缺氧的

He was inside the bubble nakedly and inhaling from a plastic pipe until it rearched human body's limitation under the pressure and being short of oxygen.

他的裸体被封闭于其中，他从一个胶管不断的吸进空气，塑料气泡中的压力和缺氧的状态一直大到人体所能承受的极限。

China Branch Shenwei developed out of the latest generation of HK series of plant gas furnace, by the barrel, purification equipment, pipes, stove and other components, plant fuel through the system of gas furnace, in a confined hypoxic conditions, the use of pyrolysis pyrolytic chemical oxidation reaction to produce a combustible gas, in support of nitrogen and oxygen the burning of the combustion to achieve the desired effect.

武汉华科神威科技发展有限公司研制开发出的最新一代HK系列植物燃气炉，由料筒、净化装置、管道、灶头等部分组成，植物燃料通过制气炉，在密闭缺氧的条件下，采用干馏热解化学氧化反应后产生一种可燃性气体，在氮气的支持和氧气的助燃达到理想的燃烧效果。

The experiments showed a significant increase in Bax mRNA levels (P.01) in neurons cultured at severe hypoxia condition compared with those cultured at normoxia. In hypoxia groups, tolbutamide increased Bax mRNA expression (P.05), while diazoxide reduced it (n=5 for each).

缺氧+二氮嗪组中基因Bax的mRNA表达水平与单纯缺氧组相比下降(P.01)，缺氧＋甲苯磺丁脲组中基因Bax的mRNA表达水平相对于单纯缺氧组也有提高(P.05)。

Methodological design, synthesis is aimed at anoxic and revulsive factor - the nucleotide of few of particularity short cable length of 1 α is acerbity, compose builds anoxic and revulsive factor - of ShR-NA of 1 α particularity recombine plasmid, stability turns catch; of cell of colonic cancer SW 480 to use chloridize cobalt preparation is anoxic and revulsive education radical, imitate tumour is anoxic condition.

方法设计、合成针对缺氧诱导因子-1α的特异性短链寡核苷酸，构建缺氧诱导因子-1α特异性shR-NA的重组质粒，稳定转染结肠癌SW 480细胞；采用氯化钴制备缺氧诱导培养基，模拟肿瘤缺氧状态。

Methods 20 rats were randomly divided into A, B, C, D and E group. The models of cerebral anoxia in group A and C were made by hypopiesia and hypoxia, and the models of hypoxia-reoxgenation were made in group B and D. 24h and 1h before establishment of models, ginsenoside Rb3 was injected peritoneally in rats of group C and D, respectively. The number and form of GABA-like immunoreactive neurons in hippocampus CA1 area of rats after brain hypoxia-reoxgenation were investigated with immunohistochemistry.

將大鼠隨机分为A、B、C、D、E5组，A、C组以低压低氧暴露製作大鼠脑缺氧模型，B、D组为缺氧－复氧模型。C、D组动物分别於制模前24h、1h 2次腹腔注射人参皂甙Rh3溶液；採用免疫组化法，观察各组大鼠在缺氧及缺氧－复氧处理后海马CA1区GABA免疫反应阳性细胞形態及数量的变化，並与正常对照组比较。

Outcome:(1) Acute ischemic damage may induce the expression of c-fos protein in Cerebral Cortex quickly. Also, the expression increases by the ischemia time delay. It may hint that ischemia injury can rise the ability of nerve cells to tolerate ischemia and anoxia. And the expression level of c-fos can be up-regulated by putting blood of 12 hand Jing Points. So we think that treated by this therapy can rise the irritability of brain to resist the development of ischemia and enhance the ability to plerosis.(2) Acute ischemic damage may induce the expression of HspVO in Cerebral Cortex quickly. Also, the expression increases by the ischemia time delay. It may hint that ischemia injury can rise the ability of nerve cells to tolerate ischemia and anoxia.

三 结果(1)急性缺血性脑损伤可快速诱导c-fos蛋白在皮层缺血区脑组织的表达，其表达有随缺血时间延长而逐渐增多的趋势，提示脑的缺血性损伤可应激性地增强神经细胞对缺血、缺氧的耐受和适应能力，而应用井穴放血的干预治疗可明显上调缺血区皮层脑组织c-fos的表达水平，进一步增强脑组织对抗缺血性损伤的应激能力，抵抗缺血性脑损害的进一步发展。

The transition from the systemic inflammatory response syndrome to seere sepsis and septic shock inoles a myriad of pathogenic changes, including circulatory abnormalities that result in global tissue hypoxia.1,2 These pathogenic changes hae been the therapeutic target of preious outcome studies.12 Although this transition occurs oer time, both out of the hospital and in the hospital, in outcome studies interentions hae usually been initiated after admission to the intensie care unit.12 In studies of goal-directed hemodynamic optimization, in particular, there was no benefit in terms of outcome with respect to normal and supranormal hemodynamic end points, as well as those guided by mixed enous oxygen saturation.9,13 In contrast, een though we enrolled patients with lower central enous oxygen saturation and lower central enous pressure than those studied by Gattinoni et al.9 and with a higher lactate concentration than those studied by Hayes et al.,13 we found significant benefits with respect to outcome when goal-directed therapy was applied at an earlier stage of disease.

从全身炎症反应综合征转为严重脓毒症和脓毒性休克牵涉大量发病的变化，包括导致全身性缺氧的循环紊乱。这些发病的变化成为先前结果研究的治疗学目标。尽管这些转变随着时间的流失而逐渐出现，既可以在院外，也可在院内，在结果研究中，干预措施往往在进入ICU之后才启动。特别的，在那些目标导向血流动力学最优化研究中，依照结果，那些以正常或超正常血流动力学为终点的措施未能显示出益处，以混合静脉血氧饱和度为导向的研究同样如此。

Basic characteristics and prevailing genetic models of Chondrites have been introduced and commented. Unusu-al features of Chondrites from the Lower-Middle Ordovi-cian transition have been reported in the west-central Utah, USA. They include that tiny Chondrites -were cut by other trace fossils associated with them and formed in softground, and that Chondrites were the earliest or shallowest tiering in the complex ichnofabrics.

介绍和评述Chondrites的基本特征和流行的成因解释，记述最近发现于美国犹他州中西部北美下-中奥陶统之交层型剖面上的Chondrites遗迹组构的不寻常特征和属性：细小的Chondrites被所有与之共生的遗迹化石交切，形成于水与沉积物界面附近缺氧的早期软底质中，Chondrites是形成最早和最浅的遗迹阶层。

Molds are aerobes, and are controlled by the absence of oxygen, Bacterial populations which will dominate a substrate may be manipulated by their oxygen requirements and its availability..

霉菌是需氧的，它受缺氧的控制。基质的控制。基质中占主导地位的各种细菌的菌数分布受各菌的需氧情况以及获得氧的难易的控制。

Mild late decelerations are a reflex central nervous system response to hypoxia, while severe late decelerations suggest direct myocardial depression.

轻度的晚期减速是中枢神经系统对缺氧的反应，而严重的晚期减速提示直接的心肌受抑。

This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。