肌蛋白

We demonstrate that the establishment of a polarized actin cytoskeleton—either as a consequence of normal cell division or through activation of the mating pheromone response—potently attenuates protein synthesis and growth.

我们表明，建立一个两极化的肌动蛋白细胞骨架，无论是作为一正常细胞分裂，或通过激活交配信息素反应抑制蛋白质合成和增长。

It was reported that the eventual fate of receptor-activated Smads was controlled by ubiquitin-mediated proteolysis.

以上结果提示，在低氧抑制成肌细胞体外分化的过程中，低氧加速了磷酸化Smad3蛋白的泛素-蛋白酶体途径的降解。

Radio-ligand binding assay was applied to determine the maximum binding and affinity changes of nicotinic acetylcholine receptor on five weeks old male Sprague-Dawley rat's lateral pterygoid muscle and superficial masseter muscle after the function mandibular advancement.

本研究在以往研究的基础上，首次对功能矫形的神经肌肉调控机理进行探索。应用放射受体分析法，从膜受体蛋白分子水平定量研究功能矫形前伸青春期SD大鼠（5周龄）下颌后，下颌前伸肌细胞膜烟碱样乙酰胆碱受体最大结合容量和亲和性的变化。

Subject: The purpose of this.thesis is to explore the therapeutic mechanism of Radix Astragli injection from the point of view of modern medicine through studying its clinical therapeutic effect on patients with chronic glomerulonephritis characteristic of "qi" deficiency in lung - kindney , and its effect on urine protein, plasma albumin, plasma cholesterol, plasma creatinine, urea, hemodynamies urine2-microgiobulin , T lymphocyte , plasma endothelin , nitric oxide .

目的：本文通过研究黄芪注射液对肺肾气瘀证慢性肾小球肾炎患者的临床治疗效果以及对蛋白尿、血浆白蛋白、血浆胆固醇、血肌酐、尿素氮、尿β_2-微球蛋白(β_2-MG)、血流变学、T细胞亚群、血浆内皮素、一氧化氮的影响，从现代医学的角度探讨其治疗作用机理。

At the same time, PDI can act as molecular chaperone that help the denatured proteins refold and inhibit the aggregation of the denatured proteins.

在神经变性疾病如帕金森症、阿尔兹海默症、肌萎缩侧索硬化症、多聚谷氨酰胺疾病中，PDI可通过抑制错误折叠蛋白的聚集以及遍在蛋白化，起到神经保护作用。

FN and LN (the non-collagenous index) out of ECM were measured by immuno-histochemistry mean, and the accumulation of ECM was measured by typeⅠand Ⅲ collagen ,α-SMA was chosen as a symbol of the activated reposit fat cell. TGF-β〓 was chosen as a symbol of the cellular factor made the reposit fat cell activated. We observed the mechanism of 286 through the variety of the upper indexes during the experiment.

用免疫组化方法测评肝细胞外基质中的非胶原指标FN、LN，胶原指标Ⅰ、Ⅲ型胶原等在实验过程中的变化作为判定ECM沉积增多的事实指标；用a-平滑肌肌动蛋白作为贮脂细胞被激活的标志物指标；用转化生长因子-β〓作为激活贮脂细胞的细胞因子指标；通过观测上述指标在实验中的变化，来探讨"268方"的作用机理。

FN and LN(the non-collagenous index) out of ECM were measured by immuno-histochemistry mean, and the accumulation of ECM was measured by type I amd III coHagen, a -SMA was chosen as a symbol ef .the activated reposit fat cell.TGF- P1 was chosen as a symbol of th ieccellular factor made the reposit fat cell activated.We observed the mi sclnanism of 286 through the variety of the upper indexes during the experiment.

用免疫组化方法测评肝细胞外基质中的非胶原指标FN、LN，胶原指标Ⅰ、Ⅲ型胶原等在实验过程中的变化作为判定ECM沉积增多的事实指标；用a-平滑肌肌动蛋白作为贮脂细胞被激活的标志物指标；用转化生长因子-β_1(TGF-β_1)作为激活贮脂细胞的细胞因子指标；通过观测上述指标在实验中的变化，来探讨"268方"的作用机理。

Objective The purpose of the present study was to observe the muscle glucose metabolism by determining the changes in sarcolemma GLUT4 protein content and muscle AMPK activity in overtraining rats.

目的：观察过度训练后大鼠骨骼肌糖原含量、AMPK活性和肌膜GLUT4蛋白含量的变化，探讨过度训练与骨骼肌葡萄糖代谢之间的联系。

CK, LDH in serum, SOD, MDA in muscular tissue, and Na+-K+-ATPase activity in sarcolemma were measured respectively.

以DPH和N[3P]M为探针，用荧光分光光度计分别测定肌细胞膜脂质流动性和膜蛋白的活动性。

Our results indicate that the deficiency of dystrophin would lead to the reduction or deficiency of SG proteins, which would cause disruption/dysfunction of dystrophin-glycoprotein complex , loss of linkage between subsarcolemmal cytoskeleton and extracellular matrix which, in turn, would lead to sarcolemma instability and eventually to muscle cell necrosis.

表明dystrophin缺乏将导致包括SG在内的dystrophin相关蛋白的显著减少，DGC复合体结构破坏或功能障碍，细胞膜下骨架与细胞外基质连系的丧失，致使肌细胞膜不稳定，在肌肉收缩时容易受损，最终肌纤维变性、坏死。

And Pharaoh spoke to Joseph, saying, Your father and your brothers have come to you.

47：5 法老对约瑟说，你父亲和你弟兄们到你这里来了。

Additionally, the approximate flattening of surface strip using lines linking midpoints on perpendicular lines between geodesic curves and the unconditional extreme value method are discussed.

提出了用测地线方程、曲面上两点间短程线来计算膜结构曲面测地线的方法，同时，采用测地线间垂线的中点连线和用无约束极值法进行空间条状曲面近似展开的分析。