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老年性耳聋

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During the process of caducity, Hearing apparatus is characteristic of presbycusis.

老年性耳聋是人类衰老过程中在听觉器官中的表现,目前老年性耳聋发病机制尚未完全阐明,无确切有效的治疗方法。

Methods All archival serial celloidin sections of temporal bones are stored in the temporal bone bank of PLA general hospital.

方法所有颞骨火棉胶切片均来自解放军总医院颞骨库,基于颞骨火棉胶切片的DNA提取、PCR扩增、原位杂交、原位PCR和免疫蛋白染色技术被用来研究老年性耳聋的分子病理机制。

The research is focus on develop a new drug that is better curativeness and fewer side-effects.

大量的研究表明,老年性耳聋与耳蜗微循环障碍有密切关系,故临床上使用改善微循环的药物治疗老年性耳聋本实验研究使用豚鼠自然衰老模拟老年性耳聋,探讨老年性耳聋的发病机制,观察耳蜗毛细胞的形态学改变和听觉生理学变化。

Objective To study the relationship among the levels of SOD、MDA and RCD in patients with presbycusis.

目的 探讨老年性耳聋SOD、MDA及红细胞变形性之间的关系,为临床上能有效地控制和延缓老年性耳聋的发生和发展提供依据。

Perhaps the most common cause of nerve deafness is presbycusis.

也许神经性耳聋最常见的原因是老年性耳聋

Presbycusis may cause a severe lose of the ability to discriminate sound.

老年性耳聋可以引起完全丧失辨别声音的能力。

Methods The Red Cell Deformabilityand the levels of the blood plasma SOD and MDA in 68 patients with presbycusis and in 50 healthy old people were measured.

测定68例老年性耳聋病人和50例健康老年人的血浆超氧化物歧化酶和脂质过氧化产物丙二醛的水平及血液红细胞变形性。

With the improvement of our living and health, our world is stepping into aging society, Presbycusis is a barrier of our normal life and communion.

随着人们生活和健康水平不断提高,社会逐步步入老龄化,老年性耳聋成为影响老年人正常生活和社会交往的一大障碍。

These results suggest that the decrease of spiral ganglion cells and the deposition of lipofuscin in the aged may be the main cause of presbycusis.

老年人螺旋神经节细胞减少,细胞质内脂褐素沉积,可能是导致老年性耳聋的主要原因之一。

Presbycusis might result from the decreased SOD and RCD,and the increased MDA in some extent.

老年性耳聋病人的SOD含量下降,红细胞变形能力降低,而丙二醛水平增高,建议在临床上对老年性耳聋病人应用增加SOD含量,提高红细胞变形能力,减少MDA含量的药物。

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