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肌内膜

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Useful tests may include full blood count and erythrocyte sedimentation rate, as well as testing for endomysial antibodies.

有效的检查包括全血细胞计数、红血球沉降速率和人肌内膜抗体检测。

Results The anatomic structures of "Jianzhongshu" for perpendicular insertion downward were skin, superficial fascia, trapezius, rhomboideus, splenius cervicis, erector spinae, suprapleural membrane and cupula of pleura in order.The anatomic structures of "Dazhu" were skin, superficial fascia, trapezius, rhomboideus, upper posterior serratus, splenius cervicis, erector spinae, levator ribs, internal intercostal membrane and costal pleura in order.

结果 肩中俞向下直刺的解剖结构依次是皮肤、浅筋膜、斜方肌、菱形肌、颈夹肌、竖脊肌、胸膜上膜、胸膜顶;大杼穴向下直刺的解剖结构依次是皮肤、浅筋膜、斜方肌、菱形肌、上后锯肌、颈夹肌、竖脊肌、肋提肌、肋间内膜、肋胸膜。

Results Compared with control group,the level of serum testosterone in medol group was increased obviously(P.01),the expressions of TGF-β1 in the endometrium and myometrium were decreased significantly(P.01), the expression of TGF-βRⅠ in myometrium was decreased obviously(P.05),while the expression of TGF-βRⅠin endometrium was increased significantly(P.05). In model group ,myometrium of the uterus was tenuis,endometrium was atrophic and the number of glandular organs was decreased.

结果:与正常组比较,ASR模型组大鼠血清T水平明显增高(P.01;子宫内膜、肌层TGF-β1表达量明显降低(P.01);肌层TGF-βRⅠ表达量明显降低(P.05),而内膜TGF-βRⅠ表达量却明显升高(P.05);与正常组比较,ASR模型组大鼠子宫肌层变薄,内膜萎缩,腺体减少。

Results The anatomic structures of "Jianzhongshu" for perpendicular insertion downward were skin, superficial fascia, trapezius, rhomboideus, splenius cervicis, erector spinae, suprapleural membrane and cupula of pleura in order.The anatomic structures of "Dazhu" were skin, superficial fascia, trapezius, rhomboideus, upper posterior serratus, splenius cervicis, erector spinae, levator ribs, internal intercostal membrane and costal pleura in order.

结果 肩中俞向下直刺的解剖结构依次是皮肤、浅筋膜、斜方肌、菱形肌、颈夹肌、竖脊肌、胸膜上膜、胸膜顶;大杼**向下直刺的解剖结构依次是皮肤、浅筋膜、斜方肌、菱形肌、上后锯肌、颈夹肌、竖脊肌、肋提肌、肋间内膜、肋胸膜。

In the normal uterus, Cytokeratins immunolabelling were detected in glandular cell, luminal epithelial cell, Vimentin immunolabelling were detected in stromal cell and endoblastic cell; CK7 immunolabelling were not detected in any tissue of the yak utenus.

研究结果显示:未妊娠时,泛角蛋白在子宫内膜腺上皮细胞、腔上皮细胞内表达,波形蛋白在子宫内膜基质细胞内表达,平滑肌肌动蛋白在子宫平滑肌和血管平滑肌内表达,牦牛子宫任何部位均不表达角蛋白7;妊娠30天左右时,泛角蛋白在子宫内膜腺上皮细胞、子宫内膜腔上皮细胞、滋养层细胞、内胚层细胞和尿囊细胞内表达,波形蛋白在子宫内膜基质细胞和内胚层细胞内表达,平滑肌肌动蛋白在子宫平滑肌和血管平滑肌内表达,角蛋白7在尿囊细胞内表达,偶尔在腔上皮细胞的细胞核边缘表达;消化法进行原代培养时,组织经胶原酶消化并通过100目和400目筛网组合可以有效地分离原代子宫内膜基质细胞和子宫内膜腺上皮细胞;分离得到的子宫内膜基质细胞活率达90%以上,并可在体外传代7次以上;分离得到的子宫内膜腺上皮细胞活率可达85%以上,并可在体外传代5次以上;RPMI1640培养基最适合子宫内膜基质细胞和子宫内膜腺上皮细胞的生长,维持子宫内膜基质细胞正常生长的FBS添加量为20%,维持子宫内膜腺上皮细胞正常生长的胎牛血清添加量为30%。

A ery good screening procedure for celiac disease is serum IgA antigliadin antibodies and/or IgA endomysial antibodies.

乳糜泻诊断的最好的筛查手段是检测血清抗麦胶蛋白抗体和或肌内膜抗体的IgA。

In this context, we have reported recently that the volume of myocardial tissue occupied by perimysial and endomysial collagen was lower in hypertensive patients with HF and depressed ejection fraction, than in hypertensive patients with HF and preserved ejection fraction and normotensive subjects.56 Of interest, the interstitial and perivascular accumulation of collagen fibres was abnormally high in the two groups of patients.

在这种情况下,我们已经说了占用体积perimysial心肌组织人肌内膜胶原水平低的高血压患者用高频和沮丧的射血分数在高血压患者的心力衰竭和保存被罚出场分数与正常血压subjects.56舒张压的利息、间质和血管堆积过高的胶原纤维是在两组病人。

A clear cause-and-effect relationship between excessive myocardial MMP-1 activity, increased mysial collagen degradation, and progression to systolic HF has been shown experimentally through the use of transgenic models and the use of pharmacological MMP activators.53,54 In addition, experimental evidence has been provided showing that following chronic neurohormonal activation, increased synthesis and release of MMPs into the local extracellular matrix of the cardiomyocyte occurs,55 which in turn could contribute to endomysial and perimysial collagen degradation and disruption.

一个明显的因果过多的心肌MMP-1关系活动, mysial增加胶原蛋白的降解和进展收缩性心力衰竭已被证明试验,通过使用转基因模型和使用的MMP药理作用此外,activators.53,54实验证据下面被提供显示慢性神经激活时,增加的MMPs合成和释放到当地的细胞外基质的心肌细胞发生,55反过来有助于人肌内膜和 perimysial胶原退化和破坏。

The pooled prevalence of positive immunoglobulin A–class antigliadin antibodies was 4.0%(95% CI, 1.7%– 7.2%), the prevalence of positive endomysial antibodies was 1.63%(95% CI, 0.7%– 3.0%), and the prevalence of tissue transglutaminase was 4.1%(95% CI, 1.9%– 7.0%).

阳性免疫球蛋白A抗体盛行率为4.0%(95% CI自1.7%-7.2%),肌内膜抗体阳性率为1.63%(95% CI为0.7%-3.0%),且组织转麸氨酸酶盛行率为4.1%(1.9%-7.0%)。

The pooled prevalence of positive immunoglobulin A–class antigliadin antibodies was 4.0%(95% CI, 1.7%– 7.2%), the prevalence of positive endomysial antibodies was 1.63%(95% CI, 0.7%– 3.0%), and the prevalence of tissue transglutaminase was 4.1%(95% CI, 1.9%– 7.0%).

阳性免疫球蛋白A抗体盛行率为4.0%(95% CI自1.7%-7.2%),肌内膜抗体阳性率为1.63%(95% CI为0.7%-3.0%),且组织转麸氨酸螨盛行率为4.1%(1.9%-7.0%)。

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