央纤维瘢痕，由纤维结缔组织及扭曲血管组成。8例为毛细血管扩张型，其余为混合型及腺瘤样增生型。结论肝结节性增生是一种肝细胞对局部血管的异常反应性增生，并非真性肿瘤，主要与肝腺瘤样增生性结节、肝间变性结节状增生、肝纤维板层癌及肝细胞腺瘤鉴别。

Objective: Augmenter of liver regeneration cloned from liver of weaning rat is a novel hepatic stimulator. It was reported that ALR mRNA expression was correlated with DNA synthesis of hepatocytes after partial hepatectomy, and could significantly promote the survial rate of intoxicated hepatitis rats.

目的：肝再生增强因子是从新生大鼠肝组织中克隆到的一种肝增殖刺激因子，其特异mRNA的表达与大鼠肝部分切除后肝细胞DNA合成密切相关，是一种特殊的促肝细胞分裂原，能特异地刺激肝细胞增殖，是肝再生的重要调控因子，能提高中毒性肝损伤动物的存活率，在肝损伤修复过程中发挥作用。

The liver fibrotic model was induced by CCl4 i.p for 5wk. Telmisartan was given at the same time model establishment in each intervened group by administration lasted for 6wk. Decollate to get the blood of all rats after 7 weeks . ALT、AST and ALB of serum was detected by automatic biochemistry equipment. Hyaluronic acid was detected by ELISA detection. Liver and spleen was obtained to calculate the index. The liver pathological changes were observed in each group of rats after HE staining to evaluate the therapeutic effect of Telmisartan; TGF-β1、PPAR-γand PDGF of liver tissue was detected by the method of immunohistochemistry respectively; The mRNA expression of TGF-β1、MMP-1、TIMP-1、α-SMA、PPAR-γand iNOS was detected by RT-PCR detection, respectively.

腹腔注射CCl4制备SD大鼠肝纤维化动物模型，连续灌胃6周，处死动物取血，检测血清ALT、AST、ALB；ELISA法测定血清透明质酸；取肝、睥，并计算肝、脾指数，组织切片HE染色后进行组织病理学疗效评估；用SP免疫组化法测定肝组织TGF-β1、PPAR-γ及PDGF蛋白表达；半定量RT-PCR方法检测肝组织TGF-β1、MMP-1、TIMP-1、α-SMA、PPAR-γ和iNOSmRNA的表达。

OVCs were isolated by density ladder centrifugation in the 4th week, and then OVC's morphology was observed under transmission electromicroscrope and immunocytochemistry were performed to detect the expression of ICAM-1. Results Observing OVC's morphology under transmission electromicroscrope showed that OVC was infantile and undifferentiated with big nucleus, clear nucleolus, large nucleoplasm-ratio, small mitochondria, and little endoplast.

结果透射电镜下观察到OVCs超微结构的改变为核大，核/浆比大，核仁小，胞浆内细胞器少，可见少量内质网和小线粒体，整个细胞呈幼稚、未分化状态；ICAM-1免疫细胞化学检测阳性；损伤肝组织HE染色和ICAM-1免疫组化检测显示，肝损伤初期，Hering管周围OVCs增生并且ICAM-1阳性表达，随着肝损伤的加重，OVCs继续增生，并且，OVCs沿增生的肝纤维向肝小叶迁移，同时，ICAM-1阳性表达也继续增多，并逐渐向肝小叶弥散分布；透射电镜下观察到，肝损伤初期，OVCs在Hering管周围增生，肝纤维随之增生，增生的OVCs与肝纤维黏附生长。

OVCs were isolated by density ladder centrifugation in the 4th week, and then OVC's morphology was observed under transmission electromicroscrope and immunocytochemistry were performed to detect the expression of ICAM-1. Results Observing OVC's morphology under transmission electromicroscrope showed that OVC was infantile and undifferentiated with big nucleus, clear nucleolus, large nucleoplasm-ratio, small mitochondria, and little endoplast. In initial stage of damaged liver, OVCs and the expression of ICAM-1 mostly distributed around Hering duct, and then gradually increased and expanded toward hepatic lobule, shown by staining paraffin sections with HE, immunochemistry and transmission electromicroscrope. Immunocytochemistry indicated that ICAM-1 expression on OVCs was positive.

结果透射电镜下观察到OVCs超微结构的改变为核大，核/浆比大，核仁小，胞浆内细胞器少，可见少量内质网和小线粒体，整个细胞呈幼稚、未分化状态；ICAM-1免疫细胞化学检测阳性；损伤肝组织HE染色和ICAM-1免疫组化检测显示，肝损伤初期，Hering管周围OVCs增生并且ICAM-1阳性表达，随着肝损伤的加重，OVCs继续增生，并且，OVCs沿增生的肝纤维向肝小叶迁移，同时，ICAM-1阳性表达也继续增多，并逐渐向肝小叶弥散分布；透射电镜下观察到，肝损伤初期，OVCs在Hering管周围增生，肝纤维随之增生，增生的OVCs与肝纤维黏附生长。

Results Observing OVC's morphology under transmission electromicroscrope showed that OVC was infantile and undifferentiated with big nucleus, clear nucleolus, large nucleoplasm-ratio, small mitochondria, and little endoplast. In initial stage of damaged liver, OVCs and the expression of ICAM-1 mostly distributed around Hering duct, and then gradually increased and expanded toward hepatic lobule, shown by staining paraffin sections with HE, immunochemistry and transmission electromicroscrope. Immunocytochemistry indicated that ICAM-1 expression on OVCs was positive.

