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细菌毒素

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Methods To remove the crust and transplant skin grart in 134 cases burn with toxemia or septemia,in order to decrease burn toxin, promote healing of wound and promote recovery of illness.

对134例烧伤合并毒败血症患者进行烧伤创面切削痂植皮,以消除细菌来源、减少烧伤毒素、促进创面修复、促进病情恢复。

Based on principle of pollen abortion, utilizing tapetum of anther cell, mitochondria of tapetum, callose well, process of cell meiosis during formation process of pollen, we adopt genetic transformation to create plant male sterility, such as, exogenous cytotoxin gene, chaperones, anti-sense RNA, degradation of callose ahead of time, disturbing information intercommunion between mitochondria and nucleolus and affected development of pollen sporule by bacteria.

根据花粉败育的原理,利用花药细胞绒毡层、绒毡层线粒体、胼胝质壁、细胞减数分裂过程等花粉形成过程中的不同环节,采取外源细胞毒素基因、分子伴侣、反义RNA技术、提早降解胼胝质、扰乱线粒体与细胞核之间的信息交流和细菌影响花粉小孢子发育等策略,使花粉不能正常形成,从而产生不育花粉,达到产生雄性不育的目的。

Function of 1 bowel protective screen and protective screen of its characteristic bowel include protective screen of alvine mucous membrane, chemical protective screen, biology protective screen and immune protective screen, its are main the function is to resist the bacterium inside alvine antrum and toxin enter system inside.

本文现就化疗药物对肠屏障功能的损伤机制及其防治进展作一综述。1肠屏障功能及其特征肠屏障包括肠黏膜屏障、化学屏障、生物屏障和免疫屏障,其主要功能是抵御肠腔内细菌及毒素进入体内。

Here we explore immunologic mechanisms of colonic carcinogenesis by a human colonic bacterium, enterotoxigenic Bacteroides fragilis.

通过研究人结肠细菌——产肠毒素脆弱类杆菌,探索结肠癌形成的免疫学机制。

In invasive clostridial infections, spores reach tissue either by contamination of traumatized areas Or from the intestinal tract.

组织肿胀影响血液的供应。细菌所产生的引起坏死的毒素和透明质酸酶均有利于感染的扩散。

Zeae, is one of the most important diseases in rice. Genetic and molecular characters of toxin producting for Erwinia chrysanthemi pv.zeae were conducted in this paper.

zeae是水稻上重要的细菌病害之一,本论文对该病菌的毒素遗传特性和产毒相关的分子标记进行了研究。

Mucous membrane protective screen is formed by the join between cell of alvine mucous membrane and cell, atrophy of cell of alvine mucous membrane, wither dies, cellular connective can send alvine path to connect flabbily appear a gender to increase, provide passageway; chemical protective screen for the bacterium inside bowel and toxin the digestive juice that shows epithelial excretive mucus and alvine memory are in bowel and digest enzymatic the chemistry that waits for material to reach its to develop.

黏膜屏障由肠黏膜细胞和细胞间的连接构成,肠黏膜细胞萎缩、凋亡、细胞连接的松弛可致肠道通透性增加,为肠内细菌及毒素提供通道;化学屏障指肠上皮细胞分泌的黏液及肠内存在的消化液和消化酶等物质及其发挥的化学功能。

Results After screening, 59 subtracted library clones were isolated which were specific for strain VIB72, and the DNA sequences of these clones were determined. Seventeen fragments showed high homology to the genes of known functions in other bacteria. This includes soluble lytic murein transglycosylase, mobilization protein, transposase (IS66), resistance-related protein (metallo-beta-lactamase and acetyltransferase family), toxin protein (DT-201 and alveicin A immunity protein), ATP-dependent endonuclease of OLD family like protein, SocE and GTP-binding protein HflX (high frequency of lysogenization).

通过对差减文库筛选,分离到59个对菌株VIB72的克隆,并对这些克隆的DNA序列进行了测定。17个基因片断与其它细菌的已知功能的基因有较高的同源性,其中包括可溶性溶胞壁质转糖基酶、转移蛋白MobA和MobC、转座子IS66、抑制相关蛋白(金属β-内酰胺酶和乙酰转移酶家族)、毒素蛋白(DT-201和alveicin A免疫蛋白)、与OLD 家族相似的ATP依赖性核酸内切酶以及SocE 和GTP结合蛋白HflX。

The toxin forms when the organisms sporulate in the gut, with the onset of diarrhea--usually without vomiting or fever--in 6-18 hours.

The illness lasts only 1-2 days在肠道中细菌形成芽胞时产生毒素,在6-8小时内引起无发热、无恶心呕吐的腹泻。1-2天后自愈。

Clostridium tetani forms tough spores that can survive for many years. If these spores get into a deep wound, they can germinate. Once this happens the germ produces a powerful poison called tetanospasmin toxin which damages the nervous system and can cause death.

破伤风梭菌形式严峻的孢子可以存活很多年,如果这些孢子进入一个深的伤口,他们可以发芽,一旦发生这种情况的细菌产生了强大的毒药,所谓的tetanospasmin毒素损害中枢神经系统,并且可以导致死亡。

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