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Objective To investigate mRNA expression of sarcoplasmic reticulum calcium regulatory proteins in myocardium from ischemia-reperfusion injury during cardiopulmonary bypass.

目的 研究体外循环术中心肌缺血-再灌注损伤后肌浆网Ca2+调节蛋白mRNA表达。

Function of the sarcoplasmic reticulum and expression of its Ca2+-ATPase gene in pressure overload-induced cardiac hypertrophy in the rat.

缺血-再灌注过程中心肌肌浆网钙摄取和Ca2+-ATPase活性的变化。

The sarcoplasmic reticulum is a system of intracellular membranes.

肌浆网属于细胞内膜系统。

To investigate the effects of Ca(superscript 2+) released from sarcoplasmic reticulum on atrial natriuretic peptide secretion.

目的 观察肌浆网(sarcoplasmic reticulum, SR)释放Ca(上标 2+)对心房钠尿肽(atria1 natriuretic peptide, ANP)分泌的影响。

Objective To observe changes in cardiac renin-angiotensin and its effect on sarcoplasmic reticulum calcium transport function, and investigate the mechanism underlying cardiac dysfunction in early postburn stage.

目的 观察烧伤早期心肌局部肾素-血管紧张素系统变化及其对心肌肌浆网钙运转功能的影响,探讨烧伤早期心功能障碍的发病机制。

Methods Forty patients with hepatobiliary and pancreatic diseases undergoing biliojejunostomy or cystojejunostomy were randomly divided into seromuscular layer anastomosis group (n=20) and two-layer anastomosis group (n=20) before operation, which received Roux-en-Y end-to-side jejunal seromuscular layer anastomosis and two-layer anastomosis, respectively.

行胆管空肠Roux-en-Y吻合的患者随机分成2组:浆肌层吻合组(n=20)和二层吻合组(n=20),浆肌层吻合组行Roux-en-Y袢处空肠-空肠浆肌层吻合,二层吻合组行Roux-en-Y袢处空肠-空肠二层吻合。

Mysoin, the protein that allows muscle to contract, was known to be essential for the beginning of development in certain amoebas, but its importance in subsequent stages was unknown.

肌浆球蛋白是一种促使肌肉结合在一起的蛋白,众所周知,这种蛋白是特定变形虫开始发育的必要物质,但是它在后期发育阶段的重要性并不为人所知。

There was no difference in its expression between the right atriums and the right appendages.

结果:房颤者肌浆网 RyR2 受体 mRNA 较窦性心律者上调,心房不同部位无差别。

Background: Opening of cardiac ATP-sensitive potassium channels is a well-characterized protective mechanism against ischemia and reperfusion injury.

心肌细胞ATP敏感钾通道的开放可以保护心肌免受缺血再灌注损伤,有证据显示肌浆网和线粒体K通道与这种保护效应有关。

SANG induced Ca2+ release from the actively loaded SR vesicles was blocked by ruthenium red and dithiothreitol, consistent with the ryanodine receptor as the site of SANG action.

此外,血根碱诱导钙离子由肌浆网释放之作用会被ruthenium red以及DTT所抑制,综合上述结果显示,血根碱之作用部位是位於ryanodine-受体。

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然而,正如其名字所指出的那样,CD盘不能写,也不能用任何方式改变其内容。

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