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16-slice spiral CT by all kind of imaging ways could clearly display the anatomy and the pathologic changes of tendon, muscle belly and ligament such as the rupture, thickening, hydropsia and circuity, provided a effective way to diagnose the closed injury of antebrachim tendon and cruciate ligaments of knee joint.

应用16层螺旋CT检查前臂肌腱损伤及膝关节交叉韧带损伤,其多种成像方法能够清晰地显示各肌腱、肌腹与韧带的断裂、迂曲、增粗和水肿等病理改变与解剖关系,为临床诊断和手术治疗提供有力的依据,具有重要的参考意义。

The injury of tendon and ligament such as the rupture, thickening, hydropsia and circuity also were showed by 16-slice spiral ct image. conclusion 16-slice spiral ct by all kinds of imaging ways could clearly display the anatomy and the pathologic changes of tendon, muscle belly and ligament such as the rupture, thickening, hydropsia and circuity, provided an effective way to diagnose the closed injury of antebrachim tendon and cruciate ligaments of knee joint.

应用16层螺旋ct检查前臂肌腱损伤及膝关节交叉韧带损伤,其多种成像方法能够清晰地显示各肌腱、肌腹与韧带的断裂、迂曲、增粗和水肿等病理改变与解剖关系,为临床诊断和手术治疗提供有力的依据,具有重要的参考意义。

There were micronodules of smooth muscle fascicles which were separated by fibrous connective tissue with hydropic changes and rich vascularity in cluster.

组织学特点:瘤细胞表现典型的平滑肌分化,伴厚壁与薄壁的大血管,血管呈丛状分布,周围基质水肿明显,肿瘤组织在肌壁中较局限地分割,扩展性生长。

Such as inflammation of joints, the lower leg, such as chronic venous insufficiency lead to long-term sustainability of the skin caused by congestive hairy; hyperthyroidism in Pretibial myxedema patch Department often long acuminatum out; hypothyroidism and limbs of children in the back outside a large number of hair growth; children with viral encephalitis and inter-brain barrier after mumps-induced systemic hairy; head injury, especially after the children more prone to hair; children with primary malnutrition or enteropathy, or other malabsorption, or severe infections caused by malnutrition could rise to a large number of multi-body hair; anorexia nervosa patients in the face, trunk and upper limbs have more hair grow; infant limb pain patients in the limbs, face, trunk and more hair; dermatomyositis patients, mainly in children can be located hairy forearm, leg and temporal, but also a broader scope; Berordinelli syndrome, that is, growth and maturity accelerated from an early age, and accompanied by malnutrition and intestinal muscle atrophy, common hepatomegaly and high blood fat, the skin of patients with rough, often hairy; addition of certain drugs such as streptomycin, such as cortisone can cause iatrogenic hairy; some women as a result of adrenal, ovarian and other diseases caused by increased androgen can also result in more hair.

如炎症性关节,小腿慢性静脉机能不全等导致长期持续的皮肤充血造成多毛;甲状腺机能亢进者在胫前粘液性水肿的斑块处常有粗毛长出;甲状腺机能减退的儿童背部和四肢外侧有大量毛发生长;儿童在病毒性脑炎及流行性腮腺炎后间脑障碍所致的全身性多毛;头部外伤后特别是儿童易发生多毛;儿童原发性营养不良或肠病,或其他吸收不良,或严重感染引起的营养不良均可引起大量全身性多毛;神经性厌食症患者可在面部、躯干和上肢有较多的毛发长出;婴儿肢痛病患者可在四肢、面部、躯干部多毛;皮肌炎患者,主要发生于儿童中,多毛可位于前臂、小腿和颞部,但范围也更广泛;Berordinelli综合征,即生长和成熟从小就加速,并伴有肠营养不良和肌肉萎缩,常见肝肿大和高血脂,其患者皮肤粗糙,常多毛;此外某些药物如链霉素、可的松等可造成医源性多毛;某些妇女因肾上腺、卵巢等疾病致雄性激素增多也可造成多毛。

The principal microscopic lesions were the inflammation,and necrosis of the mucoase membranes of proventriculus,degeneration of the parenchymatous cells o...

主要的组织学变化为腺胃粘膜上皮坏死脱落、固有层水肿、有炎性细胞浸润、肌层有淋巴细胞浸润、肌细胞坏死、腺小叶腺细胞坏死、核浓缩。

By disrupting the impermeability of the sarcolemma, extracellular cations and fluid flow down the electrochemical gradient into sarcoplasm, overwhelming the capacity of the cationic extrusion pumps14–18 and leading to swelling of the myocytes.

