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SD rat emotional stress was simulated with the communication box model. The microstructural and ultrastructural pathological change in the TMJ and external pterygoid muscle was observed to show the emotional stress influence to TMJ structure and metabolism. 90 SD rats were randomly divided into control group, emotional stress group and foot-shocked group in average.

鉴于此,本课题采用了可以模拟应激作用下人类的紧张和焦虑状态的交流箱大鼠心理应激模型,使SD大鼠处于紧张和焦虑状态,研究在心理应激状态下,大鼠TMJ的超微结构、TMJ神经相关肽的表达、以及与TMJ关系密切的咬、颞电变化。

This suggests that the model is not very sensitive to the changes of primary force and primary length of individual horizontal rectus.

结果表明,上述两个指标变化在10%范围内时,手术矫正量的变化小于1.2%,水平直的初张力变化在30%范围内时,手术矫正量变化小于4%,说明本文提出的模型对个体张力的差异和个体眼外解剖长度的差异不是很敏感。

Identify the inferior edge of the vastus medialis and bluntly dissect it off the periosteum and intermuscular septum for a distance of 10 cm proximal to the adductor tubercle. Identify the tendinous insertion of the muscle on the medial patellar retinaculum Fig.

先确认股内侧下缘,将其从骨膜和间隔表面钝性分离直至收结节近端10cm处;再确认肉在髌内侧支持带上的腱性附着点(图1-41C)。

Identify the inferior edge of the astus medialis and bluntly dissect it off the periosteum and intermuscular septum for a distance of 10 cm proximal to the adductor tubercle. Identify the tendinous insertion of the muscle on the medial patellar retinaculum Fig.

先确认股内侧下缘,将其从骨膜和间隔表面钝性分离直至收结节近端10cm处;再确认肉在髌内侧支持带上的腱性附着点(图1-41C)。

Clinical diagnostic considerations ranged from benign entities such as rhabdomyoma, intramuscular lipoma, fibromatosis, myositis ossificans, proliferative myositis, inflammatory myofibroblastic tumor, and inflammatory myopathy to malignant entities such as rhabdomyosarcoma, leiomyosarcoma, liposarcoma, and lymphoma.

这些病例的临床考虑多样,有良性的横纹瘤、内脂肪瘤、纤维瘤病、骨化性炎、增生性炎、炎性纤维母细胞瘤和炎性病,也有恶性的横纹肉瘤、平滑肉瘤、脂肪肉瘤和淋巴瘤。

Transcription, western, and immunohistochemical analysis showed increased levels of dystrophin transcript and protein, and correct localization at the sarcolemma.

用免疫组织化学分析用纳米作为载体进行寡聚核苷酸递送的实验鼠抗萎缩蛋白基因RNA和抗萎缩蛋白的水平得到提高,而且恢复了抗萎缩蛋白在肉细胞膜上的正确位置。

Deletions that terminate in in-frame codons, do not prevent translation of dystrophin and result in a truncated protein, still able to bridge actin and sarcolemma.

缺失的终止在框架密码子,并没有妨碍在截短的抗萎缩蛋白翻译和结果,仍然能够弥合动蛋白,膜。

From the viewpoint of DMD therapies, only when all components of DGC are restored simultaneously in the sarcolemma, the therapy will succeed.

从假肥大型营养不良症治疗的观点来看,无论成细胞移植治疗,还是基因治疗,都必须能够使细胞膜上所有DGC复合体蛋白同时恢复。

ObjectiveDuchenne and Becker muscular dystrophy is the most common human X-linked recessive inherited disease.The absence of dystrophin on the sarcolemma results in DMD,which is the essential physiopathologic mechanism.

假肥大营养不良是男性最常见的X-连锁隐性遗传病,而营养不良蛋白在DMD患者细胞上的表达异常是导致该病发生的最根本的病理生理机制。

In chapter one, the molecular architecture of the sarcomere, the structures of myosin and actin, and the mechanochemical actin-activated myosin ATPase cycle are introduced.

第一章主要介绍节的结构、球蛋白和动蛋白的分子结构、球蛋白工作循环中的机械化学偶联。

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But we don't care about Battlegrounds.

但我们并不在乎沙场中的显露。

Ah! don't mention it, the butcher's shop is a horror.

啊!不用提了。提到肉,真是糟透了。

Tristan, I have nowhere to send this letter and no reason to believe you wish to receive it.

Tristan ,我不知道把这信寄到哪里,也不知道你是否想收到它。