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Results Gene transcripts of key components of RAS were found in the esophageal musculature.

结果 RAS基因转录的主要元件在试管组织中被发现,免疫化学方法显示了束和血管壁中分离的血管紧张素2-1型受体染色。

It is a pressurized, thick-walled shell with an internal and external musculature, a remarkably complex internal vascular system.

眼睛是眼内和眼外产生压力的系统,具有复杂的内血管系统,产生流体和溶合物的传导系统。

However, these abnormalities have not been found in similar studies of patients with rheumatoid arthritis and with trapezius myalgia (LefBer et al 2002a,b).

但是,这些异常都没有发现类似的研究在类风湿关节炎患者与斜方痛( LefBer等人2002年,二)。

Clinical features including swallowing,myalgia, muscle weakness, skin impairment and prognosis.

临床特点(吞咽、痛、无力、皮肤损害等)及其愈后。

Objective To observe the clinical manifestations and treatment of ocular myasthenia gravis.

目的探讨眼型重症无力的临床特点及治疗。

Objective To evaluate the change of myasthenia gravis during radiotherapy for patients with malignant thymomas.

目的 评价合并重症无力的恶性胸腺瘤患者放疗中重症无力的变化情况。

Familiar with: treatment and diagnosis of myasthenia gravis crisis.

熟悉重症无力的治疗及重症无力危象的诊断。

To determine the role of recombinant human ciliary neurotrophic factor in myogenesis, we observed the effects of rhCNTF (0-10 ng/ml) on myoblast differentiation of adult human in vitro.

为探讨外源性重组人睫状神经营养因子在成细胞分化中的作用,实验观察了0-10 ng/ml rhCNTF 对成人成细胞体外分化的影响。

In conclusion, the results indicated that the calpastatin level was modulated by certain growth and differentiation factors and its continuous presence resulted in the inhibition of myoblast fusion. Calpains may play an important role in cytoskeletal reorganization that is requisite for myoblast fusion.

结果表明,细胞内calpastatin的含量是由某些生长与分化因子调控的,calpastatin的连续出现抑制了成细胞的融合,说明calpains在成细胞融合进程中,对细胞骨架的重组起关键性地作用。

The effect of leptin on promoting myoblast proliferation and cell accumulation in the S+G2/M phase were inhibited by TNF-α.

脂肪细胞因子TNFα抑制leptin对猪骨骼细胞增殖和增殖期S+G2/M阶段细胞积聚的促进作用;降低leptin对猪骨骼细胞分化期间对CK活性和MyoG表达的抑制效应。

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