肌纤维膜的不渗透性被破坏,细胞外阳离子与液体向肌浆流入,超过阳离子的泵出能力,导致肌细胞水肿。

Background: Sclenus anticus syndrome is a disease caused by scalene anterior muscle edema, proliferation and spasm, lifted the first rib, resulted in scalenus muscle interspaces constriction, crushed the brachial plexus and subclavicalar A.V.

背景:前斜角肌综合征是指各种原因引起前斜角肌水肿、增生,痉挛并上提第一肋,导致斜角肌间隙狭窄,卡压穿行其间的臂丛神经及锁骨下动静脉而引起相应临床症状的疾患。

However, it has aperitenon whose vascular system extends both within and outsidethe tendon. Achilles peritendinosis manifests at MR imagingas linear or irregular areas of altered signal intensity inthe pre–Achilles tendon fat pad, a finding that indicatesthe presence of edema ( Fig 13), or scarring of the peritenon.

然而它有肌膜囊,其血管系统延伸到肌腱的前后方,跟腱肌膜囊在MR图像上表现为肌腱前脂肪垫出现不规则或线状改变,这反映肌膜囊的水肿或瘢痕化。

The injured gill cells had a different ultrastructure, with endoplasmic reticulum dilated and vesiculated, mitochondrial cristaes partially disintegrated, gill fibre cuticles denatured and dropped. The ultrastructural damages to hepatopancreatic cells were characterized by the decrease of microvilli, swelling and breaking up of mitochondria, expansion of endoplasmic reticulum, disruption and vacuolization of nucleus, tumefaction of nuclear membrane and increase of lipid droplets. As for the toxicological changes of myocardial cells, the mitochondria were swelling, having a lot of vacuolus and disintegrating, while the myofibrils were irregular with endoplasmic reticulum dissolved.

鳃细胞的损害表现为鳃丝水肿,细胞器溶解,角质层变薄,线粒体、内质网的肿胀、解体;肝胰脏细胞的主要损害特征为肝管微绒毛减少,线粒体水肿解体,内质网扩张,细胞核空泡化,核膜水肿和脂肪滴增加;心脏细胞的毒理变化为线粒体内嵴肿胀、瓦解,肌原纤维不规则,内质网溶解。

Results Group C had the symptoms such as reduced activity, acceded, indulge in lying and weight loss after 3 weeks of immune injection, 14 out of 16 SD rats in Group B had the same symptoms as Group C after 4~5 weeks of immune injection, the serum enzymes in model groups increased significantly compared with those of the control group, model group C was much higher than model group B; the duration shorted, amplitude decreased, multiphase wave increased in electromyogram of model groups; MRI examination revealed samples from model group B and C had one positive case each, which presented T1MI isodensity or hypodensity signal, T2MI and STIR serial hyperdensity signal, revealing muscle inflammation; all rats'skeletal muscle from model group C and 11 out of model group B had pathological changes, which exhibited striated muscle focal fiber degeneration, necrotized and inflammatory cell infiltration, interstitial vessel wall thickening, random cardiac muscle samples had 3 positive changes, which had similar changes to skeletal muscle, there was 1 positive change from lung sample.

结果:模型C组于免疫注射第3周左右开始出现活动减少,倦怠嗜卧,食欲体重下降等表现,模型B组有14只SD大鼠于免疫注射第4~5周出现上述症状,较C组为轻;模型组肌酶谱中,肌酸激酶、乳酸脱氢酶、谷草转氨酶与对照组比较明显升高,模型C组较模型B组升高更显著;模型组肌电图时限缩短,波幅降低,多相波增多;磁共振检查模型B组和C组选送标本中各有一例有阳性改变,表现为T1MI等或稍低信号,T2MI及STIR序列为高信号,提示肌肉炎症水肿改变;模型B组有11只,模型C组全部大鼠骨骼肌出现病理改变,表现为横纹肌局灶性分布的肌纤维变性炎细胞浸润,间质小血管壁增厚、扩张,随机选送的心肌标本中有3例有阳性改变,表现与骨骼肌相仿,选送的肺标本中有1例有阳性改变,表现为蛋白渗出,炎症细胞浸润和小血管改变。

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