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ObjectiveSevere traumatic patients always had a severe blood loss, for maintaining blood supply of heart and head, the gut would appear ischemic and anoxemia through the mechanisms of intestinal ischemic, reperfusion and release of mediators of inflammation and so on, which could cause functional impairment of intestinal barrier and translocation of intestinal bacterium and endotoxin, accordingly, induce the occur of SIRS (systemic inflammatory response syndrome), sepsis and MODS (multiple organ dysfunction syndrome).

目的严重创伤常伴严重失血,机体为维持心、脑的血供,肠道首先出现缺血缺氧,通过肠道缺血、再灌注、炎症介质的释放等机制,引起肠道屏障功能障碍,肠内细菌及内毒素移位,从而导致SIRS、脓毒症和MODS的发生。

Automobile exhaust the highest level of carbon monoxide, it can enter through the respiratory tract of gestation, the blood was absorbed, combined with hemoglobin to form carboxyhemoglobin, lowering blood oxygen, blood weaken the oxygen supply of human tissues, resulting in tissue hypoxia, which led to headaches and other symptoms, or suffocation.

汽车尾气中一氧化碳的含量最高,它可经呼吸道进入肺泡,被血液吸收,与血红蛋白相结合,形成碳氧血红蛋白,降低血液的载氧能力,削弱血液对人体组织的供氧量,导致组织缺氧,从而引起头痛等症状,重者窒息死亡。

CPR, or cardiopulmonary resuscitation , can save a life and reduce the risk of brain damage from loss of oxygen.

心脏复苏术或是心肺复苏,可以挽救一个人的生命,减少因缺氧引起的脑损伤的风险。

Transmission electron microscope showed the evident damage of hypoxic cardiocyte ultrastructure after blocking HSP90: caryotheca crenation, chromatin margination and mitochondria vacuole.

(3)透射电镜显示阻断HSP90后的缺氧心肌细胞超微结构不但出现细胞肿胀,肌丝排列紊乱等细胞受损的表现,还可以看到核膜皱缩,染色质边集,线粒体出现空灶化等细胞凋亡的特征。

Pillars and the role of providing breathing, so that mangroves can grow well in hypoxia and soft mud beach, the waves dash against.

提供支柱与呼吸的作用,从而使红树林能很好地生长在缺氧及松软的淤泥滩涂上,抵御风浪的冲刺。

We hypothesized that nitrite would have a greater dilator effect in capacitance than in resistance vessels because of lower oxygen tension and that resistance-vessel dilation should become more pronounced during hypoxemia.

我们猜测,因为较低的氧分压,容量血管和阻力血管相比,对亚硝酸盐有更强的舒张作用。而在缺氧状态下,阻力血管的舒张更加明显。

Erythrocytosis can arise from deregulation of the erythropoietin axis resulting from defects in the oxygen-sensing pathway.

缺氧信号传导途径的缺陷导致的红细胞生成素轴调控异常会引起红细胞增多症的发生。

No ferromagnetism was observed in stoichiometric HfO2 films, but we observed weak ferromagnetism in highly oxygen-deficient HfO2 films.

对于理想配比的HfO2薄膜,没有发现铁磁行为,但在高缺氧状态下的HfO2则观测到了弱的铁磁性。

The relevant kinetics model was constructed based on the mass balance theory,combined with Monod equation and Fick law.

对A/O复合式生物膜工艺的缺氧段进行了深入研究,并以物料平衡理论为基础,结合莫诺方程和Fick定律,建立了相应的底物降解和微生物增殖的动力学模型。

Hypoxia resulting from apnea in patients with sleep apnea is an important factor in heart disease.

在睡眠呼吸暂停的病人中,由于呼吸暂停导致的缺氧是心脏病的一个重要因素。

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