细胞生成

MEBT/MEBO is efficacious in preventing progressive necrosis of deep burn wounds and promoting wound healing. Its mechanism may be that MEBO can lessen the injury such as local capillary dilation, cell edema and inflammatory infiltration, and prevent micro-thrombosis. So it can effectively improve local microcirculation in the wounds and save parabiotic tissue.

MEBT/MEBO能防止大鼠深Ⅱ度烧伤创面进行性加深，促进烧伤后的创面愈合，其作用机制为MEBT/MEBO能使创面局部毛细血管扩张、细胞水肿、炎性浸润减轻，防止微血栓生成，有效改善创面局部的微环境，挽救间生态组织。

These proteins govern osteoblast differentiation and thus allow the growth of periosteal bone.

这些蛋白掌管成骨细胞的分化，由此导致骨膜骨的生成。

AIM: To study the mechanism of the inhibitory effect of human polymorphonuclear leukocytes on the production of tumor necrosis factor-α in U937 cells.

目的：初步探讨人中性粒细胞抑制U937细胞肿瘤坏死因子-α生成的机制。

Abstract］ AIM: To study the mechanism of the inhibitory effect of human polymorphonuclear leukocytes on the production of tumor necrosis factor-α in U937 cells.

摘 要］目的：初步探讨人中性粒细胞抑制U937细胞肿瘤坏死因子-α生成的机制。

Investigate proliferative ability of bFGF transfected HUVEC in vitro and its effect on angiogenesis in nude mice.

观察bFGF基因转染的HUVEC细胞增殖能力的变化，及在裸鼠体内促血管生成作用。

Drugs inhibiting collagen hydroxylation and accumulation are expected to improe this situation, making prolyl 4-hydroxylase (P4H), the key enzyme of intracellular collagen processing, a rational target for pharmacological interention.

抑制胶原羟化和沉积的药物被临床医生所期盼，生成的羟脯氨酸酶{P4H}，肝内细胞胶原生成的关键酶，是药物介入的理想目标。

In contrast, we show herein that striatal neuroblasts are generated without decline at least for 4 months after stroke in adult rats.

然而，我们的实验显示了与此观点相反的结果，在脑卒中发生至少4个月后，我们发现，成年大鼠纹状体内神经母细胞的生成并未出现下降的趋势。

VEGF can encourage vascular endothelial cell to divise、propagate、migrate, and add vasopermeability, facilitate producing vascellum sustentaculum, to profit blood supply in ischemic region.

血管内皮生长因子能促进微血管内皮细胞分裂、增殖、迁移，增加血管通透性，促进血管支持物的生成，有利于缺血区血液的供应。

Its mechanism has not been made clear , we now have no effective method to synteresis and therapy. However, as generally acknowledged, the growth and metastasis of tumor have close cooperation with Vascularization and tumor extracellular matrix.

乳腺癌骨转移的机理目前尚未弄清，医学发展至今尚没有一种较有效的方法来预防及治疗骨转移，但肿瘤的生长及转移与血管生成及肿瘤细胞外基质密切相关的理论已逐渐得到公认。

PTX at tenuates the ALI induced by LPS or sepsis in rat. The therapeutic effects of PTX include:improve oxygenation, at tenuate pulmonary edema formation, enhance animal survival in sepsis-induced ALI. The mechanisms may related with the effects of PTX inhibiting the chemotaxis, migration, activation of inflammatory leukocytes, diminishing the production of active oxygen, and inhibiting inflammatory cytokine release partly.

4PTX对大鼠内毒素性ALI及脓毒症性ALI均有一定的防治作用，主要表现在改善动脉血氧合，减轻肺水肿形成，提高脓毒症性ALI大鼠的存活率等方面，其机理可能与抑制白细胞趋化与激活，减少肺内白细胞渗出及活性氧产生，一定程度上抑制炎性细胞因子生成等有关。

Do you know, i need you to come back

你知道吗，我需要你回来

Yang yinshu、Wang xiangsheng、Li decang,The first discovery of haemaphysalis conicinna.

1〕 杨银书，王祥生，李德昌。安徽省首次发现嗜群血蜱。