结果透射电镜下观察到OVCs超微结构的改变为核大，核/浆比大，核仁小，胞浆内细胞器少，可见少量内质网和小线粒体，整个细胞呈幼稚、未分化状态；ICAM-1免疫细胞化学检测阳性；损伤肝组织HE染色和ICAM-1免疫组化检测显示，肝损伤初期，Hering管周围OVCs增生并且ICAM-1阳性表达，随着肝损伤的加重，OVCs继续增生，并且，OVCs沿增生的肝纤维向肝小叶迁移，同时，ICAM-1阳性表达也继续增多，并逐渐向肝小叶弥散分布；透射电镜下观察到，肝损伤初期，OVCs在Hering管周围增生，肝纤维随之增生，增生的OVCs与肝纤维黏附生长。

ABSTRACT Objective To discuss the importance of the study of conformation and variation of extrahepatic bile duct before gallbladder operationMethods The images of 100 cases having normal structure of gallbladder and extrahepatic bile duct were studied;the conformations and confluence sites were analyzedResults Among the 100 cases,97 cases had clear images of extrahepatic bile duct (97%),3 cases with large amount of air bulbs in bile duct because of the laxation of the lower common bile duct still had clear images of the general conformation of extrahepatic bile duct,87 cases had clear images of gallbladder and cystic duct (87%),13 cases (13%) had partial images of gallbladder or cystic ductConclusions ERCP can clearly show out the conformation and variation of extrahepatic bile duct and cystic duct and it is of important value before gallbladder operation

目的通过对100例胆囊及肝外胆管正常结构尚存在的患者ERCP图像资料进行归纳和分析，探讨胆囊手术术前了解肝外胆道系统形态及变异的重要性。方法选出100例胆囊及肝外胆管正常结构尚存在的患者图像，对其形态、汇合位置进行分析。结果 100例患者图像中肝外胆管显示清晰者97例(97%)，3例因胆总管下端开口松弛导致大量气泡进入胆道，但肝外胆管大体形态显示尚清晰，胆囊及胆囊管显示清晰者87例(87%)，13例(13%)仅部分显示胆囊或胆囊管。结论 ERCP可以清晰显示肝外胆管及胆囊管的形态及变异情况，对于胆囊手术术前了解肝外胆道系统形态及变异有重要价值。 ERCP；胆囊管；肝外胆管

All specimens of chicken liver were contaminated with stilbestrol and 81.82% of them with arsenic. Duck liver, pig liver and fresh milk were detected with different levels of contaminants.

其中33份鸡肝、35份鸭肝、34份猪肝样品的铅检出率分别为72.72%，85.00%，73.53%；31份鸡肝和30份鸭肝样品的镉检出率均为100%；33份鸡肝、34份鸭肝和27份猪肝样品的砷检出率分别为93.34%，11.76%和33.33%；33份鸡肝样品中27份砷检测值超标，超标率为81.82%。70份鲜牛奶样品有4份检出速效氯霉素，检出率为5.72%。28份鸡肝、20份鸭肝样品全部检出乙烯雌酚。

Methods Using HIRI model in tabbits,animals were randomly divided into four groups (n=10 in each),HIR group,HIR+ LGT group,HIR+ Pro group and HIR+ LGT+ Pro group,the contents of adenosine triphosphate,adenosine diphosphate,adenosine monophosphate,total adenylic acid numberand energy charge in the liver tissue were measured at 45 minutes after reperfusion, respectively.

复制肝缺血-再灌注损伤模型，随机将40只实验兔分为肝缺血-再灌注组、肝缺血-再灌注＋LGT治疗组、肝缺血-再灌注＋PRO治疗组和肝缺血-再灌注＋LGT＋PRO治疗组。在再灌注45 min时，分别观察肝组织内三磷酸腺苷、二磷酸腺苷、一磷酸腺苷含量、总腺苷酸量、能荷及肝细胞形态学的变化。

The faciform ligament is an important sign of laparoscopic liver surgery and a way of collateral circulation of liver. The superior liver artery found and named in this research can be an important sign to judge the position of hepatic vein. The operation pathway by cutting down the perihepatic ligament next to the superior border of liver is a quick way with little blood. It is difficult to hurt diaphragm and hepatic vein, and the secundum porta hepatic can be showed clearly. As a rule, we break the hepatic vein in the hepatic parenchyma on laparoscopic hepatectomy. But in some situation, extrahepatic treatment was used.

镰状韧带是腹腔镜肝脏外科入路的重要标志，是肝脏侧枝循环的重要途径；本文所发现并命名的肝上动脉可以作为判定肝静脉位置的重要标志；紧贴肝上缘打开肝周韧带，不易损伤膈肌和肝静脉，出血少、速度快，可以使第二肝门获得良好的手术显露；腹腔镜肝切除时一般在肝实质内切断肝静脉，在个别情况也可以预先在第二肝门结扎切断肝静脉。