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结构破坏

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Compared with the temperature of 15℃, the results showed that the RWC and the CAT activity decreased significantly, MDA and PRO content increased obviously under high temperatur, then cell membrane-lipid peroxided, membrane structure be destroyed, physiologic function disordered and resistance decreased, these were main reasons of tall fescue over summering hard and get ill easily.

结果表明:在高温下,以15℃为对照,RWC含量和CAT活性明显下降,MDA和PRO含量明显增加,导致细胞膜脂过氧化,膜结构被破坏,生理功能紊乱,抗性下降,这是其越夏困难、易发病的主要原因。

Taimen is an important local wild commercial fishery species in Heilongjiang River. Due to severe damages to its population size, habitats, and spawning fields from overexploitation and environmental pollution. It is enlisted as an endangered species in the "China Red Data Book". To establish a scientific basis to conserve taimen, studies on the population genetic structure and phylogeography are necessary.

哲罗鲑是我国土著的重要经济鱼类,近些年来,由于环境的恶化及人类活动的加剧,已对其资源量、栖息地、产卵场等造成了严重的破坏,种群处于濒危状态,被列入濒危物种红色名录中,因此研究哲罗鲑的群体遗传结构、演化历史、分布动态等对其进行有效保护具有重要意义。

In addition, extensive swelling, erosion and peeling of tegumental ridges and destruction of discoidlike sensory structures were seen in female worms.

并查见皮层褶嵴有广泛和严重的肿胀、糜烂和剥落,以及雌虫盘状感觉结构的破坏。

Six patients with long-standing, terebrant basal cell carcinomas, deeply invading the orbital structures, with loss of sight of the invaded eye received cryosurgery.

冷冻治疗能有效地清除癌肿组织,对局部解剖和功能的破坏相对较小。6例眼眶部基底细胞癌,病变均穿破眼眶结构,引起失明。

Furthermore, it is also found that the addition of salts or sucrose can significantly reduce their thixotropic properties.

添加盐类与蔗醣於XG水溶液会明显改变溶液的摇变性质,加速剪应力对结构的破坏。

The nitrides with high specific surface area from oxide precursor can be prepared through temperature-programmed reduction via topotactic reaction in ammonia or nitrogen under strict reduction conditions.

在基本不破坏氧化物前驱体晶体结构和严格的氮化条件下,氧化物前驱体通过程序升温氮化经由&局部规整反应&可以制备出高表面积的过渡金属氮化物。

Furthermore, large-scale fold and fault structure hasn't been found around the Tianchi Lake, but the original prismatic joints and radial joints developed well in the trachyte, so the rock mass is dissected into fractured fragmental structure and the integration of rock mass is further destroyed.

在构造方面,长白山天池周围虽然没有发现大规模的褶皱断裂构造,但粗面岩中原生柱状节理、放射状节理十分发育,这些节理裂隙将岩体切割成十分破碎的碎裂结构,使岩体极易产生岩崩、落石,使岩体的完整性遭到了进一步的破坏。

The problems existed in the historical cultural relics and historic sites travel of our country at present analysed mainly in the article,namely:propagate uncompletely,products open the sale,product structure is single,the level of the product development is relatively low,the destruction to cultural relics and historic sites,etc.

目前我国历史文物旅游发展中存在诸多问题:宣传不到位,产品打不开销路;产品结构单一,产品开发层次较低;某些文物古迹遭到破坏。

The unmethylated probe can be cut in the recognition site by restriction endonuclease, causing the restoration of the fluorescence of TAMRA.

限制性内切酶可切割未发生甲基化修饰的探针,导致探针的发夹结构遭到破坏,引起TAMRA荧光信号的恢复。

One hand mechanical obstruct led to the increase of veinous resistance and the obstacle of microcirculation, the other hand the adhesive PMN was activated in excess, the white blood cells released a lot of enzymes, in which PMN-elastase can decompose the components of cell and many albumens, inclusive of immunoglobulin、alexin and fibrication. These components induced the injury of the pancreatic capillary vessels and cell and lysosome enzy made the tissue protein hydrolyze and produced unsaturated fatty acids, which destroyed the structure and function of cellar membrane. The inflammatory cellar factors activate other immunocytes to produce the injury and necrosis of tissue, which aggravated the pathological injury and led to shock、pyaemia and MODS. So ICAM-1 and LFA-1 played an important role in SAP. Frossard found that the expression of ICAM-1 in the rat model, especially in serum、pancreas and lung. All these showed ICAM-1 is an important factor in AP and concomitant lung injury.

胰腺小叶组织局部血管EC首先被激活,ICAM-1表达升高,与被激活的PMN表面表达的LFA-1相结合,&PMN-EC&相互作用加剧,一方面机械性阻塞毛细血管导致静脉阻力增加、微循环障碍;另一方面粘附的PMN过度吞噬或激活,当白细胞吞噬的颗粒不能被封闭隔离,连同细胞内的酶被释放出来,其中的PMN-elastase能够降解细胞基质中各种成分,水解多种蛋白,加重胰腺的毛细血管内皮细胞和腺泡的损伤;释放的溶酶体酶使组织蛋白水解,产生的不饱和脂肪酸引发脂质过氧化方应,破坏细胞膜的结构和功能;释放的炎性细胞因子,激发其他的免疫细胞的功能,导致进一步的组织损伤和坏死,加重SAP的病理损伤,最终导致休克、脓毒血症及多器官功能障碍等严重后果。

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