细胞结构

1Unequal number of univalents with decreased degree of synapsis and lagging chromosomes occurred in meiosis.The abnormal chromosomal behavior correlated with heterogenicity gave rise to unequal allocation of homogenous chromosomes to daughter cells and resulted in the deletion of functional chromosoms,thus led to the occurence of a certion proportion of aborted pollens.(2)The interaction between genetic imbalance and such environmental factors as temperature,etc.,further drew forth physiological and even structrural imbalance,with abnormal development of pollen mother cells and tapetal cells,and consequently resulted in no or very few production of pollens.The difference in the interaction between environment and genotype reflected on the unstability of pollen abortion in different years.

1在减数分裂中有数目不等的联合程度较差的单价体以及落后染色体出现，这种与异质性相关的染色体行为异常，导致同源染色体向子细胞的不均衡分配，造成具功能染色体的缺失，从而引起毛白杨一定比率的败育花粉的产生；(2)遗传上的不平衡与温度等环境因子相互作用，进一步引发毛白杨生理乃至结构上的不平衡，花粉母细胞和绒毡层细胞发育异常，从而造成不产粉或产粉较少；环境与基因型互作的差异性导致了花粉败育的年度不稳定性。

Ambient salinity and dietary protein level only had significant interaction on the body ash content, while no significant interaction effects were observed in other parameters. Tubules of shrimp fed with CP30 and CP40 had the normal structure, though CP40 increased the number of R cells. Tubules of shrimps fed with CP20 had relatively fewer R cells and arranged incompactly. And the basal membranes of partial tubules were in disrepair. While tubules of shrimps fed with CP50 compactly arranged with much ambiguous materials in B cells.

饲料蛋白质含量明显影响了凡纳滨对虾肝胰腺的组织结构，投喂CP30和CP40饵料的对虾肝小体基膜完整，投喂CP40对虾的肝小体中还出现了大量的存储细胞；而投喂CP20饵料的对虾肝小体分布松散，R细胞数量较小，并且部分肝小体基膜破损；而投喂CP50饵料的对虾的肝小体排列紧密，且B细胞内出现大量内容物质。

Monocellular Raman spectrums of normal liver cell line (LO2) and carcinoma liver cell line (SMMC-7721) were studied with laser tweezers Raman spectrums system and each cell was investigated in three different points, the result of which showed; significant differences of average Raman spectrums were found between the normal cell and the carcinoma one; And, the spectra line of carcinoma cell became weak at the whole; Additionally, the spectral peak intensity ratio of normal cell at 1658cm^(-1) and 1450cm^(-1) was 0.63 while that of carcinoma was 0.99; Moreover, the structure and the amount of protein; nucleic acid, lipids and other such molecules had also changed.

本工作利用激光镊子拉曼光谱系统研究了人正常肝细胞株(LO2)和肝癌细胞株(SMMC-7721)的单细胞拉曼光谱，对于每个细胞在不同部位测三个点。实验结果显示：正常细胞和癌细胞的平均拉曼光谱存在显著差异；癌细胞谱线强度整体变弱；正常细胞的1658cm^(-1)处峰和1450cm^(-1)处峰的强度比值为0.63，癌细胞的为0.99；癌细胞的核酸、蛋白质、脂类等重要生物分子在结构或含量上都发生了不同的改变。

Immunohistochemically, cells in myoid/myofibroblastic areas showed positive staining for α-SMA, MSA and vimentin, but negative for desmin and CD34. Electron microscopic study displayed the presence of microfilament bundles, focal dense bodies and micropinocytic vesicles, consistent with those of myofibroblasts.

免疫组织化学标记显示肌样区域细胞表达α-平滑肌肌动蛋白和肌特异性肌动抗原，不表达CD34；电镜观察证实细胞含有质膜下微丝束、局灶性致密体及微胞饮囊泡样结构，与肌纤维母细胞相一致。

Other potential autologous chondrocyte sources in vivo include auricular, nasoseptal and costal cartilage with differences function, structure, and composition which result in different discrete biochemical, physical and biomechanical properties.

体内其他作为自体软骨细胞源包括耳骨、鼻中隔、肋软骨、颞颌关节髁突软骨等，而不同的软骨源具有不同的结构、组成和功能，产生具有不同生化、物理和生物力学特性的细胞及细胞外基质。

The cell processes showed up from the edge of explants and neuroglial cell migration was observed at the early stage.

培养早期上丘脑组织块边缘出现细胞突起，胶质细胞迁移，随着培养时间延长，细胞出现凋亡，组织结构破坏。

The adhesive capacity of differentiated cells that stably expressed E-cadherin was markedly enhanced. Compact cell connection and multi-layer growth state remained at 19 days. While non-transfected ESCs gradually changed from embryoid bodies into noncohesive cell populations.

稳定表达上皮型钙粘蛋白的分化细胞间黏附能力明显增强，并在19 d时仍能保持致密的细胞连接和多层生长状态，普通胚胎干细胞则由拟胚体结构逐渐分化为松散的细胞群落。

BACKGROUND: The essence of cell differentiation is a selectively intra-cellular gene expression, which results in specific proteinic synthesis and causes changes in biochemistry, structure and function. Thus, original proteomics and a single protein analysis can not meet the requirement in study.

摘要背景：细胞分化的实质是细胞内基因的选择性表达，导致细胞新的特异性的蛋白质的合成，从而在生化、结构及功能上发生变化，原有的基因组和分析单一蛋白的模式已不能满足研究的需要。

According to the previous results, BNIPL-2 could interact with Cdc42GAP and suppress the GAP activity of Cdc42GAP toward Cdc42. BNIPL-2 also had a BCH domain (BNIP-2 and Cdc42GAP homology), which could induce short and long pseudopodia, and subsequently was needed to trigger cell migration. So we presume BNIPL-2 could be involved in cell migration and cancer metastasis.

前期实验发现，BNIPL-2可以和Cdc42GAP发生相互作用，并且抑制Cdc42GAP的活性；而且BNIPL-2中有一个重要的结构域：BCH domain，它可以诱导细胞伪足的产生，从而参与调节细胞的运动，提示了BNIPL-2可能参与细胞的运动和肿瘤转移。

This experimental study, based on the ischemia-reperfusion injury model of the isolated rabbit liver, was to investigate hepatocellular apoptosis and its mechanism during cold preservation-reperfusion of donor liver and assess the structural and functional damages of the reperfusing donor liver subsequent to rewarm ischemia. In addition, the expression and effects of ICAM-1 mRNA were mainly observed during rewarm ischemia-reperfusion of donor livers. Our experiment included three parts: The first was establishment of the isolated liver ischemia-reperfusion injury model in rabbits.

本实验在建立离体供肝缺血-再灌注损伤模型的基础上，研究供肝低温保存-再灌注过程中肝细胞凋亡现象及其相关机制；探讨作为临床肝移植中供肝的另一缺血阶段—复温缺血对再灌注后供肝结构及功能的损伤作用，并着重观察供肝复温缺血-再灌注期间组织细胞内细胞间粘附分子-1 mRNA的表达规律及其作用。

For a big chunk of credit-card losses; the number of filings (and thus charge-off rates) would be rising again, whether

年美国个人破产法的一个改动使得破产登记急速下降，而后引起了信用卡大规模的亏损。

Eph. 4:23 And that you be renewed in the spirit of your mind

弗四23 而在你们心思的灵里得以更新