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Methods: One hundred and sixty SD rats were randomly divided into four groups (40 in each group), and orally given the medicine with doses of 50, 25, or 12.5 g/kg of crude drug, or with bean oil of 0.5%CMC-Na daily for 26 weeks.

160只SD大鼠,随机分为高、中、低剂量组及空白对照组,雌雄各半,每日灌胃1次,连续给药26周,观察大鼠的一般状况、体重、摄食量和给药13

Daily administration of 10g/kg for 3 weeks rendered no significant changes in the liver function, blood picture,EKG,and pathologic examination.

每日灌胃10g/kg,共4周。在给药第2周降压作用开始显著,继续给药则降压更多。每周平均舒张压低时分别下降32,17,6,20,12mmHg(25%,14%,5%,16%,10%)。

Eighty SD rats were randomly divided into four groups: acute spinal cord injury groups ( SCI groups, n=20);administration of Ulinastatin (U groups, n=20), use Ulinastatin after SCI at half an- and 4- and 8- and 24- and 48- hour.;administration of Methylprednisolone (M groups, n=20), use Ulinastatin after SCI at half an- and 4- and 8- and 24- and 48- hour; administration of Ulinastatin and Methylprednisolone(M+U groups, n=20), use Ulinastatin and Methylprednisolone after SCI at half an- and 4- and 8- and 24- and 48- hour;Allen's weight drop model of acute spinal cord injury was prepared.

本课题利用Allen重物打击模型,选用SD大鼠80只,随机分为四组:脊髓损伤组(SCI组,n=20);使用乌司他丁组(乌组,n=20);使用甲基强的松龙组(甲组,n=20);乌司他丁+甲基强的松龙联合用药组(乌+甲组,n=20),所有给药组在脊髓损伤后(半小时、4小时、8小时、24小时、48小时)给药

In the case series, ascorbate was given orally and intravenously, but in the trials ascorbate was just given orally.

虽然受益病例中的维生素C采用口服和静脉给药,但在证明其无效的试验中,抗坏血酸只采用口服给药

ObjectiveTo probe into the hyperplasy effect of different curcumine administered approaches for the splenic lymphocyte of mice.

目的探讨姜黄素3种不同给药途径对小鼠脾淋巴细胞增殖的影响。方法通过噻唑蓝比色法比较姜黄素3种给药途径对小鼠脾淋巴细胞增殖的影响。

The mechanism of this therapeutic effect of liver fibrosis will be discussed.Method:Complex factors induced-LF rats were adopted, and dimidiated into preventing and therapy groups.

本研究采用复合因素大鼠肝纤维化模型,分预防给药和治疗给药两大实验组,每组又分为正常对照、模型对照、中药低剂量、中药高剂量、阳性对照5组。

Dy- namic levels on alanine aminotransferase,tumor necrosis factor and DHBV-DNA in sera of the duckings were determined.

给药组在第2次注射 LPS 后9、18、24、48和72h,与未给药组同期比较,血清 ALT 水平显著降低,血清 TNF 水平也明显下降,但血清 DHBV-DNA 水平无明显变化。

Results Bile flow, serum levels of total bile acid and gamma glutamyl transpe...

而给予SAMe的Ⅲ、Ⅴ组胆汁流升高,血清总胆酸和γ GT水平明显降低,肝脏病理结果正常、毛细胆管不扩张、无胆栓;临床 16例患者给药前表现为胆汁瘀积和肝功能损害,给予SAMe 3周后血总胆红素、碱性磷酸酶、γ GT、谷丙转氨酶、谷草转氨酶均较给药前的异常水平有显著下降,第 4周恢复至正常水平。

Methods We fed the mice with the maximum administration concentration drug for 7 days, and gavage administration was done for three times within 24 h.

以最大浓度药液灌胃给药,24 h内给药3次,连续观察7天。

After the rat hepatocytes were treated with iron citrate fisrt followed by CAT and FIS 24h later, it was found that the content of SOD increased, the release of AST and ALT was restrained, the level of LDH and the content of ALB decreased.

实验表明,分别对致损伤前和致损伤后的大鼠原代肝细胞给以CAT和FIS,发现在致损伤后给药效果好于致损伤前给药

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