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The cells labeled with PKH26 were seeded on the bio-derived bone to construct tissue engineered bone in vitro. Then the compound of cells and material were observed under fluorescence microscope. The compound of labeled cells and material were implanted into the rabbit thigh muscle, and the transformation of the labeled cells was observed by fluorescence microscope 14 and 28 days later.

将PKH26 标记的BMSCs 接种于生物衍生骨材料,48 h 后荧光显微镜观察细胞与材料的复合情况;将细胞-材料复合物植入成年新西兰大白兔双后肢肌袋,14、28 d 后荧光显微镜观察标记细胞的转归。

Summary: In this study, the change of adhensive struture, adhensive moleculd and actin filament in K-ras gene transfected pulmornary fibrocyte of mice, and the mechanism of invasion and metastasis of cancer cell were investigated by molecular biological techniques.

小结:本研究利用分子生物学技术,探讨K-ras基因导入的鼠肺纤维细胞—恶性转化细胞的粘附结构、粘附分子和肌动蛋白的改变,旨在探讨肿瘤细胞的浸润转移机制。

Results showed that after injection of HRP into medial recteus subnucleus of oculomotor nucleus,HRP labelled cells were found in contralateral abducens nucleus and also in vestibular nucleus and pontine ...

结果表明:注射HRP于大鼠动眼神经核内直肌亚核后,在对侧展神经核区以及前庭神经核、脑桥旁正中网状结构中发现HRP单标记细胞;在前庭神经核内,可见HRP单标记、GABA阳性和HRP/GABA双标记三类神经元,其中HRP/GABA双标记细胞占HRP标记细胞总数的47.1%。

There are close relationship among hemangiopericytoma, solitary fibrous tumor, lipomatous hemangiopericytoma and giant cell angiofibroma.

肌外皮细胞瘤、血管球外皮细胞瘤和成人的婴儿型肌纤维瘤病也属于血管外皮细胞瘤。

In small intestine,the villous epithelial cells,Paneth cells and parasympathetic ganglion cells in myenteric plexus show GnRHR immunoreactivity.

三段小肠绒毛上皮细胞、潘氏细胞、十二指肠腺上皮细胞以及小肠肌间神经丛副交感神经节细胞,均呈较强的GnRH受体免疫反应性。

Results The expression of vimentin in interstitial tissue, cytoplasm of interstitial cells, cytoplasm of Sertoli, cytoplasm of myoid cells were positive, not excepting cytoplasm of spermatogoniums.

结果 在染锰大鼠睾丸中除间质,间质细胞、支持细胞,肌样细胞细胞质波形蛋白表达阳性外,也有精原细胞的细胞质VM表达阳性。

Myoid/myofibroblastic areas in DFSP possibly represents the hyperplasia of stromal myofibroblasts, rather than true myofibroblastic differentiation of the neoplastic cells.

DFSP中的肌样/肌纤维母细胞性分化可能是肿瘤间质中肌纤维母细胞增生的结果,并非代表了瘤细胞的真性肌纤维母细胞性分化。

3Rhe distribution and change of proliferating cell nuclear antigen in seminiferous tubule: The positive staining of proliferating cell nuclear antigen was found in slices of all groups, the immunological positive products were located at karyon, usually in brown yellow granules, only a small part of them in diffuse state; the immunological positive cells were mainly the spermospores, besides a small quantity were spermiocyte, supporting the cell and myoid cell nuclear to be stained.

3增殖细胞核抗原在生精小管中的分布及变化:各组切片均可见增殖细胞核抗原阳性染色,免疫阳性产物位于细胞核,通常呈棕黄色颗粒,小部分呈弥散状;免疫阳性细胞主要是精原细胞,另外还有少量初级精母细胞、支持细胞和肌样细胞核着色。

Results The results showed that fibroblasts were the main source of extracellular matrix production in bile duct wall. The phenotype of fibroblasts in inflammatory strictured bile duct wall changed obviously, quiescent fibroblasts were activated and transformed to myofibroblasts, with massive proliferation.

结果 成纤维细胞是胆管壁中合成胶原的主要细胞;炎性狭窄胆管壁的纤维化增厚明显,其成纤维细胞表型改变,由相对静止转化为功能活跃状态,并进一步向肌成纤维细胞转化,且大量增殖。

The injured gill cells had a different ultrastructure, with endoplasmic reticulum dilated and vesiculated, mitochondrial cristaes partially disintegrated, gill fibre cuticles denatured and dropped. The ultrastructural damages to hepatopancreatic cells were characterized by the decrease of microvilli, swelling and breaking up of mitochondria, expansion of endoplasmic reticulum, disruption and vacuolization of nucleus, tumefaction of nuclear membrane and increase of lipid droplets. As for the toxicological changes of myocardial cells, the mitochondria were swelling, having a lot of vacuolus and disintegrating, while the myofibrils were irregular with endoplasmic reticulum dissolved.

鳃细胞的损害表现为鳃丝水肿,细胞器溶解,角质层变薄,线粒体、内质网的肿胀、解体;肝胰脏细胞的主要损害特征为肝管微绒毛减少,线粒体水肿解体,内质网扩张,细胞核空泡化,核膜水肿和脂肪滴增加;心脏细胞的毒理变化为线粒体内嵴肿胀、瓦解,肌原纤维不规则,内质网溶解。

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